Chronic upregulation of activated microglia immunoreactive for galectin-3/Mac-2 and nerve growth factor following diffuse axonal injury. - Wikidata - awgldk - TriplyDB

Chronic upregulation of activated microglia immunoreactive for galectin-3/Mac-2 and nerve growth factor following diffuse axonal injury.

Chronic upregulation of activated microglia immunoreactive for galectin-3/Mac-2 and nerve growth factor following diffuse axonal injury.

http://www.wikidata.org/entity/Q33939361

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Inflammatory response following diffuse axonal injury Endogenous IFN-β signaling exerts anti-inflammatory actions in experimentally induced focal cerebral ischemia Nuclear Factor Erythroid 2-Related Factor 2 (Nrf2) Mediates Neuroprotection in Traumatic Brain Injury at Least in Part by Inactivating Microglia Macrophage migration inhibitory factor promotes cell death and aggravates neurologic deficits after experimental stroke.Microglia processes associate with diffusely injured axons following mild traumatic brain injury in the micro pig.Traumatic white matter injury and glial activation: from basic science to clinics.Effects of combinatorial treatment with pituitary adenylate cyclase activating peptide and human mesenchymal stem cells on spinal cord tissue repair.Acute reduction of microglia does not alter axonal injury in a mouse model of repetitive concussive traumatic brain injury.Critical role of NADPH oxidase in neuronal oxidative damage and microglia activation following traumatic brain injury.RNA-sequencing reveals oligodendrocyte and neuronal transcripts in microglia relevant to central nervous system disease.Induction of a common microglia gene expression signature by aging and neurodegenerative conditions: a co-expression meta-analysis.Mapping the Connectome Following Traumatic Brain Injury.Galectin-1 deactivates classically activated microglia and protects from inflammation-induced neurodegeneration.Rapamycin protects neurons from brain contusion‑induced inflammatory reaction via modulation of microglial activation Galectin-3 released in response to traumatic brain injury acts as an alarmin orchestrating brain immune response and promoting neurodegeneration.Controlled cortical impact and craniotomy induce strikingly similar profiles of inflammatory gene expression, but with distinct kinetics.Morphological and genetic activation of microglia after diffuse traumatic brain injury in the rat.Microglia activation as a biomarker for traumatic brain injury Traumatic axonal injury in the mouse is accompanied by a dynamic inflammatory response, astroglial reactivity and complex behavioral changes Diffuse traumatic axonal injury in the optic nerve does not elicit retinal ganglion cell loss.Loss of galectin-3 decreases the number of immune cells in the subventricular zone and restores proliferation in a viral model of multiple sclerosis.Microglia/macrophage polarization dynamics in white matter after traumatic brain injury.Roles of programmed death protein 1/programmed death-ligand 1 in secondary brain injury after intracerebral hemorrhage in rats: selective modulation of microglia polarization to anti-inflammatory phenotype.Acute or Delayed Treatment with Anatabine Improves Spatial Memory and Reduces Pathological Sequelae at Late Time-Points after Repetitive Mild Traumatic Brain Injury.Tissue- and cell-specific localization of galectins, β-galactose-binding animal lectins, and their potential functions in health and disease.Role of Matricellular Proteins in Disorders of the Central Nervous System.Inflammation and vascular remodeling in the ventral hippocampus contributes to vulnerability to stress.Late exercise reduces neuroinflammation and cognitive dysfunction after traumatic brain injury.Functional diversity of microglia - how heterogeneous are they to begin with?Myelin loss and oligodendrocyte pathology in white matter tracts following traumatic brain injury in the rat.Diffuse Axonal Injury and Oxidative Stress: A Comprehensive Review.Depression following traumatic brain injury in mice is associated with down-regulation of hippocampal astrocyte glutamate transporters by thrombin.Peroxisome proliferator-activated receptor γ (PPARγ): A master gatekeeper in CNS injury and repair.Protective Role of Apocynin via Suppression of Neuronal Autophagy and TLR4/NF-κB Signaling Pathway in a Rat Model of Traumatic Brain Injury.Complex invasion pattern of the cerebral cortex bymicroglial cells during development of the mouse embryo.Microglial activation and traumatic brain injury.Diffuse traumatic axonal injury in mice induces complex behavioural alterations that are normalized by neutralization of interleukin-1β.Microglial Lectins in Health and Neurological Diseases.Xinnao Shutong Modulates the Neuronal Plasticity Through Regulation of Microglia/Macrophage Polarization Following Chronic Cerebral Hypoperfusion in Rats.The Importance of Inter-Species Variation in Traumatic Brain Injury-Induced Alterations of Microglial-Axonal Interactions

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Chronic upregulation of activated microglia immunoreactive for galectin-3/Mac-2 and nerve growth factor following diffuse axonal injury.

http://www.wikidata.org/entity/Q33939361

description

2010 nî lūn-bûn

@nan

2010 թուականի Մայիսին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի մայիսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

@zh-hk

2010年論文

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2010年論文

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2010年论文

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name

Chronic upregulation of activa ...... llowing diffuse axonal injury.

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Chronic upregulation of activa ...... llowing diffuse axonal injury.

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type

label

Chronic upregulation of activa ...... llowing diffuse axonal injury.

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Chronic upregulation of activa ...... llowing diffuse axonal injury.

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prefLabel

Chronic upregulation of activa ...... llowing diffuse axonal injury.

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Chronic upregulation of activa ...... llowing diffuse axonal injury.

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Journal of Neuroinflammation

P1476

Chronic upregulation of activa ...... llowing diffuse axonal injury.

@en

P2093

Charu Venkatesan

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Mark S Wainwright

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MaryAnn Chrzaszcz

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Nicole Choi

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P2860

Suppression of acute proinflammatory cytokine and chemokine upregulation by post-injury administration of a novel small molecule improves long-term neurologic outcome in a mouse model of traumatic brain injury

Epilepsy after head injury

Nerve growth factor signaling, neuroprotection, and neural repair.

Multifocal white matter ultrastructural abnormalities in mild traumatic brain injury with cognitive disability: a voxel-wise analysis of diffusion tensor imaging

Identification of two distinct macrophage subsets with divergent effects causing either neurotoxicity or regeneration in the injured mouse spinal cord

Microglia: a sensor for pathological events in the CNS

Diffusion tensor imaging in relation to cognitive and functional outcome of traumatic brain injury in children.

Microglia as mediators of inflammatory and degenerative diseases.

Intraventricular administration of BDNF increases the number of newly generated neurons in the adult olfactory bulb.

Peripheral nerve injury induces Schwann cells to express two macrophage phenotypes: phagocytosis and the galactose-specific lectin MAC-2.

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scholarly article

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10.1186/1742-2094-7-32

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7

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2010-05-27T00:00:00Z

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44635231

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1040683262

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20507613

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2891720

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