Akt-phosphorylated mitogen-activated kinase-activating death domain protein (MADD) inhibits TRAIL-induced apoptosis by blocking Fas-associated death domain (FADD) association with death receptor 4 - Wikidata - awgldk - TriplyDB

Akt-phosphorylated mitogen-activated kinase-activating death domain protein (MADD) inhibits TRAIL-induced apoptosis by blocking Fas-associated death domain (FADD) association with death receptor 4

Akt-phosphorylated mitogen-activated kinase-activating death domain protein (MADD) inhibits TRAIL-induced apoptosis by blocking Fas-associated death domain (FADD) association with death receptor 4

http://www.wikidata.org/entity/Q33991166

about

A monoclonal antibody (Mc178-Ab) targeted to the ecto-ATP synthase β-subunit-induced cell apoptosis via a mechanism involving the MAPKase and Akt pathways.Experimental meningococcal sepsis in congenic transgenic mice expressing human transferrin.Cholecystokinin octapeptide antagonizes apoptosis in human retinal pigment epithelial cells MADD knock-down enhances doxorubicin and TRAIL induced apoptosis in breast cancer cells Enhancing apoptosis in TRAIL-resistant cancer cells using fundamental response rules Knockdown of MADD and c-FLIP overcomes resistance to TRAIL-induced apoptosis in ovarian cancer cells.miR-150 Deficiency Protects against FAS-Induced Acute Liver Injury in Mice through Regulation of AKT Elucidating the identity of resistance mechanisms to prednisolone exposure in acute lymphoblastic leukemia cells through transcriptomic analysis: A computational approach.Control of mitochondrial activity by miRNAs.Vitronectin inhibits neutrophil apoptosis through activation of integrin-associated signaling pathways miR-223 Deficiency Protects against Fas-Induced Hepatocyte Apoptosis and Liver Injury through Targeting Insulin-Like Growth Factor 1 Receptor Down-modulation of expression, or dephosphorylation, of IG20/MADD in tumor necrosis factor-related apoptosis-inducing ligand-resistant thyroid cancer cells makes them susceptible to treatment with this ligand.Resistance to TRAIL in non-transformed cells is due to multiple redundant pathways.IG20/MADD plays a critical role in glucose-induced insulin secretion.Phosphoproteomic network analysis in the sea urchin Strongylocentrotus purpuratus reveals new candidates in egg activation.MADD is a downstream target of PTEN in triggering apoptosis.The mechanism underlying the effects of the cell surface ATP synthase on the regulation of intracellular acidification during acidosis.Functional genomics identified a novel protein tyrosine phosphatase receptor type F-mediated growth inhibition in hepatocarcinogenesis.Restoring TRAIL Induced Apoptosis Using Naturopathy. Hercules Joins Hand with Nature to Triumph Over Lernaean Hydra.Protective effect of RIP and c-FLIP in preventing liver cancer cell apoptosis induced by TRAIL.2-DG-Regulated RIP and c-FLIP Effect on Liver Cancer Cell Apoptosis Induced by TRAIL.

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Akt-phosphorylated mitogen-activated kinase-activating death domain protein (MADD) inhibits TRAIL-induced apoptosis by blocking Fas-associated death domain (FADD) association with death receptor 4

http://www.wikidata.org/entity/Q33991166

description

2010 nî lūn-bûn

@nan

2010 թուականի Մայիսին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի մայիսին հրատարակված գիտական հոդված

@hy

2010年の論文

@ja

2010年論文

@yue

2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

@wuu

name

Akt-phosphorylated mitogen-act ...... ociation with death receptor 4

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Akt-phosphorylated mitogen-act ...... ociation with death receptor 4

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type

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Akt-phosphorylated mitogen-act ...... ociation with death receptor 4

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Akt-phosphorylated mitogen-act ...... ociation with death receptor 4

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prefLabel

Akt-phosphorylated mitogen-act ...... ociation with death receptor 4

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Akt-phosphorylated mitogen-act ...... ociation with death receptor 4

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JBC.M110.105692

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Journal of Biological Chemistry

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Akt-phosphorylated mitogen-act ...... ociation with death receptor 4

@en

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Bellur S Prabhakar

http://www.w3.org/2001/XMLSchema#string

David Mordi

http://www.w3.org/2001/XMLSchema#string

Lakshmy Ganesh

http://www.w3.org/2001/XMLSchema#string

Nissim Hay

http://www.w3.org/2001/XMLSchema#string

Peifeng Li

http://www.w3.org/2001/XMLSchema#string

Prasad Kanteti

http://www.w3.org/2001/XMLSchema#string

Ryan Carr

http://www.w3.org/2001/XMLSchema#string

Shankar Jayarama

http://www.w3.org/2001/XMLSchema#string

P2860

Akt promotes cell survival by phosphorylating and inhibiting a Forkhead transcription factor

Contrasting effects of IG20 and its splice isoforms, MADD and DENN-SV, on tumor necrosis factor alpha-induced apoptosis and activation of caspase-8 and -3

IAP antagonists target cIAP1 to induce TNFalpha-dependent apoptosis

Antisense abrogation of DENN expression induces apoptosis of leukemia cells in vitro, causes tumor regression in vivo and alters the transcription of genes involved in apoptosis and the cell cycle

The receptor for the cytotoxic ligand TRAIL

DENN, a novel human gene differentially expressed in normal and neoplastic cells

MADD, a novel death domain protein that interacts with the type 1 tumor necrosis factor receptor and activates mitogen-activated protein kinase

X-linked IAP is a direct inhibitor of cell-death proteases

AKT/PKB signaling: navigating downstream

Akt phosphorylation of BAD couples survival signals to the cell-intrinsic death machinery

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22713-22722

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scholarly article

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10.1074/JBC.M110.105692

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P433

29

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285

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2010-05-18T00:00:00Z

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20484047

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phosphorylation

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2903385

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