PI3K rescues the detrimental effects of chronic Akt activation in the heart during ischemia/reperfusion injury - Wikidata - awgldk - TriplyDB

PI3K rescues the detrimental effects of chronic Akt activation in the heart during ischemia/reperfusion injury

PI3K rescues the detrimental effects of chronic Akt activation in the heart during ischemia/reperfusion injury

http://www.wikidata.org/entity/Q33892209

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Myostatin regulates cardiomyocyte growth through modulation of Akt signaling Akt1 regulates pathological angiogenesis, vascular maturation and permeability in vivo Akt1 in endothelial cell and angiogenesis Akt isoforms in vascular disease Optimization of cardiac metabolism in heart failure Cardiac contractile dysfunction in insulin-resistant rats fed a high-fat diet is associated with elevated CD36-mediated fatty acid uptake and esterification The critical role of Akt in cardiovascular function Regulation of phosphodiesterase 3 and inducible cAMP early repressor in the heart NO signaling and S-nitrosylation regulate PTEN inhibition in neurodegeneration SERCA2a gene therapy restores microRNA-1 expression in heart failure via an Akt/FoxO3A-dependent pathway Abnormal myocardial insulin signalling in type 2 diabetes and left-ventricular dysfunction.Arg972 Insulin receptor substrate-1 is associated with decreased serum angiotensin-converting enzyme 2 levels in acute myocardial infarction patients: in vivo and in vitro evidence Phosphoinositide-dependent kinase 1 and mTORC2 synergistically maintain postnatal heart growth and heart function in mice.Protein Kinases as Drug Development Targets for Heart Disease Therapy Functional interaction of phosphatase and tensin homologue (PTEN) with the E3 ligase NEDD4-1 during neuronal response to zinc.Mitochondrial integrity: preservation through Akt/Pim-1 kinase signaling in the cardiomyocyte Myostatin activation in patients with advanced heart failure and after mechanical unloading.Vitamin B1 analog benfotiamine prevents diabetes-induced diastolic dysfunction and heart failure through Akt/Pim-1-mediated survival pathway Akt1 in the cardiovascular system: friend or foe?Cardiac-specific expression of the tetracycline transactivator confers increased heart function and survival following ischemia reperfusion injury Resveratrol improves myocardial perfusion in a swine model of hypercholesterolemia and chronic myocardial ischemia Pathological role of serum- and glucocorticoid-regulated kinase 1 in adverse ventricular remodeling.Activated protein C has a protective effect against myocardial I/R injury by improvement of endothelial function and activation of AKT1.Cardiac-specific overexpression of metallothionein rescues against cigarette smoking exposure-induced myocardial contractile and mitochondrial damage Role of Endothelial Cells in Myocardial Ischemia-Reperfusion Injury.Nuclear targeting of Akt antagonizes aspects of cardiomyocyte hypertrophy.Enhanced cardiac Akt/protein kinase B signaling contributes to pathological cardiac hypertrophy in part by impairing mitochondrial function via transcriptional repression of mitochondrion-targeted nuclear genes.AMPK inhibition in health and disease.A small molecule activator of AKT does not reduce ischemic injury of the rat heart.Mitochondrial dysfunction in uremic cardiomyopathy.Phosphoinositide 3-kinase gamma/delta inhibition limits infarct size after myocardial ischemia/reperfusion injury.Mitochondria to nucleus translocation of AIF in mice lacking Hsp70 during ischemia/reperfusion.Cardiac expression of human type 2 iodothyronine deiodinase increases glucose metabolism and protects against doxorubicin-induced cardiac dysfunction in male mice.Differential effects of Akt1 signaling on short- versus long-term consequences of myocardial infarction and reperfusion injury Loss of receptor activity-modifying protein 3 exacerbates cardiac hypertrophy and transition to heart failure in a sex-dependent manner.Expression of SGLT1 in Human Hearts and Impairment of Cardiac Glucose Uptake by Phlorizin during Ischemia-Reperfusion Injury in Mice.The secretome of myocardial telocytes modulates the activity of cardiac stem cells.Cardiac mTOR rescues the detrimental effects of diet-induced obesity in the heart after ischemia-reperfusion.Hypercholesterolemia is associated with hyperactive cardiac mTORC1 and mTORC2 signaling.Artery/vein specification is governed by opposing phosphatidylinositol-3 kinase and MAP kinase/ERK signaling.

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P2860

PI3K rescues the detrimental effects of chronic Akt activation in the heart during ischemia/reperfusion injury

http://www.wikidata.org/entity/Q33892209

description

2005 nî lūn-bûn

@nan

2005 թուականի Յուլիսին հրատարակուած գիտական յօդուած

@hyw

2005 թվականի հուլիսին հրատարակված գիտական հոդված

@hy

2005年の論文

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2005年論文

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2005年論文

@zh-hant

2005年論文

@zh-hk

2005年論文

@zh-mo

2005年論文

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2005年论文

@wuu

name

PI3K rescues the detrimental e ...... ng ischemia/reperfusion injury

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PI3K rescues the detrimental e ...... ng ischemia/reperfusion injury

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type

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PI3K rescues the detrimental e ...... ng ischemia/reperfusion injury

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PI3K rescues the detrimental e ...... ng ischemia/reperfusion injury

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prefLabel

PI3K rescues the detrimental e ...... ng ischemia/reperfusion injury

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PI3K rescues the detrimental e ...... ng ischemia/reperfusion injury

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Journal of Clinical Investigation

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PI3K rescues the detrimental e ...... ng ischemia/reperfusion injury

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P2093

Annarosa Leri

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Anthony Rosenzweig

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Brian A Hemmings

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David A Kass

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Federica del Monte

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Hunter C Champion

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Judith K Gwathmey

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Ling Li

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Luanda Grazette

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Takashi Matsui

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P2860

Regulation of endothelium-derived nitric oxide production by the protein kinase Akt

Activation of nitric oxide synthase in endothelial cells by Akt-dependent phosphorylation

Protein kinase C-zeta-induced phosphorylation of Ser318 in insulin receptor substrate-1 (IRS-1) attenuates the interaction with the insulin receptor and the tyrosine phosphorylation of IRS-1

The PI3K-PDK1 connection: more than just a road to PKB

A simplified system for generating recombinant adenoviruses

The phosphoinositide 3-kinase pathway

The c-Jun NH(2)-terminal kinase promotes insulin resistance during association with insulin receptor substrate-1 and phosphorylation of Ser(307)

Ischemic preconditioning activates phosphatidylinositol-3-kinase upstream of protein kinase C

Characterization of sgk, a novel member of the serine/threonine protein kinase gene family which is transcriptionally induced by glucocorticoids and serum

Prevention of fat-induced insulin resistance by salicylate

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2128-2138

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10.1172/JCI23073

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8

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115

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2005-07-07T00:00:00Z

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7737817

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16007268

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P921

reperfusion injury

P932

1172227

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