Activation of lipid metabolism contributes to interleukin-8 production during Chlamydia trachomatis infection of cervical epithelial cells
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cPLA2 regulates the expression of type I interferons and intracellular immunity to Chlamydia trachomatisThe intracellular bacteria Chlamydia hijack peroxisomes and utilize their enzymatic capacity to produce bacteria-specific phospholipidsA bovine model of respiratory Chlamydia psittaci infection: challenge dose titrationChlamydia trachomatis co-opts the FGF2 signaling pathway to enhance infectionInfections and cancer: the "fifty shades of immunity" hypothesis.Eukaryotic protein recruitment into the Chlamydia inclusion: implications for survival and growth.Impact of salmonella infection on host hormone metabolism revealed by metabolomics.Loss of FADS2 function severely impairs the use of HeLa cells as an in vitro model for host response studies involving fatty acid effectsInfection of human endothelial cells with spotted Fever group rickettsiae stimulates cyclooxygenase 2 expression and release of vasoactive prostaglandinsCleavage of p65/RelA of the NF-kappaB pathway by ChlamydiaChlamydia trachomatis Relies on Autonomous Phospholipid Synthesis for Membrane Biogenesis.MAPK Activation Is Essential for Waddlia chondrophila Induced CXCL8 Expression in Human Epithelial Cells.The extracellular signal-regulated kinase/mitogen-activated protein kinase pathway induces the inflammatory factor interleukin-8 following Chlamydia trachomatis infection.Intracellular interleukin-1alpha mediates interleukin-8 production induced by Chlamydia trachomatis infection via a mechanism independent of type I interleukin-1 receptor.Metabolic host responses to infection by intracellular bacterial pathogensKilling me softly: chlamydial use of proteolysis for evading host defenses.Chlamydia preserves the mitochondrial network necessary for replication via microRNA-dependent inhibition of fission.A bovine model of a respiratory Parachlamydia acanthamoebae infection.The multifaceted role of oestrogen in enhancing Chlamydia trachomatis infection in polarized human endometrial epithelial cells.Host cell responses to Chlamydia pneumoniae in gamma interferon-induced persistence overlap those of productive infection and are linked to genes involved in apoptosis, cell cycle, and metabolism.Activation of the host cell proinflammatory interleukin-8 response by Chlamydia trachomatis.
P2860
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P2860
Activation of lipid metabolism contributes to interleukin-8 production during Chlamydia trachomatis infection of cervical epithelial cells
description
2005 nî lūn-bûn
@nan
2005 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
Activation of lipid metabolism ...... n of cervical epithelial cells
@ast
Activation of lipid metabolism ...... n of cervical epithelial cells
@en
type
label
Activation of lipid metabolism ...... n of cervical epithelial cells
@ast
Activation of lipid metabolism ...... n of cervical epithelial cells
@en
prefLabel
Activation of lipid metabolism ...... n of cervical epithelial cells
@ast
Activation of lipid metabolism ...... n of cervical epithelial cells
@en
P2093
P2860
P1476
Activation of lipid metabolism ...... n of cervical epithelial cells
@en
P2093
David P Mikolon
Elaine Y Fukuda
Erguang Li
Milena Iacobelli-Martinez
Sonya P Lad
P2860
P304
P356
10.1128/IAI.73.7.4017-4024.2005
P407
P577
2005-07-01T00:00:00Z