Mitochondrial KATP channel inhibition blunts arrhythmia protection in ischemic exercised hearts. - Wikidata - awgldk - TriplyDB

Mitochondrial KATP channel inhibition blunts arrhythmia protection in ischemic exercised hearts.

Mitochondrial KATP channel inhibition blunts arrhythmia protection in ischemic exercised hearts.

http://www.wikidata.org/entity/Q33994857

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Why Does Exercise "Trigger" Adaptive Protective Responses in the Heart?Cardioprotective effects of nitrite during exercise.Delta opioid receptors: the link between exercise and cardioprotection.Molecular mechanisms in exercise-induced cardioprotection Decreased brain K(ATP) channel contributes to exacerbating ischemic brain injury and the failure of neuroprotection by sevoflurane post-conditioning in diabetic rats.CYP2J2 overexpression protects against arrhythmia susceptibility in cardiac hypertrophy ATP-Sensitive Potassium Channel Currents in Eccentrically Hypertrophied Cardiac Myocytes of Volume-Overloaded Rats.Mechanisms Involved in Exercise-Induced Cardioprotection: A Systematic Review.Exercise-induced protection against reperfusion arrhythmia involves stabilization of mitochondrial energetics Exercise and the cardiovascular system.Evaluation of arrhythmia scoring systems and exercise-induced cardioprotection Ischemia reperfusion injury, KATP channels, and exercise-induced cardioprotection against apoptosis κ-opioid receptor is involved in the cardioprotection induced by exercise training.Reactive oxygen species are not a required trigger for exercise-induced late preconditioning in the rat heart Exercise and cardiac preconditioning against ischemia reperfusion injury.Mechanisms of exercise-induced cardioprotection.Exercise-induced cardiac preconditioning: how exercise protects your achy-breaky heart.Exercise-induced improvements in myocardial antioxidant capacity: the antioxidant players and cardioprotection.Mitochondria in the middle: exercise preconditioning protection of striated muscle.Delta Opioid Receptors and Cardioprotection.Exercise: Teaching myocytes new tricks.Exercise-Induced Cardioprotection and the Therapeutic Potential of RIPC.Involvement of the δ-opioid receptor in exercise-induced cardioprotection.Redox-dependent increases in glutathione reductase and exercise preconditioning: role of NADPH oxidase and mitochondria.Cardiac Ischemia/Reperfusion Injury: The Beneficial Effects of Exercise.Short-term exercise preserves myocardial glutathione and decreases arrhythmias after thiol oxidation and ischemia in isolated rat hearts.Nandrolone decanoate negatively reverses the beneficial effects of exercise on cardiac muscle via sarcolemmal, but not mitochondrial K(ATP) channel.Interleukin-6 mediates exercise preconditioning against myocardial ischemia reperfusion injury.AT1 and aldosterone receptors blockade prevents the chronic effect of nandrolone on the exercise-induced cardioprotection in perfused rat heart subjected to ischemia and reperfusion.

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P2860

Mitochondrial KATP channel inhibition blunts arrhythmia protection in ischemic exercised hearts.

http://www.wikidata.org/entity/Q33994857

description

2010 nî lūn-bûn

@nan

2010 թուականի Ապրիլին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի ապրիլին հրատարակված գիտական հոդված

@hy

2010年の論文

@ja

2010年論文

@yue

2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

@wuu

name

Mitochondrial KATP channel inh ...... in ischemic exercised hearts.

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Mitochondrial KATP channel inh ...... in ischemic exercised hearts.

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type

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Mitochondrial KATP channel inh ...... in ischemic exercised hearts.

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Mitochondrial KATP channel inh ...... in ischemic exercised hearts.

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Mitochondrial KATP channel inh ...... in ischemic exercised hearts.

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Mitochondrial KATP channel inh ...... in ischemic exercised hearts.

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AJPHEART.01211.2009

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American Journal of Physiology - Heart and Circulatory Physiology

P1476

Mitochondrial KATP channel inh ...... in ischemic exercised hearts.

@en

P2093

Emily Hoyt

http://www.w3.org/2001/XMLSchema#string

J Megan Irwin

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Jenna Wrieden

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John C Quindry

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Lindsey Schreiber

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Peter Hosick

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P2860

Multiprotein complex containing succinate dehydrogenase confers mitochondrial ATP-sensitive K+ channel activity

A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding

Superoxide dismutase. An enzymic function for erythrocuprein (hemocuprein)

MnSOD antisense treatment and exercise-induced protection against arrhythmias

Catalase in vitro

Synchronization of chaotic early afterdepolarizations in the genesis of cardiac arrhythmias.

Synchronized whole cell oscillations in mitochondrial metabolism triggered by a local release of reactive oxygen species in cardiac myocytes.

KATP channels and 'border zone' arrhythmias: role of the repolarization dispersion between normal and ischaemic ventricular regions.

Preconditioning: a paradigm shift in the biology of myocardial ischemia

Evidence for mitochondrial K+ channels and their role in cardioprotection.

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H175-83

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10.1152/AJPHEART.01211.2009

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1

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20435852

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2904130

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