Cytotoxic necrotizing factor type 1-positive Escherichia coli causes increased inflammation and tissue damage to the prostate in a rat prostatitis model.
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Prostate cancer and inflammation: the evidence.Loss of Nkx3.1 expression in bacterial prostatitis: a potential link between inflammation and neoplasia.Cyclomodulins in urosepsis strains of Escherichia coli.Cytotoxic necrotizing factor type 1 production by uropathogenic Escherichia coli modulates polymorphonuclear leukocyte function.Phylogenetic and pathotypic comparison of concurrent urine and rectal Escherichia coli isolates from men with febrile urinary tract infection.Preventing urinary tract infection: progress toward an effective Escherichia coli vaccine.Cytotoxic necrotizing factor type 1 delivered by outer membrane vesicles of uropathogenic Escherichia coli attenuates polymorphonuclear leukocyte antimicrobial activity and chemotaxis.Antibodies against hemolysin and cytotoxic necrotizing factor type 1 (CNF1) reduce bladder inflammation in a mouse model of urinary tract infection with toxigenic uropathogenic Escherichia coli.Escherichia coli α-hemolysin counteracts the anti-virulence innate immune response triggered by the Rho GTPase activating toxin CNF1 during bacteremia.sRNA-Mediated Regulation of P-Fimbriae Phase Variation in Uropathogenic Escherichia coli.Population structure of gut Escherichia coli and its role in development of extra-intestinal infections.Switching Rho GTPase activation into effective antibacterial defenses requires the caspase-1/IL-1beta signaling axis.Prostate carcinogenesis and inflammation: emerging insights.Two domains of cytotoxic necrotizing factor type 1 bind the cellular receptor, laminin receptor precursor protein.Experimental rodent models of prostatitis: limitations and potential.Hemolysin of uropathogenic Escherichia coli evokes extensive shedding of the uroepithelium and hemorrhage in bladder tissue within the first 24 hours after intraurethral inoculation of mice.Rho GTPase-activating bacterial toxins: from bacterial virulence regulation to eukaryotic cell biology.ZapA, a virulence factor in a rat model of Proteus mirabilis-induced acute and chronic prostatitisCytotoxic necrotizing factor type 1-neutralizing monoclonal antibody NG8 recognizes three amino acids in a C-terminal region of the toxin and reduces toxin binding to HEp-2 cells.The cytotoxic necrotizing factor of Yersinia pseudotuberculosis (CNFY) enhances inflammation and Yop delivery during infection by activation of Rho GTPases.Prostatic inflammation enhances basal-to-luminal differentiation and accelerates initiation of prostate cancer with a basal cell origin.Cytotoxic Necrotizing Factors (CNFs)-A Growing Toxin Family.The yin-yang driving urinary tract infection and how proteomics can enhance research, diagnostics, and treatment.Therapies in early development for the treatment of urinary tract inflammation.A mouse model of chronic prostatic inflammation using a human prostate cancer-derived isolate of Propionibacterium acnes.Cytotoxic necrotizing factor 1 enhances reactive oxygen species-dependent transcription and secretion of proinflammatory cytokines in human uroepithelial cells.Cytotoxic necrotizing factor 1 promotes prostate cancer progression through activating the Cdc42-PAK1 axis.Escherichia coli cytotoxic necrotizing factor 1 blocks cell cycle G2/M transition in uroepithelial cells.Proteinase-activated receptor-1 and immunomodulatory effects of a PAR1-activating peptide in a mouse model of prostatitis.Chronic bacterial inflammation induces prostatic intraepithelial neoplasia in mouse prostate.A single amino acid substitution in the enzymatic domain of cytotoxic necrotizing factor type 1 of Escherichia coli alters the tissue culture phenotype to that of the dermonecrotic toxin of Bordetella spp.Context-dependent Requirements for FimH and Other Canonical Virulence Factors in Gut Colonization by Extraintestinal Pathogenic Escherichia coli.The Bacterial Toxin CNF1 Induces Activation and Maturation of Human Monocyte-Derived Dendritic Cells.Cytotoxic Necrotizing Factor 1 Downregulates CD36 Transcription in Macrophages to Induce Inflammation During Acute Urinary Tract Infections
P2860
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P2860
Cytotoxic necrotizing factor type 1-positive Escherichia coli causes increased inflammation and tissue damage to the prostate in a rat prostatitis model.
description
2001 nî lūn-bûn
@nan
2001 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
name
Cytotoxic necrotizing factor t ...... te in a rat prostatitis model.
@ast
Cytotoxic necrotizing factor t ...... te in a rat prostatitis model.
@en
Cytotoxic necrotizing factor t ...... te in a rat prostatitis model.
@nl
type
label
Cytotoxic necrotizing factor t ...... te in a rat prostatitis model.
@ast
Cytotoxic necrotizing factor t ...... te in a rat prostatitis model.
@en
Cytotoxic necrotizing factor t ...... te in a rat prostatitis model.
@nl
prefLabel
Cytotoxic necrotizing factor t ...... te in a rat prostatitis model.
@ast
Cytotoxic necrotizing factor t ...... te in a rat prostatitis model.
@en
Cytotoxic necrotizing factor t ...... te in a rat prostatitis model.
@nl
P2093
P2860
P1476
Cytotoxic necrotizing factor t ...... te in a rat prostatitis model.
@en
P2093
P2860
P304
P356
10.1128/IAI.69.10.6515-6519.2001
P407
P577
2001-10-01T00:00:00Z