Effects of lovastatin on breast cancer cells: a proteo-metabonomic study. - Wikidata - awgldk - TriplyDB

Effects of lovastatin on breast cancer cells: a proteo-metabonomic study.

Effects of lovastatin on breast cancer cells: a proteo-metabonomic study.

http://www.wikidata.org/entity/Q33889477

about

Plant sterols as anticancer nutrients: evidence for their role in breast cancer The KLK5 protease suppresses breast cancer by repressing the mevalonate pathway GC-TOFMS analysis of metabolites in adherent MDCK cells and a novel strategy for identifying intracellular metabolic markers for use as cell amount indicators in data normalization.Adaptation to statins restricts human tumour growth in Nude mice.Statins and breast cancer prognosis: evidence and opportunities.Fluvastatin mediated breast cancer cell death: a proteomic approach to identify differentially regulated proteins in MDA-MB-231 cells.Differential effects of lovastatin on cisplatin responses in normal human mesothelial cells versus cancer cells: implication for therapy.Influence of histatin 5 on Candida albicans mitochondrial protein expression assessed by quantitative mass spectrometry.Metabolic characterization of triple negative breast cancer.Mitochondria-targeted nitroxides exacerbate fluvastatin-mediated cytostatic and cytotoxic effects in breast cancer cells Inhibition of NF-κB Pathway and Modulation of MAPK Signaling Pathways in Glioblastoma and Implications for Lovastatin and Tumor Necrosis Factor-Related Apoptosis Inducing Ligand (TRAIL) Combination Therapy.Metabolic Profiling Comparison of Human Pancreatic Ductal Epithelial Cells and Three Pancreatic Cancer Cell Lines using NMR Based Metabonomics.Effects of Lovastatin on MDA-MB-231 Breast Cancer Cells: An Antibody Microarray Analysis.Transcriptional profiling of breast cancer cells in response to mevinolin: Evidence of cell cycle arrest, DNA degradation and apoptosis.Simvastatin-induced breast cancer cell death and deactivation of PI3K/Akt and MAPK/ERK signalling are reversed by metabolic products of the mevalonate pathway.Lovastatin lowers the risk of breast cancer: a population-based study using logistic regression with a random effects model Metabolomics approaches and applications in prostate cancer research.Inhibition of N-Myc down regulated gene 1 in in vitro cultured human glioblastoma cells.Simvastatin prevents triple-negative breast cancer metastasis in pre-clinical models through regulation of FOXO3a.Anti-cancer characteristics of mevinolin against three different solid tumor cell lines was not solely p53-dependent.How plausible is the use of dietary n-3 PUFA in the adjuvant therapy of cancer?Combined spectroscopic and molecular docking approach to probing binding interactions between lovastatin and calf thymus DNA.Cell cycle inhibition without disruption of neurogenesis is a strategy for treatment of aberrant cell cycle diseases: an update.Simvastatin Reduces Cancerogenic Potential of Renal Cancer Cells via Geranylgeranyl Pyrophosphate and Mevalonate Pathway.

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P2860

Effects of lovastatin on breast cancer cells: a proteo-metabonomic study.

http://www.wikidata.org/entity/Q33889477

description

2010 nî lūn-bûn

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2010 թուականի Մարտին հրատարակուած գիտական յօդուած

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2010 թվականի մարտին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Effects of lovastatin on breast cancer cells: a proteo-metabonomic study.

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Effects of lovastatin on breast cancer cells: a proteo-metabonomic study.

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type

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Effects of lovastatin on breast cancer cells: a proteo-metabonomic study.

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Effects of lovastatin on breast cancer cells: a proteo-metabonomic study.

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Effects of lovastatin on breast cancer cells: a proteo-metabonomic study.

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Effects of lovastatin on breast cancer cells: a proteo-metabonomic study.

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Breast Cancer Research

P1476

Effects of lovastatin on breast cancer cells: a proteo-metabonomic study.

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Jelena Klawitter

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Jost Klawitter

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Touraj Shokati

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Uwe Christians

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Vanessa Moll

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P2860

Ras-GTPase activating protein (GAP): a putative effector for Ras

Fatty acid synthesis: a potential selective target for antineoplastic therapy

Minichromosome maintenance proteins are direct targets of the ATM and ATR checkpoint kinases

Structural mechanism for statin inhibition of HMG-CoA reductase

Rho GTPases in cell biology

On the Origin of Cancer Cells

Association of statin use with a pathologic complete response to neoadjuvant chemoradiation for rectal cancer

MST/MLK2, a member of the mixed lineage kinase family, directly phosphorylates and activates SEK1, an activator of c-Jun N-terminal kinase/stress-activated protein kinase

Cdc42--the centre of polarity

All ras proteins are polyisoprenylated but only some are palmitoylated

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