Transfer, analysis, and reversion of the fibrous dysplasia cellular phenotype in human skeletal progenitors. - Wikidata - awgldk - TriplyDB

Transfer, analysis, and reversion of the fibrous dysplasia cellular phenotype in human skeletal progenitors.

Transfer, analysis, and reversion of the fibrous dysplasia cellular phenotype in human skeletal progenitors.

http://www.wikidata.org/entity/Q33925044

about

"Mesenchymal" stem cells in human bone marrow (skeletal stem cells): a critical discussion of their nature, identity, and significance in incurable skeletal disease.Stem cells and bone diseases: new tools, new perspective Osteoblast-specific expression of the fibrous dysplasia (FD)-causing mutation Gsα(R201C) produces a high bone mass phenotype but does not reproduce FD in the mouse.Constitutive expression of Gsα(R201C) in mice produces a heritable, direct replica of human fibrous dysplasia bone pathology and demonstrates its natural history.Loss of Gsα early in the osteoblast lineage favors adipogenic differentiation of mesenchymal progenitors and committed osteoblast precursors.Cell sources for bone regeneration: the good, the bad, and the ugly (but promising).Wnt/β-catenin signaling is differentially regulated by Gα proteins and contributes to fibrous dysplasia.[Fibrous dysplasia: overview]Denosumab treatment for fibrous dysplasia.Skeletal progenitors and the GNAS gene: fibrous dysplasia of bone read through stem cells.Cyclic AMP signaling in bone marrow stromal cells has reciprocal effects on the ability of mesenchymal stem cells to differentiate into mature osteoblasts versus mature adipocytes.No Identical "Mesenchymal Stem Cells" at Different Times and Sites: Human Committed Progenitors of Distinct Origin and Differentiation Potential Are Incorporated as Adventitial Cells in Microvessels.Dental perspectives in fibrous dysplasia and McCune-Albright syndrome.Prompt clinical and biochemical response to denosumab in a young adult patient with craniofacial fibrous dysplasia.Gsα Controls Cortical Bone Quality by Regulating Osteoclast Differentiation via cAMP/PKA and β-Catenin Pathways.The meaning, the sense and the significance: translating the science of mesenchymal stem cells into medicine.Fibrous Dysplasia/McCune-Albright Syndrome: Clinical and Translational Perspectives.Heterotrimeric G proteins in the control of parathyroid hormone actions.Back to the future: moving beyond "mesenchymal stem cells".MESENCHYMAL STROMAL CELLS AND THEIR ORTHOPAEDIC APPLICATIONS.Expression of an active Gαs mutant in skeletal stem cells is sufficient and necessary for fibrous dysplasia initiation and maintenance.Constitutive stimulatory G protein activity in limb mesenchyme impairs bone growth.

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P2860

Transfer, analysis, and reversion of the fibrous dysplasia cellular phenotype in human skeletal progenitors.

http://www.wikidata.org/entity/Q33925044

description

2010 nî lūn-bûn

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2010 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2010 թվականի մայիսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Transfer, analysis, and revers ...... in human skeletal progenitors.

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Transfer, analysis, and revers ...... in human skeletal progenitors.

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Transfer, analysis, and revers ...... in human skeletal progenitors.

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Transfer, analysis, and revers ...... in human skeletal progenitors.

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Transfer, analysis, and revers ...... in human skeletal progenitors.

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Transfer, analysis, and revers ...... in human skeletal progenitors.

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Journal of Bone and Mineral Research

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Transfer, analysis, and revers ...... in human skeletal progenitors.

@en

P2093

Alessia Funari

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Benedetto Sacchetti

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Cristina Remoli

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Mara Riminucci

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Paolo Bianco

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Stefania Piersanti

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Stefano Michienzi

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P2860

A system for stable expression of short interfering RNAs in mammalian cells

Relevant cAMP-specific phosphodiesterase isoforms in human pituitary: effect of Gs(alpha) mutations

Self-renewing osteoprogenitors in bone marrow sinusoids can organize a hematopoietic microenvironment

CREB activation induces adipogenesis in 3T3-L1 cells.

FGF-23 in fibrous dysplasia of bone and its relationship to renal phosphate wasting

Fibrous dysplasia of bone in the McCune-Albright syndrome: abnormalities in bone formation.

Quantification using real-time PCR technology: applications and limitations.

Activating mutations of the stimulatory G protein in the McCune-Albright syndrome.

Allele-specific silencing of dominant disease genes.

Conditional suppression of cellular genes: lentivirus vector-mediated drug-inducible RNA interference.

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1103-1116

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10.1359/JBMR.091036

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5

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25

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Isabella Saggio

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2010-05-01T00:00:00Z

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38052717

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19874199

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5524372

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