Beta-amyloid peptide blocks the fast-inactivating K+ current in rat hippocampal neurons.
about
Exploring the mechanism of beta-amyloid toxicity attenuation by multivalent sialic acid polymers through the use of mathematical models.Automated parallel recordings of topologically identified single ion channelsComputational study of hippocampal-septal theta rhythm changes due to β-amyloid-altered ionic channelsEffect of synthetic aβ peptide oligomers and fluorinated solvents on Kv1.3 channel properties and membrane conductance.Changes in the physiology of CA1 hippocampal pyramidal neurons in preplaque CRND8 mice.Linking aβ42-induced hyperexcitability to neurodegeneration, learning and motor deficits, and a shorter lifespan in an Alzheimer's modelTau facilitates Aβ-induced loss of mitochondrial membrane potential independent of cytosolic calcium fluxes in mouse cortical neuronsProgressive effect of beta amyloid peptides accumulation on CA1 pyramidal neurons: a model study suggesting possible treatmentsEffect of beta-amyloid block of the fast-inactivating K+ channel on intracellular Ca2+ and excitability in a modeled neuron.Alzheimer's disease-associated peptide Aβ42 mobilizes ER Ca(2+) via InsP3R-dependent and -independent mechanisms.Noradrenaline activation of neurotrophic pathways protects against neuronal amyloid toxicity.Amyloid-beta1-42 reduces neuronal excitability in mouse dentate gyrus.Cortical neurons transgenic for human Abeta40 or Abeta42 have similar vulnerability to apoptosis despite their different amyloidogenic properties.Suppression of neuronal and cardiac transient outward currents by viral gene transfer of dominant-negative Kv4.2 constructs.Acceleration of amyloid fibril formation by specific binding of Abeta-(1-40) peptide to ganglioside-containing membrane vesicles.Development of a novel diffusion-based method to estimate the size of the aggregated Abeta species responsible for neurotoxicity.Beta-amyloid induced changes in A-type K⁺ current can alter hippocampo-septal network dynamics.Capturing intracellular Ca2+ dynamics in computational models of neurodegenerative diseases.Resveratrol Attenuates Aβ-Induced Early Hippocampal Neuron Excitability Impairment via Recovery of Function of Potassium Channels.Evolution of adaptive neural networks: the role of voltage-dependent K+ channels.
P2860
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P2860
Beta-amyloid peptide blocks the fast-inactivating K+ current in rat hippocampal neurons.
description
1996 nî lūn-bûn
@nan
1996 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
1996 թվականի հունվարին հրատարակված գիտական հոդված
@hy
1996年の論文
@ja
1996年論文
@yue
1996年論文
@zh-hant
1996年論文
@zh-hk
1996年論文
@zh-mo
1996年論文
@zh-tw
1996年论文
@wuu
name
Beta-amyloid peptide blocks the fast-inactivating K+ current in rat hippocampal neurons.
@ast
Beta-amyloid peptide blocks the fast-inactivating K+ current in rat hippocampal neurons.
@en
Beta-amyloid peptide blocks the fast-inactivating K+ current in rat hippocampal neurons.
@nl
type
label
Beta-amyloid peptide blocks the fast-inactivating K+ current in rat hippocampal neurons.
@ast
Beta-amyloid peptide blocks the fast-inactivating K+ current in rat hippocampal neurons.
@en
Beta-amyloid peptide blocks the fast-inactivating K+ current in rat hippocampal neurons.
@nl
prefLabel
Beta-amyloid peptide blocks the fast-inactivating K+ current in rat hippocampal neurons.
@ast
Beta-amyloid peptide blocks the fast-inactivating K+ current in rat hippocampal neurons.
@en
Beta-amyloid peptide blocks the fast-inactivating K+ current in rat hippocampal neurons.
@nl
P2093
P2860
P1433
P1476
Beta-amyloid peptide blocks the fast-inactivating K+ current in rat hippocampal neurons.
@en
P2093
P2860
P304
P356
10.1016/S0006-3495(96)79570-X
P407
P577
1996-01-01T00:00:00Z