Chronic nicotine administration exacerbates tau pathology in a transgenic model of Alzheimer's disease. - Wikidata - awgldk - TriplyDB

Chronic nicotine administration exacerbates tau pathology in a transgenic model of Alzheimer's disease.

Chronic nicotine administration exacerbates tau pathology in a transgenic model of Alzheimer's disease.

http://www.wikidata.org/entity/Q33863598

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Molecular interplay between mammalian target of rapamycin (mTOR), amyloid-beta, and Tau: effects on cognitive impairments.Methylene blue reduces aβ levels and rescues early cognitive deficit by increasing proteasome activity Natural Phytochemicals in the Treatment and Prevention of Dementia: An Overview Amyloid beta: multiple mechanisms of toxicity and only some protective effects?Statins reduce the neurofibrillary tangle burden in a mouse model of tauopathy Cholinergic treatments with emphasis on m1 muscarinic agonists as potential disease-modifying agents for Alzheimer's disease Formulation of a medical food cocktail for Alzheimer's disease: beneficial effects on cognition and neuropathology in a mouse model of the disease Autoimmune manifestations in the 3xTg-AD model of Alzheimer's disease.Amyloidogenic light chains induce cardiomyocyte contractile dysfunction and apoptosis via a non-canonical p38alpha MAPK pathway.Detailed immunohistochemical characterization of temporal and spatial progression of Alzheimer's disease-related pathologies in male triple-transgenic mice Memantine improves cognition and reduces Alzheimer's-like neuropathology in transgenic mice.Nicotinic Receptors: Role in Addiction and Other Disorders of the Brain.Increased risk of dementia in patients with osteoporosis: a population-based retrospective cohort analysis.Genetically altering Abeta distribution from the brain to the vasculature ameliorates tau pathology Smoking and increased Alzheimer's disease risk: a review of potential mechanisms Cholinesterase inhibitors may increase phosphorylated tau in Alzheimer's disease.Increased risk of dementia in patients exposed to nitrogen dioxide and carbon monoxide: a population-based retrospective cohort study.Inducing autophagy by rapamycin before, but not after, the formation of plaques and tangles ameliorates cognitive deficits.Role of vascular risk factors and vascular dysfunction in Alzheimer's disease.Gene-environment interaction research and transgenic mouse models of Alzheimer's disease.Nicotine: specific role in angiogenesis, proliferation and apoptosis.Cigarette smoking accelerated brain aging and induced pre-Alzheimer-like neuropathology in rats Genetic knockdown of brain-derived neurotrophic factor in 3xTg-AD mice does not alter Aβ or tau pathology Brain imaging of nicotinic receptors in Alzheimer's disease.Impaired attention in the 3xTgAD mouse model of Alzheimer's disease: rescue by donepezil (Aricept).Smoking and Alzheimer's disease among Mongolian and Han Chinese aged 55 years and over living in the Inner Mongolia farming area of China.A dynamic relationship between intracellular and extracellular pools of Abeta.Increased hippocampal excitability in the 3xTgAD mouse model for Alzheimer's disease in vivo.Functional alpha7 nicotinic receptors are expressed on immature granule cells of the postnatal dentate gyrus Functional genomic screen and network analysis reveal novel modifiers of tauopathy dissociated from tau phosphorylation.Lithium reduces tau phosphorylation but not A beta or working memory deficits in a transgenic model with both plaques and tangles.Amyloid beta and the longest-lived rodent: the naked mole-rat as a model for natural protection from Alzheimer's disease.Children of persons with Alzheimer disease: what does the future hold?β-arrestin1 regulates γ-secretase complex assembly and modulates amyloid-β pathology.Natural products as a source of Alzheimer's drug leads.Anti-inflammatory and immune therapy for Alzheimer's disease: current status and future directions.Abeta inhibits the proteasome and enhances amyloid and tau accumulation Nicotinic receptors containing the alpha7 subunit: a model for rational drug design.Neuronal nicotinic receptors as novel targets for inflammation and neuroprotection: mechanistic considerations and clinical relevance.Alpha7 nicotinic acetylcholine receptor is required for amyloid pathology in brain endothelial cells induced by Glycoprotein 120, methamphetamine and nicotine.

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P2860

Chronic nicotine administration exacerbates tau pathology in a transgenic model of Alzheimer's disease.

http://www.wikidata.org/entity/Q33863598

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2005年の論文

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Chronic nicotine administratio ...... model of Alzheimer's disease.

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Chronic nicotine administratio ...... model of Alzheimer's disease.

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Chronic nicotine administratio ...... model of Alzheimer's disease.

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Proceedings of the National Academy of Sciences of the United States of America

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Chronic nicotine administratio ...... model of Alzheimer's disease.

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Antonella Caccamo

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Frances M Leslie

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Frank M LaFerla

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Kevin Liang

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Kim N Green

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Levina Tran

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Salvatore Oddo

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Yiling Chen

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P2860

beta-Amyloid(1-42) binds to alpha7 nicotinic acetylcholine receptor with high affinity. Implications for Alzheimer's disease pathology

A subtype of nicotinic cholinergic receptor in rat brain is composed of alpha 4 and beta 2 subunits and is up-regulated by chronic nicotine treatment

Tau protein kinase I is essential for amyloid beta-protein-induced neurotoxicity

Measuring nicotinic receptors with characteristics of alpha4beta2, alpha3beta2 and alpha3beta4 subtypes in rat tissues by autoradiography

Beta-amyloid activates the mitogen-activated protein kinase cascade via hippocampal alpha7 nicotinic acetylcholine receptors: In vitro and in vivo mechanisms related to Alzheimer's disease

Triple-transgenic model of Alzheimer's disease with plaques and tangles: intracellular Abeta and synaptic dysfunction

Evidence that neurones accumulating amyloid can undergo lysis to form amyloid plaques in Alzheimer's disease.

The nicotinic acetylcholine receptor, smoking, and Alzheimer's disease.

p38 mitogen activated protein kinase as a therapeutic target for Alzheimer's disease.

Acetylcholinesterase inhibition in Alzheimer's Disease.

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3046-3051

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102

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8026816

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15705720

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Alzheimer's disease

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