Human cytomegalovirus US2 endoplasmic reticulum-lumenal domain dictates association with major histocompatibility complex class I in a locus-specific manner.
about
Protein disulphide isomerase is required for signal peptide peptidase-mediated protein degradationThe evolution of natural killer cell receptorsA Coevolutionary Arms Race between Hosts and Viruses Drives Polymorphism and Polygenicity of NK Cell Receptors.Antigen presentation subverted: Structure of the human cytomegalovirus protein US2 bound to the class I molecule HLA-A2Crystal structure of adenovirus E3-19K bound to HLA-A2 reveals mechanism for immunomodulationStructural Mechanism of ER Retrieval of MHC Class I by CowpoxThe Structure of the Cytomegalovirus-Encoded m04 Glycoprotein, a Prototypical Member of the m02 Family of ImmunoevasinsDissection of the dislocation pathway for type I membrane proteins with a new small molecule inhibitor, eeyarestatinSignal peptide peptidase is required for dislocation from the endoplasmic reticulumKnow Thyself: NK-Cell Inhibitory Receptors Prompt Self-Tolerance, Education, and Viral Control.Can Selective MHC Downregulation Explain the Specificity and Genetic Diversity of NK Cell Receptors?Rhesus cytomegalovirus contains functional homologues of US2, US3, US6, and US11.The endoplasmic reticulum lumenal domain of the adenovirus type 2 E3-19K protein binds to peptide-filled and peptide-deficient HLA-A*1101 molecules.The MHC class I antigen presentation pathway: strategies for viral immune evasion.Human cytomegalovirus protein pp71 disrupts major histocompatibility complex class I cell surface expression.Kaposi's sarcoma-associated herpesvirus K3 utilizes the ubiquitin-proteasome system in routing class major histocompatibility complexes to late endocytic compartments.Presentation of an immunodominant immediate-early CD8+ T cell epitope resists human cytomegalovirus immunoevasion.Human cytomegalovirus US3 chimeras containing US2 cytosolic residues acquire major histocompatibility class I and II protein degradation properties.Binding of human cytomegalovirus US2 to major histocompatibility complex class I and II proteins is not sufficient for their degradation.Natural killer cell receptors for major histocompatibility complex class I and related molecules in cytomegalovirus infection.Human cytomegalovirus US2 causes similar effects on both major histocompatibility complex class I and II proteins in epithelial and glial cellsViral modulation of antigen presentation: manipulation of cellular targets in the ER and beyond.A Novel MHC-I Surface Targeted for Binding by the MCMV m06 Immunoevasin Revealed by Solution NMR.A bipartite trigger for dislocation directs the proteasomal degradation of an endoplasmic reticulum membrane glycoproteinPathogen evasion strategies for the major histocompatibility complex class I assembly pathway.Innate immune signaling pathways in animals: beyond reductionism.Diverse immune evasion strategies by human cytomegalovirus.Herpesviruses placating the unwilling host: manipulation of the MHC class II antigen presentation pathway.How do pathogens drive the evolution of paired receptors?Classical and non-classical MHC I molecule manipulation by human cytomegalovirus: so many targets—but how many arrows in the quiver?Visualization of the ER-to-cytosol dislocation reaction of a type I membrane protein.Human cytomegalovirus gene products US2 and US11 differ in their ability to attack major histocompatibility class I heavy chains in dendritic cells.Human herpesvirus 7 u21 downregulates classical and nonclassical class I major histocompatibility complex molecules from the cell surfaceUbiquitination of MHC class I heavy chains is essential for dislocation by human cytomegalovirus-encoded US2 but not US11.A structural determinant of human cytomegalovirus US2 dictates the down-regulation of class I major histocompatibility molecules.Dissection of the interaction of the human cytomegalovirus-derived US2 protein with major histocompatibility complex class I molecules: prominent role of a single arginine residue in human leukocyte antigen-A2.Structural and functional analysis of human cytomegalovirus US3 protein.Structure of the Adenovirus Type 4 (Species E) E3-19K/HLA-A2 Complex Reveals Species-Specific Features in MHC Class I Recognition.Membrane-specific, host-derived factors are required for US2- and US11-mediated degradation of major histocompatibility complex class I molecules.Feeling manipulated: cytomegalovirus immune manipulation.
P2860
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P2860
Human cytomegalovirus US2 endoplasmic reticulum-lumenal domain dictates association with major histocompatibility complex class I in a locus-specific manner.
description
2001 nî lūn-bûn
@nan
2001 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
name
Human cytomegalovirus US2 endo ...... I in a locus-specific manner.
@ast
Human cytomegalovirus US2 endo ...... I in a locus-specific manner.
@en
type
label
Human cytomegalovirus US2 endo ...... I in a locus-specific manner.
@ast
Human cytomegalovirus US2 endo ...... I in a locus-specific manner.
@en
prefLabel
Human cytomegalovirus US2 endo ...... I in a locus-specific manner.
@ast
Human cytomegalovirus US2 endo ...... I in a locus-specific manner.
@en
P2093
P2860
P1433
P1476
Human cytomegalovirus US2 endo ...... I in a locus-specific manner.
@en
P2093
P2860
P304
P356
10.1128/JVI.75.11.5197-5204.2001
P577
2001-06-01T00:00:00Z