Transcriptional activation of the Axl and PDGFR-α by c-Met through a ras- and Src-independent mechanism in human bladder cancer. - Wikidata - awgldk - TriplyDB

Transcriptional activation of the Axl and PDGFR-α by c-Met through a ras- and Src-independent mechanism in human bladder cancer.

Transcriptional activation of the Axl and PDGFR-α by c-Met through a ras- and Src-independent mechanism in human bladder cancer.

http://www.wikidata.org/entity/Q33874421

about

An overview of the c-MET signaling pathway Biomarker development in MET-targeted therapy Met in urological cancers LY2801653 is an orally bioavailable multi-kinase inhibitor with potent activity against MET, MST1R, and other oncoproteins, and displays anti-tumor activities in mouse xenograft models Comparison of the gene expression profiles of human fetal cortical astrocytes with pluripotent stem cell derived neural stem cells identifies human astrocyte markers and signaling pathways and transcription factors active in human astrocytes Analysis of progress and challenges for various patterns of c-MET-targeted molecular imaging: a systematic review.Animal model of naturally occurring bladder cancer: characterization of four new canine transitional cell carcinoma cell lines.Redundant kinase activation and resistance of EGFR-tyrosine kinase inhibitors.The c-MET Network as Novel Prognostic Marker for Predicting Bladder Cancer Patients with an Increased Risk of Developing Aggressive Disease.Combined inhibition of AXL, Lyn and p130Cas kinases block migration of triple negative breast cancer cells.Targeting Axl with an high-affinity inhibitory aptamer.Profiling phospho-signaling networks in breast cancer using reverse-phase protein arrays HGF/Met and FOXM1 form a positive feedback loop and render pancreatic cancer cells resistance to Met inhibition and aggressive phenotypes.The receptor AXL diversifies EGFR signaling and limits the response to EGFR-targeted inhibitors in triple-negative breast cancer cells.The receptor tyrosine kinase Axl in cancer: biological functions and therapeutic implications.Emerging drugs for urothelial carcinoma.Targeting the VEGF pathway in metastatic bladder cancer.The role of Lutheran/basal cell adhesion molecule in human bladder carcinogenesis.MET: roles in epithelial-mesenchymal transition and cancer stemness.Key Roles of AXL and MER Receptor Tyrosine Kinases in Resistance to Multiple Anticancer Therapies.Fra-1 controls motility of bladder cancer cells via transcriptional upregulation of the receptor tyrosine kinase AXL.Expression and role of TYRO3 and AXL as potential therapeutical targets in leiomyosarcoma.Relationship Between Increased Expression of the Axl/Gas6 Signal Cascade and Prognosis of Patients with Upper Tract Urothelial Carcinoma.

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P2860

Transcriptional activation of the Axl and PDGFR-α by c-Met through a ras- and Src-independent mechanism in human bladder cancer.

http://www.wikidata.org/entity/Q33874421

description

2011 nî lūn-bûn

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2011 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2011年の論文

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2011年論文

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2011年論文

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Chen-Yun Yeh

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Cheng-Huang Shen

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Chung-Ta Lee

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Giri Raghavaraju

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Hsiao-Sheng Liu

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Hsuan-Heng Yeh

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Jung-Hsien Chiang

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Jyh-Wei Shin

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Nan-Haw Chow

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Shin-Mei Shin

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P2860

Universal Reference RNA as a standard for microarray experiments

Overexpression of platelet-derived growth factor receptor alpha in breast cancer is associated with tumour progression

The protein tyrosine kinase family of the human genome

The RON and MET oncogenes are co-expressed in human ovarian carcinomas and cooperate in activating invasiveness

Hepatocyte growth factor/scatter factor activates the ETS1 transcription factor by a RAS-RAF-MEK-ERK signaling pathway

Fibroblast growth factor-2 represses platelet-derived growth factor receptor-alpha (PDGFR-alpha) transcription via ERK1/2-dependent Sp1 phosphorylation and an atypical cis-acting element in the proximal PDGFR-alpha promoter

Global survey of phosphotyrosine signaling identifies oncogenic kinases in lung cancer

Ligand-independent activation of c-Met by fibronectin and α(5)β(1)-integrin regulates ovarian cancer invasion and metastasis.

Global gene expression profiling of dimethylnitrosamine-induced liver fibrosis: from pathological and biochemical data to microarray analysis.

Hepatocyte growth factor/scatter factor-induced intracellular signalling.

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