Genetic and phenotypic studies of hypomorphic lin-12 mutants in Caenorhabditis elegans.
about
p24 proteins and quality control of LIN-12 and GLP-1 trafficking in Caenorhabditis elegansSuppressors of a lin-12 hypomorph define genes that interact with both lin-12 and glp-1 in Caenorhabditis elegansRas effector switching promotes divergent cell fates in C. elegans vulval patterning.SEL1L deficiency impairs growth and differentiation of pancreatic epithelial cells.sel-11 and cdc-42, two negative modulators of LIN-12/Notch activity in C. elegansThe Caenorhabditis elegans CDT-2 ubiquitin ligase is required for attenuation of EGFR signalling in vulva precursor cells.Caenorhabditis elegans SUR-5, a novel but conserved protein, negatively regulates LET-60 Ras activity during vulval induction.APH-1 is a multipass membrane protein essential for the Notch signaling pathway in Caenorhabditis elegans embryosSpatial and temporal patterns of lin-12 expression during C. elegans hermaphrodite development.The Caenorhabditis elegans sel-1 gene, a negative regulator of lin-12 and glp-1, encodes a predicted extracellular protein.New positive regulators of lin-12 activity in Caenorhabditis elegans include the BRE-5/Brainiac glycosphingolipid biosynthesis enzyme.Genetic analysis of Caenorhabditis elegans glp-1 mutants suggests receptor interaction or competition.hecd-1 modulates notch activity in Caenorhabditis elegans.Evidence for functional redundancy between C. elegans ADAM proteins SUP-17/Kuzbanian and ADM-4/TACESuppressors of the egg-laying defective phenotype of sel-12 presenilin mutants implicate the CoREST corepressor complex in LIN-12/Notch signaling in C. elegans.lin-35/Rb and the CoREST ortholog spr-1 coordinately regulate vulval morphogenesis and gonad development in C. elegansLIN-12/Notch signaling instructs postsynaptic muscle arm development by regulating UNC-40/DCC and MADD-2 in Caenorhabditis elegans.Assessment of normal and mutant human presenilin function in Caenorhabditis elegans.SKR-1, a homolog of Skp1 and a member of the SCF(SEL-10) complex, regulates sex-determination and LIN-12/Notch signaling in C. elegans.Sequence analyses of presenilin mutations linked to familial Alzheimer's disease.A conserved Six-Eya cassette acts downstream of Wnt signaling to direct non-myogenic versus myogenic fates in the C. elegans postembryonic mesoderm.Neurogenesis and Alzheimer's disease: at the crossroads.Loss of spr-5 bypasses the requirement for the C.elegans presenilin sel-12 by derepressing hop-1.The FoxF/FoxC factor LET-381 directly regulates both cell fate specification and cell differentiation in C. elegans mesoderm development.Canalization of C. elegans Vulva Induction against Anatomical VariabilityGene free methodology for cell fate dynamics during development.The DSL ligand APX-1 is required for normal ovulation in C. elegans.
P2860
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P2860
Genetic and phenotypic studies of hypomorphic lin-12 mutants in Caenorhabditis elegans.
description
1993 nî lūn-bûn
@nan
1993 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
1993 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
1993年の論文
@ja
1993年論文
@yue
1993年論文
@zh-hant
1993年論文
@zh-hk
1993年論文
@zh-mo
1993年論文
@zh-tw
1993年论文
@wuu
name
Genetic and phenotypic studies of hypomorphic lin-12 mutants in Caenorhabditis elegans.
@ast
Genetic and phenotypic studies of hypomorphic lin-12 mutants in Caenorhabditis elegans.
@en
type
label
Genetic and phenotypic studies of hypomorphic lin-12 mutants in Caenorhabditis elegans.
@ast
Genetic and phenotypic studies of hypomorphic lin-12 mutants in Caenorhabditis elegans.
@en
prefLabel
Genetic and phenotypic studies of hypomorphic lin-12 mutants in Caenorhabditis elegans.
@ast
Genetic and phenotypic studies of hypomorphic lin-12 mutants in Caenorhabditis elegans.
@en
P2860
P1433
P1476
Genetic and phenotypic studies of hypomorphic lin-12 mutants in Caenorhabditis elegans
@en
P2093
I Greenwald
M Sundaram
P2860
P304
P407
P577
1993-11-01T00:00:00Z