Perinatal exposure to bisphenol-a and the development of metabolic syndrome in CD-1 mice. - Wikidata - awgldk - TriplyDB

Perinatal exposure to bisphenol-a and the development of metabolic syndrome in CD-1 mice.

Perinatal exposure to bisphenol-a and the development of metabolic syndrome in CD-1 mice.

http://www.wikidata.org/entity/Q33874480

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Plastics derived endocrine disruptors (BPA, DEHP and DBP) induce epigenetic transgenerational inheritance of obesity, reproductive disease and sperm epimutations The estrogenic endocrine disrupting chemical bisphenol A (BPA) and obesity Bisphenol A Interaction With Brain Development and Functions The estrogenic content of rodent diets, bedding, cages, and water bottles and its effect on bisphenol A studies Early life factors and type 2 diabetes mellitus Do interactions between gut ecology and environmental chemicals contribute to obesity and diabetes?Exposure to bisphenol-A during pregnancy partially mimics the effects of a high-fat diet altering glucose homeostasis and gene expression in adult male mice Influence of Bisphenol A on Type 2 Diabetes Mellitus Role of environmental chemicals in diabetes and obesity: a National Toxicology Program workshop review Endocrine disrupting chemicals and the developmental programming of adipogenesis and obesity EDC-2: The Endocrine Society's Second Scientific Statement on Endocrine-Disrupting Chemicals Prenatal exposure to phenols and growth in boys Urinary bisphenol A and obesity: NHANES 2003-2006 Maternal bisphenol a exposure during pregnancy and its association with adipokines in Mexican-American children Prenatal and postnatal bisphenol A exposure and body mass index in childhood in the CHAMACOS cohort Perinatal bisphenol A exposure and adult glucose homeostasis: identifying critical windows of exposure Bisphenol-A impairs insulin action and up-regulates inflammatory pathways in human subcutaneous adipocytes and 3T3-L1 cells Perinatal exposure to perfluorooctane sulfonate affects glucose metabolism in adult offspring Developmental programming: interaction between prenatal BPA exposure and postnatal adiposity on metabolic variables in female sheep Metabolism Disrupting Chemicals and Metabolic Disorders Critical evaluation of key evidence on the human health hazards of exposure to bisphenol A.Bisphenol A, obesity, and type 2 diabetes mellitus: genuine concern or unnecessary preoccupation?Epigenetics meets endocrinology.What Are We Putting in Our Food That Is Making Us Fat? Food Additives, Contaminants, and Other Putative Contributors to Obesity.Designing Endocrine Disruption Out of the Next Generation of Chemicals Bisphenol A exposure during pregnancy disrupts glucose homeostasis in mothers and adult male offspring.Pharmacologic sex hormones in pregnancy in relation to offspring obesity.The impact of neonatal bisphenol-A exposure on sexually dimorphic hypothalamic nuclei in the female rat.Childhood obesity and environmental chemicals.Impact of gestational bisphenol A on oxidative stress and free fatty acids: Human association and interspecies animal testing studies.Getting big on BPA: role for BPA in obesity?Obesogens, stem cells and the developmental programming of obesity.Developmental exposure to endocrine-disrupting chemicals programs for reproductive tract alterations and obesity later in life.Early programing of uterine tissue by bisphenol A: Critical evaluation of evidence from animal exposure studies.Bisphenol A diglycidyl ether induces adipogenic differentiation of multipotent stromal stem cells through a peroxisome proliferator-activated receptor gamma-independent mechanism.Soy but not bisphenol A (BPA) or the phytoestrogen genistin alters developmental weight gain and food intake in pregnant rats and their offspring.Perinatal bisphenol A exposure promotes hyperactivity, lean body composition, and hormonal responses across the murine life course.Effects of low doses of bisphenol A on the metabolome of perinatally exposed CD-1 mice Adipocytes under assault: environmental disruption of adipose physiology.Sex-dependent effects of developmental exposure to bisphenol A and ethinyl estradiol on metabolic parameters and voluntary physical activity.

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P2860

Perinatal exposure to bisphenol-a and the development of metabolic syndrome in CD-1 mice.

http://www.wikidata.org/entity/Q33874480

description

2010 nî lūn-bûn

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2010 թուականի Մարտին հրատարակուած գիտական յօդուած

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2010 թվականի մարտին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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Perinatal exposure to bisphenol-a and the development of metabolic syndrome in CD-1 mice.

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Perinatal exposure to bisphenol-a and the development of metabolic syndrome in CD-1 mice.

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Perinatal exposure to bisphenol-a and the development of metabolic syndrome in CD-1 mice.

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Perinatal exposure to bisphenol-a and the development of metabolic syndrome in CD-1 mice.

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Perinatal exposure to bisphenol-a and the development of metabolic syndrome in CD-1 mice.

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Perinatal exposure to bisphenol-a and the development of metabolic syndrome in CD-1 mice.

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Perinatal exposure to bisphenol-a and the development of metabolic syndrome in CD-1 mice

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April M Haller

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Joyce E Sorrell

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Ronald J Jandacek

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Stephen C Woods

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P2860

Urogenital abnormalities in men exposed to diethylstilbestrol in utero: a cohort study

NTP-CERHR expert panel report on the reproductive and developmental toxicity of bisphenol A

Bisphenol-A and the great divide: a review of controversies in the field of endocrine disruption

Exposure to environmentally relevant doses of the xenoestrogen bisphenol-A alters development of the fetal mouse mammary gland

Prenatal exposure to bisphenol a at environmentally relevant doses adversely affects the murine female reproductive tract later in life

In vivo effects of bisphenol A in laboratory rodent studies

Developmental exposure to endocrine disruptors and the obesity epidemic

Perinatal exposure to low doses of bisphenol A affects body weight, patterns of estrous cyclicity, and plasma LH levels

Structural evidence for endocrine disruptor bisphenol A binding to human nuclear receptor ERR gamma

Overexpression of GPR40 in pancreatic beta-cells augments glucose-stimulated insulin secretion and improves glucose tolerance in normal and diabetic mice.

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metabolic syndrome

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