about
Time course and cellular localization of SARS-CoV nucleoprotein and RNA in lungs from fatal cases of SARS.Influenza and SARS-coronavirus activating proteases TMPRSS2 and HAT are expressed at multiple sites in human respiratory and gastrointestinal tractsReduction of natural killer but not effector CD8 T lymphocytes in three consecutive cases of severe/lethal H1N1/09 influenza A virus infection.SARS-coronavirus open reading frame-9b suppresses innate immunity by targeting mitochondria and the MAVS/TRAF3/TRAF6 signalosome.MicroRNome analysis unravels the molecular basis of SARS infection in bronchoalveolar stem cells.MyD88 is required for protection from lethal infection with a mouse-adapted SARS-CoV.Pathogenicity of severe acute respiratory coronavirus deletion mutants in hACE-2 transgenic mice.Cellular immune responses to severe acute respiratory syndrome coronavirus (SARS-CoV) infection in senescent BALB/c mice: CD4+ T cells are important in control of SARS-CoV infection.Coronaviruses post-SARS: update on replication and pathogenesis.A systematic molecular pathology study of a laboratory confirmed H5N1 human caseChronic widespread musculoskeletal pain, fatigue, depression and disordered sleep in chronic post-SARS syndrome; a case-controlled studyPathology and pathogenesis of severe acute respiratory syndromeThe coronavirus nucleocapsid is a multifunctional protein.The SARS coronavirus E protein interacts with PALS1 and alters tight junction formation and epithelial morphogenesis.Immunopathogenesis of coronavirus infections: implications for SARS.An immunosuppressed Syrian golden hamster model for SARS-CoV infection.Interferon priming enables cells to partially overturn the SARS coronavirus-induced block in innate immune activationEpithelial cells lining salivary gland ducts are early target cells of severe acute respiratory syndrome coronavirus infection in the upper respiratory tracts of rhesus macaques.Unconventional use of LC3 by coronaviruses through the alleged subversion of the ERAD tuning pathway.Severe acute respiratory syndrome coronavirus infection of mice transgenic for the human Angiotensin-converting enzyme 2 virus receptor.Molecular pathology in the lungs of severe acute respiratory syndrome patients.Multi-Organ Damage in Human Dipeptidyl Peptidase 4 Transgenic Mice Infected with Middle East Respiratory Syndrome-Coronavirus.Severe neurologic syndrome associated with Middle East respiratory syndrome corona virus (MERS-CoV)Brain Invasion by Mouse Hepatitis Virus Depends on Impairment of Tight Junctions and Beta Interferon Production in Brain Microvascular Endothelial Cells.A novel subset of putative stem/progenitor CD34+Oct-4+ cells is the major target for SARS coronavirus in human lung.Middle East respiratory syndrome coronavirus infection: virus-host cell interactions and implications on pathogenesisCharacterization and Demonstration of the Value of a Lethal Mouse Model of Middle East Respiratory Syndrome Coronavirus Infection and DiseaseSARS-CoV replicates in primary human alveolar type II cell cultures but not in type I-like cells.Dysregulated Type I Interferon and Inflammatory Monocyte-Macrophage Responses Cause Lethal Pneumonia in SARS-CoV-Infected Mice.A review of genetic methods and models for analysis of coronavirus-induced severe pneumonitis.Severe acute respiratory syndrome coronavirus infection causes neuronal death in the absence of encephalitis in mice transgenic for human ACE2.The spike protein of the emerging betacoronavirus EMC uses a novel coronavirus receptor for entry, can be activated by TMPRSS2, and is targeted by neutralizing antibodies.Regulation of cell death during infection by the severe acute respiratory syndrome coronavirus and other coronaviruses.Spatiotemporal interplay of severe acute respiratory syndrome coronavirus and respiratory mucosal cells drives viral dissemination in rhesus macaques.Acute kidney injury in emerging, non-tropical infections.Angiotensin-converting enzyme 2 in the brain: properties and future directions.Differential virological and immunological outcome of severe acute respiratory syndrome coronavirus infection in susceptible and resistant transgenic mice expressing human angiotensin-converting enzyme 2.Spontaneous intracranial hemorrhage in a patient with Middle East respiratory syndrome corona virus.Studies of severe acute respiratory syndrome coronavirus pathology in human cases and animal models.Animal models in virus research: their utility and limitations.
P2860
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P2860
description
2005 nî lūn-bûn
@nan
2005 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
name
Multiple organ infection and the pathogenesis of SARS.
@ast
Multiple organ infection and the pathogenesis of SARS.
@en
type
label
Multiple organ infection and the pathogenesis of SARS.
@ast
Multiple organ infection and the pathogenesis of SARS.
@en
prefLabel
Multiple organ infection and the pathogenesis of SARS.
@ast
Multiple organ infection and the pathogenesis of SARS.
@en
P2093
P2860
P356
P1476
Multiple organ infection and the pathogenesis of SARS.
@en
P2093
Anthony S-Y Leong
Bingquan Wu
Danzhen Xu
Dongshia Gao
Encong Gong
Haohao Zhong
Hongquan Shao
Hui Zhuang
P2860
P304
P356
10.1084/JEM.20050828
P407
P577
2005-07-25T00:00:00Z