Deficiency of Atf3, an adaptive-response gene, protects islets and ameliorates inflammation in a syngeneic mouse transplantation model.
about
Adult cardiac expression of the activating transcription factor 3, ATF3, promotes ventricular hypertrophyATF3 is a novel regulator of mouse neutrophil migrationA systems biology analysis of brain microvascular endothelial cell lipotoxicityThe roles of ATF3, an adaptive-response gene, in high-fat-diet-induced diabetes and pancreatic beta-cell dysfunction.Lipotoxic brain microvascular injury is mediated by activating transcription factor 3-dependent inflammatory and oxidative stress pathways.Different molecular mechanisms involved in spontaneous and oxidative stress-induced mitochondrial fragmentation in tripeptidyl peptidase-1 (TPP-1)-deficient fibroblasts.Failure of the adaptive unfolded protein response in islets of obese mice is linked with abnormalities in β-cell gene expression and progression to diabetesUpregulation of ATF3 inhibits expression of the pro-inflammatory cytokine IL-6 during Neisseria gonorrhoeae infectionIntermittent hypoxia exacerbates pancreatic β-cell dysfunction in a mouse model of diabetes mellitusATF3 expression in the corpus luteum: possible role in luteal regression.A method for murine islet isolation and subcapsular kidney transplantationAtf3 mutant mice show reduced axon regeneration and impaired regeneration-associated gene induction after peripheral nerve injury.Hyperoxia-induced activation of the integrated stress response in the newborn rat lung.ATF3 inhibits the inflammation induced by Mycoplasma pneumonia in vitro and in vivo.Activating transcription factor 3 and the nervous system.Feedback regulation by Atf3 in the endothelin-1-responsive transcriptome of cardiomyocytes: Egr1 is a principal Atf3 targetInduction of ATF3 gene network by triglyceride-rich lipoprotein lipolysis products increases vascular apoptosis and inflammationHuman islets express a marked proinflammatory molecular signature prior to transplantation.Physiologic Doses of Bilirubin Contribute to Tolerance of Islet Transplants by Suppressing the Innate Immune Response.Nano-Encapsulation of Bilirubin in Pluronic F127-Chitosan Improves Uptake in β Cells and Increases Islet Viability and Function after Hypoxic Stress.ATF3 expression in cardiomyocytes preserves homeostasis in the heart and controls peripheral glucose tolerance.Pancreatic β-cells activate a JunB/ATF3-dependent survival pathway during inflammation
P2860
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P2860
Deficiency of Atf3, an adaptive-response gene, protects islets and ameliorates inflammation in a syngeneic mouse transplantation model.
description
2010 nî lūn-bûn
@nan
2010 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի մարտին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Deficiency of Atf3, an adaptiv ...... c mouse transplantation model.
@ast
Deficiency of Atf3, an adaptiv ...... c mouse transplantation model.
@en
type
label
Deficiency of Atf3, an adaptiv ...... c mouse transplantation model.
@ast
Deficiency of Atf3, an adaptiv ...... c mouse transplantation model.
@en
prefLabel
Deficiency of Atf3, an adaptiv ...... c mouse transplantation model.
@ast
Deficiency of Atf3, an adaptiv ...... c mouse transplantation model.
@en
P2093
P2860
P1433
P1476
Deficiency of Atf3, an adaptiv ...... ic mouse transplantation model
@en
P2093
P2860
P2888
P304
P356
10.1007/S00125-010-1696-X
P577
2010-03-28T00:00:00Z
P5875
P6179
1003226349