Increased levels of forkhead box M1B transcription factor in transgenic mouse hepatocytes prevent age-related proliferation defects in regenerating liver.
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Regulation of the transcription factor FOXM1c by Cyclin E/CDK2Chk2 mediates stabilization of the FoxM1 transcription factor to stimulate expression of DNA repair genesIncreased levels of the FoxM1 transcription factor accelerate development and progression of prostate carcinomas in both TRAMP and LADY transgenic miceFoxm1b transcription factor is essential for development of hepatocellular carcinomas and is negatively regulated by the p19ARF tumor suppressorThe Role of Forkhead Box Protein M1 in Breast Cancer Progression and Resistance to TherapyFOXM1, a typical proliferation-associated transcription factorPulmonary mastocytosis and enhanced lung inflammation in mice heterozygous null for the Foxf1 geneActivin A, p15INK4b signaling, and cell competition promote stem/progenitor cell repopulation of livers in aging ratsThe forkhead box m1 transcription factor is essential for embryonic development of pulmonary vasculatureForkhead box M1B transcriptional activity requires binding of Cdk-cyclin complexes for phosphorylation-dependent recruitment of p300/CBP coactivatorsPostnatal development, maturation and aging in the mouse cochlea and their effects on hair cell regeneration.Association between hepatocyte nuclear factor 6 (HNF-6) and FoxA2 DNA binding domains stimulates FoxA2 transcriptional activity but inhibits HNF-6 DNA binding.The Forkhead Box m1b transcription factor is essential for hepatocyte DNA replication and mitosis during mouse liver regenerationTransgenic expression of FoxM1 promotes endothelial repair following lung injury induced by polymicrobial sepsis in mice.Farnesoid X receptor alleviates age-related proliferation defects in regenerating mouse livers by activating forkhead box m1b transcriptionDespite its strong transactivation domain, transcription factor FOXM1c is kept almost inactive by two different inhibitory domains.Bile acid signaling and liver regeneration.FoxM1 overexpression promotes epithelial-mesenchymal transition and metastasis of hepatocellular carcinoma.Multiple faces of FoxM1 transcription factor: lessons from transgenic mouse models.Sox2 activates cell proliferation and differentiation in the respiratory epithelium.Foxm1 mediates LIF/Stat3-dependent self-renewal in mouse embryonic stem cells and is essential for the generation of induced pluripotent stem cellsLiver regeneration and aging: a current perspective.A cell-penetrating ARF peptide inhibitor of FoxM1 in mouse hepatocellular carcinoma treatment.Quinoline-based clioquinol and nitroxoline exhibit anticancer activity inducing FoxM1 inhibition in cholangiocarcinoma cells.A mouse model of accelerated liver aging caused by a defect in DNA repairGlobal gene expression profile of normal and regenerating liver in young and old mice.Characterization of gene expression profiles associated with glioma progression using oligonucleotide-based microarray analysis and real-time reverse transcription-polymerase chain reactionTransgenic expression of the human growth hormone minigene promotes pancreatic β-cell proliferationFoxm1 mediates cross talk between Kras/mitogen-activated protein kinase and canonical Wnt pathways during development of respiratory epitheliumOverexpression of FoxM1 is associated with tumor progression in patients with clear cell renal cell carcinoma.Induction of cell proliferation in old rat liver can reset certain gene expression levels characteristic of old liver to those associated with young liver.Transcriptional activation by growth hormone of HNF-6-regulated hepatic genes, a potential mechanism for improved liver repair during biliary injury in mice.Decreased expression of surfactant protein genes is associated with an increased expression of Forkhead box M1 gene in the fetal lung tissues of premature rabbits.Impaired liver regeneration in aged mice can be rescued by silencing Hippo core kinases MST1 and MST2.Oncogene FOXK1 enhances invasion of colorectal carcinoma by inducing epithelial-mesenchymal transition.The Fox genes in the liver: from organogenesis to functional integration.Insights into a Critical Role of the FOXO3a-FOXM1 Axis in DNA Damage Response and Genotoxic Drug Resistance.Rapid hepatocyte nuclear translocation of the Forkhead Box M1B (FoxM1B) transcription factor caused a transient increase in size of regenerating transgenic hepatocytes.On a FOX hunt: functions of FOX transcriptional regulators in bladder cancer.Silencing of FOXM1 transcription factor expression by adenovirus-mediated RNA interference inhibits human hepatocellular carcinoma growth.
P2860
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P2860
Increased levels of forkhead box M1B transcription factor in transgenic mouse hepatocytes prevent age-related proliferation defects in regenerating liver.
description
2001 nî lūn-bûn
@nan
2001 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
name
Increased levels of forkhead b ...... defects in regenerating liver.
@ast
Increased levels of forkhead b ...... defects in regenerating liver.
@en
type
label
Increased levels of forkhead b ...... defects in regenerating liver.
@ast
Increased levels of forkhead b ...... defects in regenerating liver.
@en
prefLabel
Increased levels of forkhead b ...... defects in regenerating liver.
@ast
Increased levels of forkhead b ...... defects in regenerating liver.
@en
P2093
P2860
P356
P1476
Increased levels of forkhead b ...... defects in regenerating liver.
@en
P2093
P2860
P304
11468-11473
P356
10.1073/PNAS.201360898
P407
P577
2001-09-01T00:00:00Z