Herpes simplex virus vector-mediated gene delivery of glutamic acid decarboxylase reduces detrusor overactivity in spinal cord-injured rats - Wikidata - awgldk - TriplyDB

Herpes simplex virus vector-mediated gene delivery of glutamic acid decarboxylase reduces detrusor overactivity in spinal cord-injured rats

Herpes simplex virus vector-mediated gene delivery of glutamic acid decarboxylase reduces detrusor overactivity in spinal cord-injured rats

http://www.wikidata.org/entity/Q33895720

about

Gene therapy for lower urinary tract dysfunction Neural control of the lower urinary tract Neural mechanisms underlying lower urinary tract dysfunction Changes in afferent activity after spinal cord injury.Intravesical TRPV4 blockade reduces repeated variate stress-induced bladder dysfunction by increasing bladder capacity and decreasing voiding frequency in male rats Suppression of detrusor-sphincter dyssynergia by herpes simplex virus vector mediated gene delivery of glutamic acid decarboxylase in spinal cord injured rats.The role of brain-derived neurotrophic factor (BDNF) in the development of neurogenic detrusor overactivity (NDO).Cellular therapies for treating pain associated with spinal cord injury Plasticity in reflex pathways to the lower urinary tract following spinal cord injury HSV vector-mediated GAD67 suppresses neuropathic pain induced by perineural HIV gp120 in rats through inhibition of ROS and Wnt5a Gene therapy for the treatment of chronic peripheral nervous system pain.Treatment of overactive bladder: what is on the horizon?Central nervous targets for the treatment of bladder dysfunction.Emerging drugs for overactive bladder.New Frontiers of Basic Science Research in Neurogenic Lower Urinary Tract Dysfunction.Improvement in detrusor-sphincter dyssynergia by bladder-wall injection of replication-defective herpes simplex virus vector-mediated gene delivery of kynurenine aminotransferase II in spinal cord injury rats.Effect of urethral wall injection of replication-defective herpes simplex virus-mediated gene transfer of kynurenine aminotransferase on urethral pressure in spinal cord-injured rats.Morphological changes in different populations of bladder afferent neurons detected by herpes simplex virus (HSV) vectors with cell-type-specific promoters in mice with spinal cord injury.Herpes simplex virus vector-mediated gene transfer of kynurenine aminotransferase improves detrusor overactivity in spinal cord-injured rats.Roles of Glycinergic and Gamma-aminobutyric-ergic Mechanisms in the Micturition Reflex in Rats.

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P2860

Herpes simplex virus vector-mediated gene delivery of glutamic acid decarboxylase reduces detrusor overactivity in spinal cord-injured rats

http://www.wikidata.org/entity/Q33895720

description

2009 nî lūn-bûn

@nan

2009 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի փետրվարին հրատարակված գիտական հոդված

@hy

2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年论文

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name

Herpes simplex virus vector-me ...... ty in spinal cord-injured rats

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Herpes simplex virus vector-me ...... ty in spinal cord-injured rats

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Herpes simplex virus vector-me ...... ty in spinal cord-injured rats

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Herpes simplex virus vector-me ...... ty in spinal cord-injured rats

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D Wolfe

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J C Glorioso

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J R Goss

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K Sugaya

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M B Chancellor

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M Miyazato

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N Yoshimura

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W C de Groat

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W F Goins

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P2860

Effects of acute bolus and chronic continuous intrathecal baclofen on genitourinary dysfunction due to spinal cord pathology.

Herpes simplex virus type 1 vector-mediated expression of nerve growth factor protects dorsal root ganglion neurons from peroxide toxicity

Gene therapy for bladder overactivity and nociception with herpes simplex virus vectors expressing preproenkephalin

Immunohistochemical analysis of primary sensory neurons latently infected with herpes simplex virus type 1.

Gene transfer of glutamic acid decarboxylase reduces neuropathic pain.

Herpes simplex virus type 2 (HSV-2) establishes latent infection in a different population of ganglionic neurons than HSV-1: role of latency-associated transcripts.

Functional interactions between herpes simplex virus immediate-early proteins during infection: gene expression as a consequence of ICP27 and different domains of ICP4.

The herpes simplex virus immediate-early protein ICP0 affects transcription from the viral genome and infected-cell survival in the absence of ICP4 and ICP27

Mechanisms underlying the recovery of lower urinary tract function following spinal cord injury.

A novel latency-active promoter is contained within the herpes simplex virus type 1 UL flanking repeats.

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660-668

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10.1038/GT.2009.5

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5

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16

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24023485

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19225548

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vector-borne disease

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2881227

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