Normal skeletal development of mice lacking matrilin 1: redundant function of matrilins in cartilage?
about
Matrilin-2 interacts with itself and with other extracellular matrix proteinsDistribution and evolution of von Willebrand/integrin A domains: widely dispersed domains with roles in cell adhesion and elsewhereDecreased chondrocyte proliferation and dysregulated apoptosis in the cartilage growth plate are key features of a murine model of epiphyseal dysplasia caused by a matn3 mutation.Articular cartilage and changes in arthritis: noncollagenous proteins and proteoglycans in the extracellular matrix of cartilageBeta1 integrins regulate chondrocyte rotation, G1 progression, and cytokinesisFunctional knockout of the matrilin-3 gene causes premature chondrocyte maturation to hypertrophy and increases bone mineral density and osteoarthritisGDF-5 deficiency in mice leads to disruption of tail tendon form and function.Tenomodulin is necessary for tenocyte proliferation and tendon maturationDisruption of the talin gene arrests mouse development at the gastrulation stage.Twinfilin-2a is dispensable for mouse developmentLoss of matrilin 1 does not exacerbate the skeletal phenotype in a mouse model of multiple epiphyseal dysplasia caused by a Matn3 V194D mutationJoint TGF-β type II receptor-expressing cells: ontogeny and characterization as joint progenitorsExtracellular matrix protein Matrilin-4 regulates stress-induced HSC proliferation via CXCR4Extracellular matrix and developing growth plate.Review of the Genetic Basis of Jaw Malformations.Structural and functional investigations of Matrilin-1 A-domains reveal insights into their role in cartilage ECM assembly.Chondromodulin I is dispensable during enchondral ossification and eye development.Zebrafish (Danio rerio) matrilins: shared and divergent characteristics with their mammalian counterpartsMatrilin-3 is dispensable for mouse skeletal growth and development.Molecular structure, processing, and tissue distribution of matrilin-4.Matrilin-1 is essential for zebrafish development by facilitating collagen II secretionLineage tracing using matrilin-1 gene expression reveals that articular chondrocytes exist as the joint interzone forms.The heterozygous Lemd3 +/GT mouse is not a murine model for osteopoikilosis in humans.Pericellular matrilins regulate activation of chondrocytes by cyclic load-induced matrix deformation.
P2860
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P2860
Normal skeletal development of mice lacking matrilin 1: redundant function of matrilins in cartilage?
description
1999 nî lūn-bûn
@nan
1999 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
1999 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
name
Normal skeletal development of ...... ion of matrilins in cartilage?
@ast
Normal skeletal development of ...... ion of matrilins in cartilage?
@en
type
label
Normal skeletal development of ...... ion of matrilins in cartilage?
@ast
Normal skeletal development of ...... ion of matrilins in cartilage?
@en
prefLabel
Normal skeletal development of ...... ion of matrilins in cartilage?
@ast
Normal skeletal development of ...... ion of matrilins in cartilage?
@en
P2093
P2860
P356
P1476
Normal skeletal development of ...... ion of matrilins in cartilage?
@en
P2093
E B Hunziker
J F Bateman
P2860
P304
P356
10.1128/MCB.19.11.7841
P407
P577
1999-11-01T00:00:00Z