Regulation of the pancreatic pro-endocrine gene neurogenin3. - Wikidata - awgldk - TriplyDB

Regulation of the pancreatic pro-endocrine gene neurogenin3.

Regulation of the pancreatic pro-endocrine gene neurogenin3.

http://www.wikidata.org/entity/Q33945269

about

Impaired glucose homeostasis in transgenic mice expressing the human transient neonatal diabetes mellitus locus, TNDM Basic helix-loop-helix transcription factors and enteroendocrine cell differentiation Repression by Groucho/TLE/Grg proteins: genomic site recruitment generates compacted chromatin in vitro and impairs activator binding in vivo Hes6 promotes cortical neurogenesis and inhibits Hes1 transcription repression activity by multiple mechanisms Genome-edited human stem cell-derived beta cells: a powerful tool for drilling down on type 2 diabetes GWAS biology Stem Cells in the Intestine: Possible Roles in Pathogenesis of Irritable Bowel Syndrome Notch Signaling in Pancreatic Development Transcription factor regulation of pancreatic organogenesis, differentiation and maturation Target gene analysis by microarrays and chromatin immunoprecipitation identifies HEY proteins as highly redundant bHLH repressors Identification of the bHLH factor Math6 as a novel component of the embryonic pancreas transcriptional network Early pancreatic development requires the vertebrate Suppressor of Hairless (RBPJ) in the PTF1 bHLH complex Hes1 is required for appropriate morphogenesis and differentiation during mouse thyroid gland development The Cdk4-E2f1 pathway regulates early pancreas development by targeting Pdx1+ progenitors and Ngn3+ endocrine precursors.Precommitment low-level Neurog3 expression defines a long-lived mitotic endocrine-biased progenitor pool that drives production of endocrine-committed cells.Precursor cells in mouse islets generate new beta-cells in vivo during aging and after islet injury.Homotypic clusters of transcription factor binding sites are a key component of human promoters and enhancers Lack of TCF2/vHNF1 in mice leads to pancreas agenesis Neurogenin3 is differentially required for endocrine cell fate specification in the intestinal and gastric epithelium.Neurogenin 3 Expressing Cells in the Human Exocrine Pancreas Have the Capacity for Endocrine Cell Fate.Historical perspective: beginnings of the beta-cell: current perspectives in beta-cell development The insulin gene in diabetes.A mouse model for monitoring islet cell genesis and developing therapies for diabetes Ectopic pancreas formation in Hes1 -knockout mice reveals plasticity of endodermal progenitors of the gut, bile duct, and pancreas.OVO homologue-like 1 (Ovol1) transcription factor: a novel target of neurogenin-3 in rodent pancreas.Sox9: a master regulator of the pancreatic program.Adenoviruses Expressing PDX-1, BETA2/NeuroD and MafA Induces the Transdifferentiation of Porcine Neonatal Pancreas Cell Clusters and Adult Pig Pancreatic Cells into Beta-Cells.Hnf1b controls pancreas morphogenesis and the generation of Ngn3+ endocrine progenitors The Krüppel-like zinc finger protein GLIS3 transactivates neurogenin 3 for proper fetal pancreatic islet differentiation in mice.Elevated Hedgehog/Gli signaling causes beta-cell dedifferentiation in mice.Musashi expression in β-cells coordinates insulin expression, apoptosis and proliferation in response to endoplasmic reticulum stress in diabetes.Differential levels of Neurod establish zebrafish endocrine pancreas cell fates.The Basic Helix-Loop-Helix Transcription Factor NEUROG3 Is Required for Development of the Human Endocrine Pancreas.The transcriptional co-repressor Grg3/Tle3 promotes pancreatic endocrine progenitor delamination and β-cell differentiation.Estradiol meets notch signaling in developing neurons.Sox9 coordinates a transcriptional network in pancreatic progenitor cells A Notch-dependent molecular circuitry initiates pancreatic endocrine and ductal cell differentiation Feedback control of growth, differentiation, and morphogenesis of pancreatic endocrine progenitors in an epithelial plexus niche.A distal intergenic region controls pancreatic endocrine differentiation by acting as a transcriptional enhancer and as a polycomb response element.Glis3 regulates neurogenin 3 expression in pancreatic β-cells and interacts with its activator, Hnf6.The potential for stem cell therapy in diabetes.

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P2860

Regulation of the pancreatic pro-endocrine gene neurogenin3.

http://www.wikidata.org/entity/Q33945269

description

2001 nî lūn-bûn

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2001 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2001 թվականի մայիսին հրատարակված գիտական հոդված

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2001年の論文

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2001年論文

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2001年論文

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2001年論文

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2001年論文

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2001年論文

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2001年论文

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name

Regulation of the pancreatic pro-endocrine gene neurogenin3.

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Regulation of the pancreatic pro-endocrine gene neurogenin3.

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type

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Regulation of the pancreatic pro-endocrine gene neurogenin3.

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Regulation of the pancreatic pro-endocrine gene neurogenin3.

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prefLabel

Regulation of the pancreatic pro-endocrine gene neurogenin3.

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Regulation of the pancreatic pro-endocrine gene neurogenin3.

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Regulation of the pancreatic pro-endocrine gene neurogenin3.

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D Scheel

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J C Lee

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J Wang

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M S German

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R G Mirmira

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5

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Hirotaka Watada

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