Replication of phenotypically mixed human immunodeficiency virus type 1 virions containing catalytically active and catalytically inactive reverse transcriptase.
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Human immunodeficiency virus type 1 cDNAs produced in the presence of APOBEC3G exhibit defects in plus-strand DNA transfer and integrationHIV-1 reverse transcriptionTime-Resolved Imaging of Single HIV-1 Uncoating In Vitro and in Living CellsPersistence of viral reservoirs in multiple tissues after antiretroviral therapy suppression in a macaque RT-SHIV modelHIV-1 Reverse Transcriptase Structure with RNase H Inhibitor Dihydroxy Benzoyl Naphthyl Hydrazone Bound at a Novel SiteFluorescent image analysis of HIV-1 and HIV-2 uncoating kinetics in the presence of old world monkey TRIM5αHRP2 determines the efficiency and specificity of HIV-1 integration in LEDGF/p75 knockout cells but does not contribute to the antiviral activity of a potent LEDGF/p75-binding site integrase inhibitor.HIV-1 uses dynamic capsid pores to import nucleotides and fuel encapsidated DNA synthesisPerturbation of the P-body component Mov10 inhibits HIV-1 infectivity.Mechanism of reduction in titers from lentivirus vectors carrying large inserts in the 3'LTR.Elimination of retroviral infectivity by N-ethylmaleimide with preservation of functional envelope glycoproteinsLens epithelium-derived growth factor fusion proteins redirect HIV-1 DNA integration.ATPgammaS disrupts human immunodeficiency virus type 1 virion core integrity.Mechanism for nucleoside analog-mediated abrogation of HIV-1 replication: balance between RNase H activity and nucleotide excisionVirion instability of human immunodeficiency virus type 1 reverse transcriptase (RT) mutated in the protease cleavage site between RT p51 and the RT RNase H domain.Mutations in the RNase H domain of HIV-1 reverse transcriptase affect the initiation of DNA synthesis and the specificity of RNase H cleavage in vivo.Analysis of 2-LTR circle junctions of viral DNA in infected cells.Reduced fitness in cell culture of HIV-1 with nonnucleoside reverse transcriptase inhibitor-resistant mutations correlates with relative levels of reverse transcriptase content and RNase H activity in virions.Characterization of the polymerase and RNase H activities of human foamy virus reverse transcriptase.Mutation of the catalytic domain of the foamy virus reverse transcriptase leads to loss of processivity and infectivity.Nuclear localization of human immunodeficiency virus type 1 preintegration complexes (PICs): V165A and R166A are pleiotropic integrase mutants primarily defective for integration, not PIC nuclear import.The level of reverse transcriptase (RT) in human immunodeficiency virus type 1 particles affects susceptibility to nonnucleoside RT inhibitors but not to lamivudineHuman immunodeficiency virus type 1 nucleocapsid zn(2+) fingers are required for efficient reverse transcription, initial integration processes, and protection of newly synthesized viral DNA.Analysis of HIV-1 replication block due to substitutions at F61 residue of reverse transcriptase reveals additional defects involving the RNase H functionBlinded, multicenter comparison of methods to detect a drug-resistant mutant of human immunodeficiency virus type 1 at low frequency.4'-C-methyl-2'-deoxyadenosine and 4'-C-ethyl-2'-deoxyadenosine inhibit HIV-1 replicationSlower uncoating is associated with impaired replicative capability of simian-tropic HIV-1Inserting a nuclear targeting signal into a replication-competent Moloney murine leukemia virus affects viral export and is not sufficient for cell cycle-independent infection.A cell-based strategy to assess intrinsic inhibition efficiencies of HIV-1 reverse transcriptase inhibitors.Impaired infectivity of ritonavir-resistant HIV is rescued by heat shock protein 90AB1.Chemokine CXCL8 promotes HIV-1 replication in human monocyte-derived macrophages and primary microglia via nuclear factor-κB pathway.The requirement for nucleoporin NUP153 during human immunodeficiency virus type 1 infection is determined by the viral capsid.Mutation of amino acids in the connection domain of human immunodeficiency virus type 1 reverse transcriptase that contact the template-primer affects RNase H activity.Transduction of bone-marrow-derived mesenchymal stem cells by using lentivirus vectors pseudotyped with modified RD114 envelope glycoproteins.Requirements for efficient minus strand strong-stop DNA transfer in human immunodeficiency virus 1.The effects of RNase H inhibitors and nevirapine on the susceptibility of HIV-1 to AZT and 3TC.Efficiency of human immunodeficiency virus type 1 postentry infection processes: evidence against disproportionate numbers of defective virions.The HIV-1 reverse transcriptase mutants G190S and G190A, which confer resistance to non-nucleoside reverse transcriptase inhibitors, demonstrate reductions in RNase H activity and DNA synthesis from tRNA(Lys, 3) that correlate with reductions in repHIV-1 capsid-targeting domain of cleavage and polyadenylation specificity factor 6A Novel Leu92 Mutant of HIV-1 Reverse Transcriptase with a Selective Deficiency in Strand Transfer Causes a Loss of Viral Replication.
P2860
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P2860
Replication of phenotypically mixed human immunodeficiency virus type 1 virions containing catalytically active and catalytically inactive reverse transcriptase.
description
2001 nî lūn-bûn
@nan
2001 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
name
Replication of phenotypically ...... nactive reverse transcriptase.
@ast
Replication of phenotypically ...... nactive reverse transcriptase.
@en
type
label
Replication of phenotypically ...... nactive reverse transcriptase.
@ast
Replication of phenotypically ...... nactive reverse transcriptase.
@en
prefLabel
Replication of phenotypically ...... nactive reverse transcriptase.
@ast
Replication of phenotypically ...... nactive reverse transcriptase.
@en
P2093
P2860
P1433
P1476
Replication of phenotypically ...... nactive reverse transcriptase.
@en
P2093
A L Ferris
J G Julias
S H Hughes
P2860
P304
P356
10.1128/JVI.75.14.6537-6546.2001
P577
2001-07-01T00:00:00Z