Inflammaging increases susceptibility to cigarette smoke-induced COPD. - Wikidata - awgldk - TriplyDB

Inflammaging increases susceptibility to cigarette smoke-induced COPD.

Inflammaging increases susceptibility to cigarette smoke-induced COPD.

http://www.wikidata.org/entity/Q37327421

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Inducible Bronchus-Associated Lymphoid Tissue: Taming Inflammation in the Lung AMP-activated protein kinase reduces inflammatory responses and cellular senescence in pulmonary emphysema Elimination of p19(ARF)-expressing cells enhances pulmonary function in mice.Aging and Lung Disease. Clinical Impact and Cellular and Molecular Pathways.MicroRNA Signature of Cigarette Smoking and Evidence for a Putative Causal Role of MicroRNAs in Smoking-Related Inflammation and Target Organ Damage.Cholesterol metabolism promotes B-cell positioning during immune pathogenesis of chronic obstructive pulmonary disease.The Continuum of Aging and Age-Related Diseases: Common Mechanisms but Different Rates.Lung cellular senescence is independent of aging in a mouse model of COPD/emphysema.Protective effect of water extract of guibi-tang against pulmonary inflammation induced by cigarette smoke and lipopolysaccharide

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Inflammaging increases susceptibility to cigarette smoke-induced COPD.

http://www.wikidata.org/entity/Q37327421

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on 29 May 2015

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Inflammaging increases susceptibility to cigarette smoke-induced COPD.

@en

Inflammaging increases susceptibility to cigarette smoke-induced COPD.

@nl

type

label

Inflammaging increases susceptibility to cigarette smoke-induced COPD.

@en

Inflammaging increases susceptibility to cigarette smoke-induced COPD.

@nl

prefLabel

Inflammaging increases susceptibility to cigarette smoke-induced COPD.

@en

Inflammaging increases susceptibility to cigarette smoke-induced COPD.

@nl

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ONCOTARGET.4027

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Inflammaging increases susceptibility to cigarette smoke-induced COPD.

@en

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Gerrit John-Schuster

http://www.w3.org/2001/XMLSchema#string

Katrin Hager

http://www.w3.org/2001/XMLSchema#string

Oliver Eickelberg

http://www.w3.org/2001/XMLSchema#string

Stefanie Günter

http://www.w3.org/2001/XMLSchema#string

P2860

The hallmarks of aging

A distinct lineage of CD4 T cells regulates tissue inflammation by producing interleukin 17

Global strategy for the diagnosis, management, and prevention of chronic obstructive pulmonary disease. NHLBI/WHO Global Initiative for Chronic Obstructive Lung Disease (GOLD) Workshop summary

The nature of small-airway obstruction in chronic obstructive pulmonary disease

Aging enhances susceptibility to cigarette smoke-induced inflammation through bronchiolar chemokines.

Chronic inflammation induces telomere dysfunction and accelerates ageing in mice.

IL-17RA is required for CCL2 expression, macrophage recruitment, and emphysema in response to cigarette smoke.

Chronic cigarette smoke exposure generates pathogenic T cells capable of driving COPD-like disease in Rag2-/- mice.

Critical transition in tissue homeostasis accompanies murine lung senescence

The development of inducible bronchus-associated lymphoid tissue depends on IL-17.

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30068-30083

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scholarly article

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10.18632/ONCOTARGET.4027

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21

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7

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Ali Önder Yildirim

Thomas M Conlon

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2015-05-29T00:00:00Z

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281081220

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26284585

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5058664

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