FGF-23 in fibrous dysplasia of bone and its relationship to renal phosphate wasting - Wikidata - awgldk - TriplyDB

FGF-23 in fibrous dysplasia of bone and its relationship to renal phosphate wasting

FGF-23 in fibrous dysplasia of bone and its relationship to renal phosphate wasting

http://www.wikidata.org/entity/Q34227442

about

McCune-Albright syndrome The FGF23-Klotho axis: endocrine regulation of phosphate homeostasis Effects of PTH on osteocyte function FGF23-mediated regulation of systemic phosphate homeostasis: is Klotho an essential player?Receptor specificity of the fibroblast growth factor family. The complete mammalian FGF family FGF23 in Acute and Chronic Illness Fibroblast growth factor receptor signaling in hereditary and neoplastic disease: biologic and clinical implications Miscellaneous non-inflammatory musculoskeletal conditions. Hyperphosphatemic familial tumoral calcinosis (FGF23, GALNT3 and αKlotho)FGF23 and Phosphate Wasting Disorders Skeletal secretion of FGF-23 regulates phosphate and vitamin D metabolism Fibroblast Growth Factor 23: A New Dimension to Diseases of Calcium-Phosphorus Metabolism Role of FGF23 in vitamin D and phosphate metabolism: implications in chronic kidney disease McCune-Albright syndrome and the extraskeletal manifestations of fibrous dysplasia Fibrous dysplasia and fibroblast growth factor-23 regulation Arterial stiffness, vascular calcification and bone metabolism in chronic kidney disease Calcium and phosphate impact cardiovascular risk.Homozygous ablation of fibroblast growth factor-23 results in hyperphosphatemia and impaired skeletogenesis, and reverses hypophosphatemia in Phex-deficient mice Role of matrix extracellular phosphoglycoprotein in the pathogenesis of X-linked hypophosphatemia Regulation of fibroblast growth factor-23 signaling by klotho Bone and muscle: Interactions beyond mechanical.Targeted ablation of Fgf23 demonstrates an essential physiological role of FGF23 in phosphate and vitamin D metabolism FGF-23 in bone biology.Circulating fibroblast growth factor 23 in patients with end-stage renal disease treated by peritoneal dialysis is intact and biologically active Is fibroblast growth factor receptor 4 a suitable target of cancer therapy?"Mesenchymal" stem cells in human bone marrow (skeletal stem cells): a critical discussion of their nature, identity, and significance in incurable skeletal disease.Effects of Different 1-34 Parathyroid Hormone Dosages on Fibroblast Growth Factor-23 Secretion in Human Bone Marrow Cells following Osteogenic Differentiation.Transfer, analysis, and reversion of the fibrous dysplasia cellular phenotype in human skeletal progenitors.Stem cells and bone diseases: new tools, new perspective Osteoblast-specific expression of the fibrous dysplasia (FD)-causing mutation Gsα(R201C) produces a high bone mass phenotype but does not reproduce FD in the mouse.Mutations that cause osteoglophonic dysplasia define novel roles for FGFR1 in bone elongation The calcemic response to continuous parathyroid hormone (PTH)(1-34) infusion in end-stage kidney disease varies according to bone turnover: a potential role for PTH(7-84)FGF23 decreases renal NaPi-2a and NaPi-2c expression and induces hypophosphatemia in vivo predominantly via FGF receptor 1.Establishment of sandwich ELISA for soluble alpha-Klotho measurement: Age-dependent change of soluble alpha-Klotho levels in healthy subjects Hypophosphatemic rickets: revealing novel control points for phosphate homeostasis.Analysis of the biochemical mechanisms for the endocrine actions of fibroblast growth factor-23 Tumor-induced osteomalacia.The changing face of hypophosphatemic disorders in the FGF-23 era Fracture incidence in polyostotic fibrous dysplasia and the McCune-Albright syndrome.Applicability of fibroblast growth factor 23 for evaluation of risk of vertebral fracture and chronic kidney disease-mineral bone disease in elderly chronic kidney disease patients Relationship between Fibroblast Growth Factor-23 and Mineral Metabolism in Chronic Kidney Disease

P2860

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P2860

FGF-23 in fibrous dysplasia of bone and its relationship to renal phosphate wasting

http://www.wikidata.org/entity/Q34227442

description

2003 nî lūn-bûn

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2003 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2003 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2003年の論文

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2003年論文

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2003年論文

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2003年論文

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2003年論文

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2003年論文

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2003年论文

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name

FGF-23 in fibrous dysplasia of bone and its relationship to renal phosphate wasting

@ast

FGF-23 in fibrous dysplasia of bone and its relationship to renal phosphate wasting

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FGF-23 in fibrous dysplasia of bone and its relationship to renal phosphate wasting

@nl

type

label

FGF-23 in fibrous dysplasia of bone and its relationship to renal phosphate wasting

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FGF-23 in fibrous dysplasia of bone and its relationship to renal phosphate wasting

@en

FGF-23 in fibrous dysplasia of bone and its relationship to renal phosphate wasting

@nl

prefLabel

FGF-23 in fibrous dysplasia of bone and its relationship to renal phosphate wasting

@ast

FGF-23 in fibrous dysplasia of bone and its relationship to renal phosphate wasting

@en

FGF-23 in fibrous dysplasia of bone and its relationship to renal phosphate wasting

@nl

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P1433

Journal of Clinical Investigation

P1476

FGF-23 in fibrous dysplasia of bone and its relationship to renal phosphate wasting

@en

P2093

Alessandro Corsi

http://www.w3.org/2001/XMLSchema#string

Anurag Gupta

http://www.w3.org/2001/XMLSchema#string

Kenneth E White

http://www.w3.org/2001/XMLSchema#string

Mara Riminucci

http://www.w3.org/2001/XMLSchema#string

Michael T Collins

http://www.w3.org/2001/XMLSchema#string

Natasha Cherman

http://www.w3.org/2001/XMLSchema#string

Paolo Bianco

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Steven Waguespack

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P2860

MEPE, a new gene expressed in bone marrow and tumors causing osteomalacia

Autosomal dominant hypophosphataemic rickets is associated with mutations in FGF23

A gene (PEX) with homologies to endopeptidases is mutated in patients with X-linked hypophosphatemic rickets. The HYP Consortium

Cloning and characterization of FGF23 as a causative factor of tumor-induced osteomalacia

Use of avidin-biotin-peroxidase complex (ABC) in immunoperoxidase techniques: a comparison between ABC and unlabeled antibody (PAP) procedures

The autosomal dominant hypophosphatemic rickets (ADHR) gene is a secreted polypeptide overexpressed by tumors that cause phosphate wasting

Autosomal-dominant hypophosphatemic rickets (ADHR) mutations stabilize FGF-23

Tumor-induced osteomalacia and the regulation of phosphate homeostasis.

Disorders of phosphate metabolism.

Fibrous dysplasia of bone in the McCune-Albright syndrome: abnormalities in bone formation.

P304

683-692

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scholarly article

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10.1172/JCI18399

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P407

P433

5

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P478

112

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P50

Michael J Econs

Tamara S. Hannon

P577

2003-09-01T00:00:00Z

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12952917

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P932

182207

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