Transcriptional regulation of the TATA-binding protein by Ras cellular signaling
about
PTEN represses RNA Polymerase I transcription by disrupting the SL1 complexStimulation of Myc transactivation by the TATA binding protein in promoter-reporter assays.Enhanced RNA polymerase III-dependent transcription is required for oncogenic transformationDifferential gene expression in chemically induced mouse lung adenomas.Heterozygous disruption of the TATA-binding protein gene in DT40 cells causes reduced cdc25B phosphatase expression and delayed mitosis.Signal pathways which promote invasion and metastasis: critical and distinct contributions of extracellular signal-regulated kinase and Ral-specific guanine exchange factor pathwaysAlcohol induces RNA polymerase III-dependent transcription through c-Jun by co-regulating TATA-binding protein (TBP) and Brf1 expression.TBP is differentially regulated by c-Jun N-terminal kinase 1 (JNK1) and JNK2 through Elk-1, controlling c-Jun expression and cell proliferation.Doubly Spliced RNA of Hepatitis B Virus Suppresses Viral Transcription via TATA-Binding Protein and Induces Stress Granule Assembly.Therapeutic interventions to disrupt the protein synthetic machinery in melanomaPTEN represses RNA polymerase III-dependent transcription by targeting the TFIIIB complex.The JNKs differentially regulate RNA polymerase III transcription by coordinately modulating the expression of all TFIIIB subunitsAn internal segment (residues 58-119) of the hepatitis B virus X protein is sufficient to activate MAP kinase pathways in mouse liver.Baculovirus infection raises the level of TATA-binding protein that colocalizes with viral DNA replication sites.Increased expression of TATA-binding protein, the central transcription factor, can contribute to oncogenesis.The mitogen-activated protein (MAP) kinase ERK induces tRNA synthesis by phosphorylating TFIIIB.p53 represses RNA polymerase III transcription by targeting TBP and inhibiting promoter occupancy by TFIIIB.Epidermal growth factor receptor 1 (EGFR1) and its variant EGFRvIII regulate TATA-binding protein expression through distinct pathways.Epidermal growth factor enhances cellular TATA binding protein levels and induces RNA polymerase I- and III-dependent gene activity.Increased global transcription activity as a mechanism of replication stress in cancerElevated TATA-binding protein expression drives vascular endothelial growth factor expression in colon cancer.Association between an insertion/deletion polymorphism within 3'UTR of SGSM3 and risk of hepatocellular carcinoma.Transcribing malignancy: transcription-associated genomic instability in cancer.
P2860
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P2860
Transcriptional regulation of the TATA-binding protein by Ras cellular signaling
description
2000 nî lūn-bûn
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2000 թուականի Յուլիսին հրատարակուած գիտական յօդուած
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2000 թվականի հուլիսին հրատարակված գիտական հոդված
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2000年の論文
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2000年論文
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2000年論文
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2000年論文
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2000年論文
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2000年論文
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2000年论文
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Transcriptional regulation of the TATA-binding protein by Ras cellular signaling
@ast
Transcriptional regulation of the TATA-binding protein by Ras cellular signaling
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type
label
Transcriptional regulation of the TATA-binding protein by Ras cellular signaling
@ast
Transcriptional regulation of the TATA-binding protein by Ras cellular signaling
@en
prefLabel
Transcriptional regulation of the TATA-binding protein by Ras cellular signaling
@ast
Transcriptional regulation of the TATA-binding protein by Ras cellular signaling
@en
P2093
P2860
P1476
Transcriptional regulation of the TATA-binding protein by Ras cellular signaling
@en
P2093
D L Johnson
N Mandavia
S A Johnson
P2860
P304
P356
10.1128/MCB.20.14.5000-5009.2000
P407
P577
2000-07-01T00:00:00Z