Concerted action of wild-type and mutant TNF receptors enhances inflammation in TNF receptor 1-associated periodic fever syndrome. - Wikidata - awgldk - TriplyDB

Concerted action of wild-type and mutant TNF receptors enhances inflammation in TNF receptor 1-associated periodic fever syndrome.

Concerted action of wild-type and mutant TNF receptors enhances inflammation in TNF receptor 1-associated periodic fever syndrome.

http://www.wikidata.org/entity/Q34006517

about

Targeting mitochondrial reactive oxygen species as novel therapy for inflammatory diseases and cancers Lighting the fires within: the cell biology of autoinflammatory diseases Beyond the NLRP3 inflammasome: autoinflammatory diseases reach adolescence Involvement of the same TNFR1 residue in mendelian and multifactorial inflammatory disorders.Role of tumour necrosis factor (TNF)-α and TNFRSF1A R92Q mutation in the pathogenesis of TNF receptor-associated periodic syndrome and multiple sclerosis.IL-1 blockade in autoinflammatory syndromes.Monogenic autoinflammatory diseases: new insights into clinical aspects and pathogenesis.The phenotype of TNF receptor-associated autoinflammatory syndrome (TRAPS) at presentation: a series of 158 cases from the Eurofever/EUROTRAPS international registry.Mitochondrial reactive oxygen species promote production of proinflammatory cytokines and are elevated in TNFR1-associated periodic syndrome (TRAPS)A pro-inflammatory signalome is constitutively activated by C33Y mutant TNF receptor 1 in TNF receptor-associated periodic syndrome (TRAPS)Long-term clinical profile of children with the low-penetrance R92Q mutation of the TNFRSF1A gene Interleukin-1 as a common denominator from autoinflammatory to autoimmune disorders: premises, perils, and perspectives Cell stress increases ATP release in NLRP3 inflammasome-mediated autoinflammatory diseases, resulting in cytokine imbalance Tumour necrosis factor receptor trafficking dysfunction opens the TRAPS door to pro-inflammatory cytokine secretion.Autoinflammation: translating mechanism to therapy.The novel S59P mutation in the TNFRSF1A gene identified in an adult onset TNF receptor associated periodic syndrome (TRAPS) constitutively activates NF-κB pathway Nanoparticulate STING agonists are potent lymph node-targeted vaccine adjuvants.The role of interleukin-1 beta in the pathophysiology of Schnitzler's syndrome.Molecular mechanisms in genetically defined autoinflammatory diseases: disorders of amplified danger signaling.Treating inflammation by blocking interleukin-1 in a broad spectrum of diseases.The Central Role of Anti-IL-1 Blockade in the Treatment of Monogenic and Multi-Factorial Autoinflammatory Diseases.Monogenic autoinflammatory syndromes: state of the art on genetic, clinical, and therapeutic issues Clinical relevance and functional consequences of the TNFRSF1A multiple sclerosis locus.Monogenic autoinflammatory diseases: disorders of amplified danger sensing and cytokine dysregulation.Canakinumab reverses overexpression of inflammatory response genes in tumour necrosis factor receptor-associated periodic syndrome.Caspase-8: not so silently deadly.Setting up TRAPS.Mitochondrial reactive oxygen species drive proinflammatory cytokine production.Genetics of monogenic autoinflammatory diseases: past successes, future challenges.Tumor necrosis factor receptor-associated periodic syndrome (TRAPS) in Japan: a review of the literature.Tumor necrosis factor receptor-associated periodic syndrome as a model linking autophagy and inflammation in protein aggregation diseases.Key facts and hot spots on tumor necrosis factor receptor-associated periodic syndrome.The labyrinth of autoinflammatory disorders: a snapshot on the activity of a third-level center in Italy.The inflammasome and IL-1β: implications for the treatment of inflammatory diseases.Protein misfolding and dysregulated protein homeostasis in autoinflammatory diseases and beyond.Canakinumab efficacy and long-term tocilizumab administration in tumor necrosis factor receptor-associated periodic syndrome (TRAPS).Redox stress unbalances the inflammatory cytokine network: role in autoinflammatory patients and healthy subjects.A systematic approach to autoinflammatory syndromes: a spelling booklet for the beginner.Canakinumab for the treatment of TNF-receptor associated periodic syndrome.Tumour necrosis factor receptor I blockade shows that TNF-dependent and TNF-independent mechanisms synergise in TNF receptor associated periodic syndrome.

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P2860

Concerted action of wild-type and mutant TNF receptors enhances inflammation in TNF receptor 1-associated periodic fever syndrome.

http://www.wikidata.org/entity/Q34006517

description

2010 nî lūn-bûn

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2010 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2010 թվականի մայիսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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Concerted action of wild-type ...... iated periodic fever syndrome.

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Concerted action of wild-type ...... iated periodic fever syndrome.

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Concerted action of wild-type ...... iated periodic fever syndrome.

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Concerted action of wild-type ...... iated periodic fever syndrome.

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Concerted action of wild-type ...... iated periodic fever syndrome.

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Concerted action of wild-type ...... iated periodic fever syndrome.

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Concerted action of wild-type ...... iated periodic fever syndrome.

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Concerted action of wild-type ...... iated periodic fever syndrome.

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Concerted action of wild-type ...... iated periodic fever syndrome.

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Concerted action of wild-type ...... iated periodic fever syndrome.

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Adrian A Lobito

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Adrianna J Jackson

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Anthony C Cruz

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Ariel C Bulua

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Daniel L Kastner

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Heiyoung Park

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Heleen D de Koning

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Hirsh Komarow

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Jae Jin Chae

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Rachel Ettinger

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P2860

A proposed classification of the immunological diseases

Endoplasmic reticulum stress activates cleavage of CREBH to induce a systemic inflammatory response

Induction of TNF receptor I-mediated apoptosis via two sequential signaling complexes

Horror autoinflammaticus: the molecular pathophysiology of autoinflammatory disease (*)

Gout-associated uric acid crystals activate the NALP3 inflammasome

TNF-R1 signaling: a beautiful pathway

Reactive oxygen species promote TNFalpha-induced death and sustained JNK activation by inhibiting MAP kinase phosphatases

A diagnostic score for molecular analysis of hereditary autoinflammatory syndromes with periodic fever in children

TNF receptor-deficient mice reveal divergent roles for p55 and p75 in several models of inflammation

Mice lacking the tumour necrosis factor receptor 1 are resistant to TNF-mediated toxicity but highly susceptible to infection by Listeria monocytogenes

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