IL-17RA is required for CCL2 expression, macrophage recruitment, and emphysema in response to cigarette smoke.
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A critical role for dendritic cells in the evolution of IL-1β-mediated murine airway diseaseMechanisms underlying helper T-cell plasticity: implications for immune-mediated disease.Signaling network map of the aryl hydrocarbon receptorCytokine inhibition in the treatment of COPD.Directing traffic: IL-17 and IL-22 coordinate pulmonary immune defense.Cigarette smoke exposure exacerbates lung inflammation and compromises immunity to bacterial infection.Macrophage activation state determines the response to rhinovirus infection in a mouse model of allergic asthma.Cigarette smoke activates the proto-oncogene c-src to promote airway inflammation and lung tissue destruction.Molecular processes that drive cigarette smoke-induced epithelial cell fate of the lung.Smoking enhances the proinflammatory effects of nucleotides on cytokine release from human lung.Promotion of lung tumor growth by interleukin-17T helper 17 cells play a critical pathogenic role in lung cancer.IL-17A and serum amyloid A are elevated in a cigarette smoke cessation model associated with the persistence of pigmented macrophages, neutrophils and activated NK cellsIL-17A modulates oxidant stress-induced airway hyperresponsiveness but not emphysema.Exposure to atmospheric particulate matter enhances Th17 polarization through the aryl hydrocarbon receptor.Targeting IL-1β and IL-17A driven inflammation during influenza-induced exacerbations of chronic lung inflammationInterleukin-22: immunobiology and pathology.Association of Lung Inflammatory Cells with Small Airways Function and Exhaled Breath Markers in Smokers - Is There a Specific Role for Mast Cells?Transcriptional profiling and targeted proteomics reveals common molecular changes associated with cigarette smoke-induced lung emphysema development in five susceptible mouse strains.Distinct Contributions of Neutrophils and CCR2+ Monocytes to Pulmonary Clearance of Different Klebsiella pneumoniae Strains.Experimental therapeutics of Nrf2 as a target for prevention of bacterial exacerbations in COPDPathogenesis of chronic obstructive pulmonary diseaseProbing Cellular and Molecular Mechanisms of Cigarette Smoke-Induced Immune Response in the Progression of Chronic Obstructive Pulmonary Disease Using Multiscale Network Modeling.Innate cellular sources of interleukin-17A regulate macrophage accumulation in cigarette- smoke-induced lung inflammation in mice.Pathogenic mechanism of second hand smoke induced inflammation and COPD.The Aryl Hydrocarbon Receptor Influences Transplant Outcomes in Response to Environmental Signals.Simvastatin Suppresses Airway IL-17 and Upregulates IL-10 in Patients With Stable COPD.Experimental Pneumocystis lung infection promotes M2a alveolar macrophage-derived MMP12 productionMaternal smoking and the retinoid pathway in the developing lung.COPD and squamous cell lung cancer: aberrant inflammation and immunity is the common link.Functional characterization of T cell populations in a mouse model of chronic obstructive pulmonary diseaseIL-17A is essential to the development of elastase-induced pulmonary inflammation and emphysema in mice.The aryl hydrocarbon receptor: a novel target for immunomodulation in organ transplantation.Inflammation and emphysema in cigarette smoke-exposed mice when instilled with poly (I:C) or infected with influenza A or respiratory syncytial virusesModerate Aerobic Training Improves Cardiorespiratory Parameters in Elastase-Induced Emphysema.Antiinflammatory effects of bromodomain and extraterminal domain inhibition in cystic fibrosis lung inflammationSerum amyloid A promotes lung neutrophilia by increasing IL-17A levels in the mucosa and γδ T cells.Scabies increased the risk and severity of COPD: a nationwide population-based study.Role of the aryl hydrocarbon receptor (AhR) in lung inflammationInflammaging increases susceptibility to cigarette smoke-induced COPD.
P2860
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P2860
IL-17RA is required for CCL2 expression, macrophage recruitment, and emphysema in response to cigarette smoke.
description
2011 nî lūn-bûn
@nan
2011 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
IL-17RA is required for CCL2 e ...... n response to cigarette smoke.
@ast
IL-17RA is required for CCL2 e ...... n response to cigarette smoke.
@en
type
label
IL-17RA is required for CCL2 e ...... n response to cigarette smoke.
@ast
IL-17RA is required for CCL2 e ...... n response to cigarette smoke.
@en
prefLabel
IL-17RA is required for CCL2 e ...... n response to cigarette smoke.
@ast
IL-17RA is required for CCL2 e ...... n response to cigarette smoke.
@en
P2093
P2860
P1433
P1476
IL-17RA is required for CCL2 e ...... n response to cigarette smoke.
@en
P2093
A McGarry Houghton
Derek A Pociask
James L Kreindler
Jay K Kolls
Jeremy P McAleer
John F Alcorn
Matthew R Keyser
Mingquan Zheng
Steven D Shapiro
P2860
P304
P356
10.1371/JOURNAL.PONE.0020333
P407
P577
2011-05-27T00:00:00Z