CR1 Knops blood group alleles are not associated with severe malaria in the Gambia - Wikidata - awgldk - TriplyDB

CR1 Knops blood group alleles are not associated with severe malaria in the Gambia

CR1 Knops blood group alleles are not associated with severe malaria in the Gambia

http://www.wikidata.org/entity/Q33849061

about

How malaria has affected the human genome and what human genetics can teach us about malaria Complement receptor 1 polymorphisms associated with resistance to severe malaria in Kenya Extensive genomic variability of knops blood group polymorphisms is associated with sickle cell disease in Africa Infections of People with Complement Deficiencies and Patients Who Have Undergone Splenectomy Increased survival in B-cell-deficient mice during experimental cerebral malaria suggests a role for circulating immune complexes.Heterogeneity of alleles encoding high- and low-prevalence red blood cell antigens across Africa: useful data to facilitate transfusion in African patients.Geographical distribution of complement receptor type 1 variants and their associated disease risk.Identification of the Kna/Knb polymorphism and a method for Knops genotyping.Blood groups and malaria: fresh insights into pathogenesis and identification of targets for intervention.Adhesion of Plasmodium falciparum-infected erythrocytes to human cells: molecular mechanisms and therapeutic implications.Complement receptor 1 and malaria.Lack of evidence from studies of soluble protein fragments that Knops blood group polymorphisms in complement receptor-type 1 are driven by malaria.Human genetic polymorphisms in the Knops blood group are not associated with a protective advantage against Plasmodium falciparum malaria in Southern Ghana.A human complement receptor 1 polymorphism that reduces Plasmodium falciparum rosetting confers protection against severe malaria.Association between Knops blood group polymorphisms and susceptibility to malaria in an endemic area of the Brazilian Amazon Blood Groups in Infection and Host Susceptibility.Blood group O protects against severe Plasmodium falciparum malaria through the mechanism of reduced rosetting High Incidence of Malaria Along the Sino-Burmese Border Is Associated With Polymorphisms of CR1, IL-1A, IL-4R, IL-4, NOS, and TNF, But Not With G6PD Deficiency.Severity of malaria in relation to a complement receptor 1 polymorphism: a case-control study.Human gene copy number variation and infectious disease.Complement receptor 1 and the molecular pathogenesis of malaria.Knops blood group polymorphism and susceptibility to Mycobacterium tuberculosis infection.Knops blood group haplotypes among distinct Brazilian populations.Evidence for malaria selection of a CR1 haplotype in Sardinia.No Evidence that Knops Blood Group Polymorphisms Affect Complement Receptor 1 Clustering on Erythrocytes.Two complement receptor one alleles have opposing associations with cerebral malaria and interact with α+thalassaemia.

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CR1 Knops blood group alleles are not associated with severe malaria in the Gambia

http://www.wikidata.org/entity/Q33849061

description

2003 nî lūn-bûn

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2003 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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2003 թվականի հուլիսին հրատարակված գիտական հոդված

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2003年の論文

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2003年論文

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2003年論文

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2003年論文

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2003年論文

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2003年論文

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2003年论文

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name

CR1 Knops blood group alleles are not associated with severe malaria in the Gambia

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CR1 Knops blood group alleles are not associated with severe malaria in the Gambia

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type

label

CR1 Knops blood group alleles are not associated with severe malaria in the Gambia

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CR1 Knops blood group alleles are not associated with severe malaria in the Gambia

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prefLabel

CR1 Knops blood group alleles are not associated with severe malaria in the Gambia

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CR1 Knops blood group alleles are not associated with severe malaria in the Gambia

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SJ.GENE.6363980

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Genes and Immunity

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CR1 Knops blood group alleles are not associated with severe malaria in the Gambia

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P2093

A V S Hill

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D T McNamara

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J Alexandra Rowe

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J Fitness

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J M Moulds

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L J Kasehagen

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P A Zimmerman

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P2860

Detecting recent positive selection in the human genome from haplotype structure

Inherited resistance to HIV-1 conferred by an inactivating mutation in CC chemokine receptor 5: studies in populations with contrasting clinical phenotypes, defined racial background, and quantified risk

MEGA2: molecular evolutionary genetics analysis software

P. falciparum rosetting mediated by a parasite-variant erythrocyte membrane protein and complement-receptor 1

Common west African HLA antigens are associated with protection from severe malaria

Adaptive protein evolution at the Adh locus in Drosophila

DnaSP version 3: an integrated program for molecular population genetics and molecular evolution analysis

Parasite virulence factors during falciparum malaria: rosetting, cytoadherence, and modulation of cytoadherence by cytokines

Structure-function relationships of complement receptor type 1.

Neurological sequelae of cerebral malaria in children.

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