Fas/Fas ligand system mediates epithelial injury, but not pulmonary host defenses, in response to inhaled bacteria - Wikidata - awgldk - TriplyDB

Fas/Fas ligand system mediates epithelial injury, but not pulmonary host defenses, in response to inhaled bacteria

Fas/Fas ligand system mediates epithelial injury, but not pulmonary host defenses, in response to inhaled bacteria

http://www.wikidata.org/entity/Q34009048

about

Selective induction of tumor necrosis receptor factor 6/decoy receptor 3 release by bacterial antigens in human monocytes and myeloid dendritic cells Apoptosis and the airway epithelium A rapid increase in macrophage-derived versican and hyaluronan in infectious lung disease Febrile-range hyperthermia augments lipopolysaccharide-induced lung injury by a mechanism of enhanced alveolar epithelial apoptosis Fas ligand-expressing lymphocytes enhance alveolar macrophage apoptosis in the resolution of acute pulmonary inflammation.Reprint of: A rapid increase in macrophage-derived versican and hyaluronan in infectious lung disease.Pneumococcal infections in humans are associated with increased apoptosis and trafficking of type 1 cytokine-producing T cells The Role of Fas-FasL Signaling Pathway in Induction of Apoptosis in Patients with Sulfur Mustard-Induced Chronic Bronchiolitis.Pathogenesis of indirect (secondary) acute lung injury.Death receptors mediate the adverse effects of febrile-range hyperthermia on the outcome of lipopolysaccharide-induced lung injury.Silencing of Fas, but not caspase-8, in lung epithelial cells ameliorates pulmonary apoptosis, inflammation, and neutrophil influx after hemorrhagic shock and sepsis.Acute lung injury: epidemiology, pathogenesis, and treatment.Fas-induced pulmonary apoptosis and inflammation during indirect acute lung injury.Anti-KC autoantibody:KC complexes cause severe lung inflammation in mice via IgG receptors Enhanced expression of Fas and FasL modulates apoptosis in the lungs of severe P. falciparum malaria patients with pulmonary edema.Apoptosis and epithelial injury in the lungs Stroke-induced immunodeficiency promotes spontaneous bacterial infections and is mediated by sympathetic activation reversal by poststroke T helper cell type 1-like immunostimulation.Group B Streptococcus Evades Host Immunity by Degrading Hyaluronan.Therapeutic accessibility of caspase-mediated cell death as a key pathomechanism in indirect acute lung injury A decoy receptor 3 analogue reduces localised defects in phagocyte function in pneumococcal pneumonia.Fas-ligand-induced apoptosis of respiratory epithelial cells causes disruption of postcanalicular alveolar development Sustained lipopolysaccharide-induced lung inflammation in mice is attenuated by functional deficiency of the Fas/Fas ligand system.Animal models of human pneumonia.Epithelial cell apoptosis and neutrophil recruitment in acute lung injury-a unifying hypothesis? What we have learned from small interfering RNAs.The Fas/FasL pathway impairs the alveolar fluid clearance in mouse lungs.Apoptotic response of Chang cells to infection with Pseudomonas aeruginosa strains PAK and PAO-I: molecular ordering of the apoptosis signaling cascade and role of type IV pili.Keratinocyte growth factor induces Akt kinase activity and inhibits Fas-mediated apoptosis in A549 lung epithelial cells.Inflammatory cells as a source of airspace extracellular superoxide dismutase after pulmonary injury.Cytokine-Ion Channel Interactions in Pulmonary Inflammation.Apoptotic cell death in rat lung following mustard gas inhalation.Enhancement of the endotoxin recognition pathway by ventilation with a large tidal volume in rabbits.TLR-4 pathway mediates the inflammatory response but not bacterial elimination in E. coli pneumonia.Exudate macrophages attenuate lung injury by the release of IL-1 receptor antagonist in gram-negative pneumonia.

P2860

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P2860

Fas/Fas ligand system mediates epithelial injury, but not pulmonary host defenses, in response to inhaled bacteria

http://www.wikidata.org/entity/Q34009048

description

2001 nî lūn-bûn

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2001 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2001 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2001年の論文

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2001年論文

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2001年論文

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2001年論文

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2001年論文

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IAI.69.9.5768-5776.2001

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Infection and Immunity

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Fas/Fas ligand system mediates ...... n response to inhaled bacteria

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C Vathanaprida

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C W Frevert

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G Matute-Bello

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J T Ruzinski

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V A Wong

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W C Liles

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P2860

Molecular cloning and expression of the Fas ligand, a novel member of the tumor necrosis factor family

Expression of three members of the TNF-R family of receptors (4-1BB, lymphotoxin-beta receptor, and Fas) in human lung

The polypeptide encoded by the cDNA for human cell surface antigen Fas can mediate apoptosis

Perforin-independent CD8(+) T-cell-mediated cytotoxicity of alveolar epithelial cells is preferentially mediated by tumor necrosis factor-alpha: relative insensitivity to Fas ligand.

Fas (CD95) induces alveolar epithelial cell apoptosis in vivo: implications for acute pulmonary inflammation

Simultaneous in situ hybridization and TUNEL to identify cells undergoing apoptosis.

Fas-induced apoptosis of alveolar epithelial cells requires ANG II generation and receptor interaction.

Sp3 regulates fas expression in lung epithelial cells

Receptor-dependent mechanisms of cell stimulation by bacterial endotoxin.

Regulation of apoptosis in neutrophils--Fas track to death?

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69

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11500454

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98694

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