Caspase-1 dependent IL-1β secretion is critical for host defense in a mouse model of Chlamydia pneumoniae lung infection - Wikidata - awgldk - TriplyDB

Caspase-1 dependent IL-1β secretion is critical for host defense in a mouse model of Chlamydia pneumoniae lung infection

Caspase-1 dependent IL-1β secretion is critical for host defense in a mouse model of Chlamydia pneumoniae lung infection

http://www.wikidata.org/entity/Q33952490

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Investigating the role of nucleotide-binding oligomerization domain-like receptors in bacterial lung infection Detection of superoxide anion and hydrogen peroxide production by cellular NADPH oxidases Innate immune responses to Chlamydia pneumoniae infection: role of TLRs, NLRs, and the inflammasome The sst1 resistance locus regulates evasion of type I interferon signaling by Chlamydia pneumoniae as a disease tolerance mechanism Plasmacytoid dendritic cells play a role for effective innate immune responses during Chlamydia pneumoniae infection in mice Inflammasomes and Their Role in Innate Immunity of Sexually Transmitted Infections Integrating Inflammasome Signaling in Sexually Transmitted Infections Danger signals, inflammasomes, and the intricate intracellular lives of chlamydiae Initiation and perpetuation of NLRP3 inflammasome activation and assembly Housing conditions modulate the severity of Mycoplasma pulmonis infection in mice deficient in class A scavenger receptor Mitochondria in lung disease Regulation and Function of the Nucleotide Binding Domain Leucine-Rich Repeat-Containing Receptor, Pyrin Domain-Containing-3 Inflammasome in Lung Disease Assessing a potential role of host Pannexin 1 during Chlamydia trachomatis infection Oxidized mitochondrial DNA activates the NLRP3 inflammasome during apoptosis Alarmins, inflammasomes and immunity.Role of extracellular nucleotides in the immune response against intracellular bacteria and protozoan parasites Nontypeable Haemophilus Influenzae Infection Upregulates the NLRP3 Inflammasome and Leads to Caspase-1-Dependent Secretion of Interleukin-1β - A Possible Pathway of Exacerbations in COPD Inflammasomes.Significant role of IL-1 signaling, but limited role of inflammasome activation, in oviduct pathology during Chlamydia muridarum genital infection Mast cells play an important role in chlamydia pneumoniae lung infection by facilitating immune cell recruitment into the airway Murine Gammaherpesvirus 68 Pathogenesis Is Independent of Caspase-1 and Caspase-11 in Mice and Impairs Interleukin-1β Production upon Extrinsic Stimulation in Culture.Chlamydia muridarum infection of macrophages elicits bactericidal nitric oxide production via reactive oxygen species and cathepsin B.Chlamydia pneumoniae promotes dysfunction of pancreatic beta cells.Mouse Model of Respiratory Tract Infection Induced by Waddlia chondrophila.Impact of micro-environmental changes on respiratory tract infections with intracellular bacteria.Nod1, but not the ASC inflammasome, contributes to induction of IL-1β secretion in human trophoblasts after sensing of Chlamydia trachomatis Detection of a microbial metabolite by STING regulates inflammasome activation in response to Chlamydia trachomatis infection.Signaling through purinergic receptors for ATP induces human cutaneous innate and adaptive Th17 responses: implications in the pathogenesis of psoriasis Haemophilus ducreyi infection induces activation of the NLRP3 inflammasome in nonpolarized but not in polarized human macrophages.IRE1α mediates PKR activation in response to Chlamydia trachomatis infection Ugonin U stimulates NLRP3 inflammasome activation and enhances inflammasome-mediated pathogen clearance Quercetin Inhibits Inflammasome Activation by Interfering with ASC Oligomerization and Prevents Interleukin-1 Mediated Mouse Vasculitis.Host defenses against bacterial lower respiratory tract infection.Subversion of Cell-Autonomous Host Defense by Chlamydia Infection.Regulation where autophagy intersects the inflammasome.NOD-Like Receptors in Lung Diseases.NLRP3 inflammasome mediates interleukin-1β production in immune cells in response to Acinetobacter baumannii and contributes to pulmonary inflammation in mice.Neutrophils mediate Salmonella Typhimurium clearance through the GBP4 inflammasome-dependent production of prostaglandins.13th ERS Lung Science Conference. The most important take home messages: News from the Underground.The transcription factor GFI1 negatively regulates NLRP3 inflammasome activation in macrophages.

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Caspase-1 dependent IL-1β secretion is critical for host defense in a mouse model of Chlamydia pneumoniae lung infection

http://www.wikidata.org/entity/Q33952490

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2011 nî lūn-bûn

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2011 թուականի Յունիսին հրատարակուած գիտական յօդուած

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2011 թվականի հունիսին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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Caspase-1 dependent IL-1β secr ...... ydia pneumoniae lung infection

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Caspase-1 dependent IL-1β secr ...... ydia pneumoniae lung infection

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Caspase-1 dependent IL-1β secr ...... ydia pneumoniae lung infection

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Justin Karlin

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Kenichi Shimada

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Moshe Arditi

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Norika Chiba

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Shuang Chen

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V Krishnan Ramanujan

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P2860

Interleukin 1 is processed and released during apoptosis

Inflammasomes: too big to miss

Innate immune activation through Nalp3 inflammasome sensing of asbestos and silica

Mouse model of X-linked chronic granulomatous disease, an inherited defect in phagocyte superoxide production

A role for mitochondria in NLRP3 inflammasome activation

Altered cytokine export and apoptosis in mice deficient in interleukin-1 beta converting enzyme

Cryopyrin activates the inflammasome in response to toxins and ATP

Mice deficient in IL-1 beta-converting enzyme are defective in production of mature IL-1 beta and resistant to endotoxic shock

Molecular mechanisms of iNOS induction by IL-1 beta and IFN-gamma in rat aortic smooth muscle cells

Autophagy proteins regulate innate immune responses by inhibiting the release of mitochondrial DNA mediated by the NALP3 inflammasome

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10.1371/JOURNAL.PONE.0021477

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6

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6

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Timothy R Crother

Laurent Vergnes

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2011-06-23T00:00:00Z

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51469798

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3121765

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