Enhanced cardiac Akt/protein kinase B signaling contributes to pathological cardiac hypertrophy in part by impairing mitochondrial function via transcriptional repression of mitochondrion-targeted nuclear genes.

Enhanced cardiac Akt/protein kinase B signaling contributes to pathological cardiac hypertrophy in part by impairing mitochondrial function via transcriptional repression of mitochondrion-targeted nuclear genes.

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http://www.wikidata.org/entity/Q35069561
Insulin Signaling and Heart Failure.Post-translational modifications of the cardiac proteome in diabetes and heart failureEffect of lithium on ventricular remodelling in infarcted rats via the Akt/mTOR signalling pathwaysExendin-4 protects against post-myocardial infarction remodelling via specific actions on inflammation and the extracellular matrixIntegromics network meta-analysis on cardiac aging offers robust multi-layer modular signatures and reveals micronome synergism.Phosphoinositide dependent protein kinase 1 is required for exercise-induced cardiac hypertrophy but not the associated mitochondrial adaptations.Deletion of IGF-1 Receptors in Cardiomyocytes Attenuates Cardiac Aging in Male Mice.Insulin and IGF-1 receptors regulate FoxO-mediated signaling in muscle proteostasis.Genetic disruption of the cardiomyocyte circadian clock differentially influences insulin-mediated processes in the heart.Dysregulation of microRNAs and renin-angiotensin system in high salt diet-induced cardiac dysfunction in uninephrectomized rats.Inhibiting Insulin-Mediated β2-Adrenergic Receptor Activation Prevents Diabetes-Associated Cardiac Dysfunction.Exercise-Induced Changes in Glucose Metabolism Promote Physiological Cardiac Growth.Glucose transporter 4-deficient hearts develop maladaptive hypertrophy in response to physiological or pathological stresses.OPA1 deficiency promotes secretion of FGF21 from muscle that prevents obesity and insulin resistance.Activation of IGF-1 receptors and Akt signaling by systemic hyperinsulinemia contributes to cardiac hypertrophy but does not regulate cardiac autophagy in obese diabetic mice.Integrative analysis of transcriptomics and clinical data uncovers the tumor-suppressive activity of MITF in prostate cancerAcetylation of Mitochondrial Proteins in the Heart: The Role of SIRT3
P2860

Enhanced cardiac Akt/protein kinase B signaling contributes to pathological cardiac hypertrophy in part by impairing mitochondrial function via transcriptional repression of mitochondrion-targeted nuclear genes.

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http://www.wikidata.org/entity/Q35069561
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2014 nî lūn-bûn
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2014 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի դեկտեմբերին հրատարակված գիտական հոդված
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2014年の論文
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2014年論文
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2014年論文
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2014年論文
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2014年論文
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2014年論文
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2014年论文
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name
Enhanced cardiac Akt/protein k ...... ndrion-targeted nuclear genes.
@ast
Enhanced cardiac Akt/protein k ...... ndrion-targeted nuclear genes.
@en
type
label
Enhanced cardiac Akt/protein k ...... ndrion-targeted nuclear genes.
@ast
Enhanced cardiac Akt/protein k ...... ndrion-targeted nuclear genes.
@en
prefLabel
Enhanced cardiac Akt/protein k ...... ndrion-targeted nuclear genes.
@ast
Enhanced cardiac Akt/protein k ...... ndrion-targeted nuclear genes.
@en
P1433
P1476
P2093
P2860
P304
P31
P356
P407
P433
P478
P50
P577
P698
P921
P932
P356
P1433
P1476
Enhanced cardiac Akt/protein k ...... ondrion-targeted nuclear genes
@en
P2093
P2860
P304
P31
P356
P407
P433
P478
P50
P577
P698
P921
P932