The tetrodotoxin-resistant sodium channel SNS has a specialized function in pain pathways. - Wikidata - awgldk - TriplyDB

The tetrodotoxin-resistant sodium channel SNS has a specialized function in pain pathways.

The tetrodotoxin-resistant sodium channel SNS has a specialized function in pain pathways.

http://www.wikidata.org/entity/Q33871067

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Association between tetrodotoxin resistant channels and lipid rafts regulates sensory neuron excitability Role of acid-sensing ion channel 3 in sub-acute-phase inflammation Na(v) 1.8-null mice show stimulus-dependent deficits in spinal neuronal activity.Overview of the voltage-gated sodium channel family Modulation of the cardiac sodium channel Nav1.5 by fibroblast growth factor homologous factor 1B De novo mutations in the sodium-channel gene SCN1A cause severe myoclonic epilepsy of infancy Nociceptor-specific gene deletion reveals a major role for Nav1.7 (PN1) in acute and inflammatory pain Pain as a channelopathy Distinct Nav1.7-dependent pain sensations require different sets of sensory and sympathetic neurons Structural determinants of drugs acting on the Nav1.8 channel Synthetic ciguatoxins selectively activate Nav1.8-derived chimeric sodium channels expressed in HEK293 cells Developmental neurotoxicity of pyrethroid insecticides: critical review and future research needs Neuropathic pain develops normally in mice lacking both Na(v)1.7 and Na(v)1.8.Conotoxins That Could Provide Analgesia through Voltage Gated Sodium Channel Inhibition Main ion channels and receptors associated with visceral hypersensitivity in irritable bowel syndrome The Specification and Maturation of Nociceptive Neurons from Human Embryonic Stem Cells Two tetrodotoxin-resistant sodium channels in human dorsal root ganglion neurons Neuromodulation of Na+ channels: an unexpected form of cellular plasticity Annexin II light chain regulates sensory neuron-specific sodium channel expression Arthritis and pain. Neurogenic origin of joint pain Endogenous opioids contribute to insensitivity to pain in humans and mice lacking sodium channel Nav1.7 Nav1.7 and other voltage-gated sodium channels as drug targets for pain relief Analgesic activity of a novel use-dependent sodium channel blocker, crobenetine, in mono-arthritic rats Pain genes Global Nav1.7 knockout mice recapitulate the phenotype of human congenital indifference to pain Inhibition of Inactive States of Tetrodotoxin-Sensitive Sodium Channels Reduces Spontaneous Firing of C-Fiber Nociceptors and Produces Analgesia in Formalin and Complete Freund's Adjuvant Models of Pain Relationship of axonal voltage-gated sodium channel 1.8 (NaV1.8) mRNA accumulation to sciatic nerve injury-induced painful neuropathy in rats Cellular and Molecular Mechanisms of Pain A gain-of-function voltage-gated sodium channel 1.8 mutation drives intense hyperexcitability of A- and C-fiber neurons.In vivo characterization of distinct modality-specific subsets of somatosensory neurons using GCaMP.Multiple sodium channel isoforms mediate the pathological effects of Pacific ciguatoxin-1.Activity and connectivity changes of central projection areas revealed by functional magnetic resonance imaging in NaV1.8-deficient mice upon cold signaling.Spinal inflammatory hyperalgesia is mediated by prostaglandin E receptors of the EP2 subtype.Structures of muO-conotoxins from Conus marmoreus. I nhibitors of tetrodotoxin (TTX)-sensitive and TTX-resistant sodium channels in mammalian sensory neurons.Nav1.9 channel contributes to mechanical and heat pain hypersensitivity induced by subacute and chronic inflammation.Chemosensory properties of murine nasal and cutaneous trigeminal neurons identified by viral tracing.Bilateral downregulation of Nav1.8 in dorsal root ganglia of rats with bone cancer pain induced by inoculation with Walker 256 breast tumor cells Correlation of Nav1.8 and Nav1.9 sodium channel expression with neuropathic pain in human subjects with lingual nerve neuromas.Aquaporin-1 tunes pain perception by interaction with Na(v)1.8 Na+ channels in dorsal root ganglion neurons.Identifying the Ion Channels Responsible for Signaling Gastro-Intestinal Based Pain

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P2860

The tetrodotoxin-resistant sodium channel SNS has a specialized function in pain pathways.

http://www.wikidata.org/entity/Q33871067

description

1999 nî lūn-bûn

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P2860

Improved patch-clamp techniques for high-resolution current recording from cells and cell-free membrane patches

Onset and progression of motor deficits in motor neuron degeneration (mnd) mice are unaltered by the glycine/NMDA receptor antagonist L-701,324 or the MAO-B inhibitor R(-)-deprenyl

A tetrodotoxin-resistant voltage-gated sodium channel expressed by sensory neurons

Disruption of the proto-oncogene int-2 in mouse embryo-derived stem cells: a general strategy for targeting mutations to non-selectable genes

Molecular cloning of a putative tetrodotoxin-resistant rat heart Na+ channel isoform.

Assembly and cell surface expression of heteromeric and homomeric gamma-aminobutyric acid type A receptors.

Hyperalgesic agents increase a tetrodotoxin-resistant Na+ current in nociceptors.

Gene-targeting studies of mammalian behavior: is it the mutation or the background genotype?

A tetrodotoxin-resistant sodium current mediates inflammatory pain in the rat.

NaN, a novel voltage-gated Na channel, is expressed preferentially in peripheral sensory neurons and down-regulated after axotomy.

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