TWISTing stemness, inflammation and proliferation of epithelial ovarian cancer cells through MIR199A2/214. - Wikidata - awgldk - TriplyDB

TWISTing stemness, inflammation and proliferation of epithelial ovarian cancer cells through MIR199A2/214.

TWISTing stemness, inflammation and proliferation of epithelial ovarian cancer cells through MIR199A2/214.

http://www.wikidata.org/entity/Q33927452

about

MicroRNA profiling of a CD133(+) spheroid-forming subpopulation of the OVCAR3 human ovarian cancer cell line Flexible and versatile as a chameleon-sophisticated functions of microRNA-199a MicroRNAs in NF-kappaB signaling Osteopontin-A Master Regulator of Epithelial-Mesenchymal Transition MicroRNA Regulation of Epithelial to Mesenchymal Transition Regulation of ovarian cancer stem cells or tumor-initiating cells CADM1 inhibits squamous cell carcinoma progression by reducing STAT3 activity.MicroRNA profiling as tool for in vitro developmental neurotoxicity testing: the case of sodium valproate The Role of Long Non-Coding RNAs in Ovarian Cancer.Ovulation and extra-ovarian origin of ovarian cancer ER stress negatively modulates the expression of the miR-199a/214 cluster to regulates tumor survival and progression in human hepatocellular cancer.The role of microRNAs in ovarian cancer Distinct microRNA expression profiles in prostate cancer stem/progenitor cells and tumor-suppressive functions of let-7.miR-214 targets ATF4 to inhibit bone formation.TWIST and ovarian cancer stem cells: implications for chemoresistance and metastasis Regulation of inflammation by the NF-κB pathway in ovarian cancer stem cells.TWIST1 drives cisplatin resistance and cell survival in an ovarian cancer model, via upregulation of GAS6, L1CAM, and Akt signalling.Parallel analysis of mRNA and microRNA microarray profiles to explore functional regulatory patterns in polycystic kidney disease: using PKD/Mhm rat model.Biomolecular events in cancer revealed by attractor metagenes.Characterization of microRNA expression profiles and the discovery of novel microRNAs involved in cancer during human embryonic development.microRNA-214 contributes to melanoma tumour progression through suppression of TFAP2C.Ovarian cancer stem cells and inflammation Methylation of an intronic region regulates miR-199a in testicular tumor malignancy.Genomics of the NF-κB signaling pathway: hypothesized role in ovarian cancer.MicroRNA profiling reveals that miR-21, miR486 and miR-214 are upregulated and involved in cell survival in Sézary syndrome.Molecular mechanisms of regulation and action of microRNA-199a in testicular germ cell tumor and glioblastomas.Prevalence of epithelial ovarian cancer stem cells correlates with recurrence in early-stage ovarian cancer.Circular RNA ITCH has inhibitory effect on ESCC by suppressing the Wnt/β-catenin pathway.MiR-199a attenuates endometrial stromal cell invasiveness through suppression of the IKKβ/NF-κB pathway and reduced interleukin-8 expression.RNA regulators of host immunity and pathogen adaptive responses in the oral cavity Families of microRNAs Expressed in Clusters Regulate Cell Signaling in Cervical Cancer.Enhanced epithelial to mesenchymal transition (EMT) and upregulated MYC in ectopic lesions contribute independently to endometriosis.Epithelial-mesenchymal transition in ovarian carcinoma Targeted cancer therapy--are the days of systemic chemotherapy numbered?Prognostic significance of MyD88 expression by human epithelial ovarian carcinoma cells.MicroRNA-214 induces dendritic cell switching from tolerance to immunity by targeting β-Catenin signaling Erythropoietin-mediated expression of placenta growth factor is regulated via activation of hypoxia-inducible factor-1α and post-transcriptionally by miR-214 in sickle cell disease.MicroRNA miR-214 regulates ovarian cancer cell stemness by targeting p53/Nanog.The microRNA (miR)-199a/214 cluster mediates opposing effects of progesterone and estrogen on uterine contractility during pregnancy and labor.Current status and implications of microRNAs in ovarian cancer diagnosis and therapy.

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TWISTing stemness, inflammation and proliferation of epithelial ovarian cancer cells through MIR199A2/214.

http://www.wikidata.org/entity/Q33927452

description

2010 nî lūn-bûn

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2010 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2010 թվականի ապրիլին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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TWISTing stemness, inflammatio ...... er cells through MIR199A2/214.

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TWISTing stemness, inflammatio ...... er cells through MIR199A2/214.

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A B Alvero

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C Glackin

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G Mor

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R Chen

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P2860

Human Dermo-1 has attributes similar to twist in early bone development

Regulation of IKKbeta by miR-199a affects NF-kappaB activity in ovarian cancer cells

Twist-1 regulates the miR-199a/214 cluster during development

MicroRNA-dependent localization of targeted mRNAs to mammalian P-bodies

MicroRNAs: genomics, biogenesis, mechanism, and function

Twist, a master regulator of morphogenesis, plays an essential role in tumor metastasis

TWIST, a basic helix-loop-helix transcription factor, can regulate the human osteogenic lineage

Cancer stem cells--perspectives on current status and future directions: AACR Workshop on cancer stem cells

Molecular phenotyping of human ovarian cancer stem cells unravels the mechanisms for repair and chemoresistance

A twist code determines the onset of osteoblast differentiation

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10.1038/ONC.2010.111

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