Suppression of neuronal and cardiac transient outward currents by viral gene transfer of dominant-negative Kv4.2 constructs.
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Modulation of Kv4.2 channel expression and gating by dipeptidyl peptidase 10 (DPP10)I SA channel complexes include four subunits each of DPP6 and Kv4.2Multiprotein assembly of Kv4.2, KChIP3 and DPP10 produces ternary channel complexes with ISA-like propertiesA role for frequenin, a Ca2+-binding protein, as a regulator of Kv4 K+-currentsNovel functional properties of Ca(2+) channel beta subunits revealed by their expression in adult rat heart cellsEffects of phrixotoxins on the Kv4 family of potassium channels and implications for the role of Ito1 in cardiac electrogenesisCardiovascular Action of Insulin in Health and Disease: Endothelial L-Arginine Transport and Cardiac Voltage-Dependent Potassium ChannelsCloning and expression of the human kv4.3 potassium channelPalmitoylation of KChIP splicing variants is required for efficient cell surface expression of Kv4.3 channelsFunctional consequences of the arrhythmogenic G306R KvLQT1 K+ channel mutant probed by viral gene transfer in cardiomyocytesKrüppel-like factor 4 (gut-enriched Krüppel-like factor) inhibits cell proliferation by blocking G1/S progression of the cell cycleEvidence for the presence of a novel Kv4-mediated A-type K(+) channel-modifying factorKv4 potassium channel subunits control action potential repolarization and frequency-dependent broadening in rat hippocampal CA1 pyramidal neuronesInhibitory effects of polyunsaturated fatty acids on Kv4/KChIP potassium channels.Spinocerebellar ataxia type 19/22 mutations alter heterocomplex Kv4.3 channel function and gating in a dominant mannerRegulation of voltage-gated K+ channel expression in the developing mammalian myocardium.Molecular determinants of cardiac transient outward potassium current (I(to)) expression and regulationMolecular basis of functional voltage-gated K+ channel diversity in the mammalian myocardium.Membrane potential depolarization as a triggering mechanism for Vpu-mediated HIV-1 release.Isoform-specific lidocaine block of sodium channels explained by differences in gatingKv4 channels exhibit modulation of closed-state inactivation in inside-out patches.Expression of inwardly rectifying potassium channels by an inducible adenoviral vector reduced the neuronal hyperexcitability and hyperalgesia produced by chronic compression of the spinal ganglion.Enterocyte differentiation marker intestinal alkaline phosphatase is a target gene of the gut-enriched Kruppel-like factor.Molecular dissection of cardiac repolarization by in vivo Kv4.3 gene transferA revised view of cardiac sodium channel "blockade" in the long-QT syndromeMolecular dissection of the inward rectifier potassium current (IK1) in rabbit cardiomyocytes: evidence for heteromeric co-assembly of Kir2.1 and Kir2.2.DPP10 splice variants are localized in distinct neuronal populations and act to differentially regulate the inactivation properties of Kv4-based ion channelsWhen the DREAM is gone: from basic science to future prospectives in pain management and beyond.Functional blockade of the voltage-gated potassium channel Kv1.3 mediates reversion of T effector to central memory lymphocytes through SMAD3/p21cip1 signaling.Charged residues between the selectivity filter and S6 segments contribute to the permeation phenotype of the sodium channel.Distinct gene-specific mechanisms of arrhythmia revealed by cardiac gene transfer of two long QT disease genes, HERG and KCNE1.KChIP2 genotype dependence of transient outward current (Ito) properties in cardiomyocytes isolated from male and female mice.Inactivation gating of Kv4 potassium channels: molecular interactions involving the inner vestibule of the pore.Four kinetically distinct depolarization-activated K+ currents in adult mouse ventricular myocytes.A structural rearrangement in the sodium channel pore linked to slow inactivation and use dependence.Extracellular sodium interacts with the HERG channel at an outer pore site.Kv4.3 is not required for the generation of functional Ito,f channels in adult mouse ventricles.A role for DPPX modulating external TEA sensitivity of Kv4 channels.Molecular Basis of Functional Myocardial Potassium Channel Diversity.Recurrent intrauterine fetal loss due to near absence of HERG: clinical and functional characterization of a homozygous nonsense HERG Q1070X mutation.
P2860
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P2860
Suppression of neuronal and cardiac transient outward currents by viral gene transfer of dominant-negative Kv4.2 constructs.
description
1997 nî lūn-bûn
@nan
1997年の論文
@ja
1997年学术文章
@wuu
1997年学术文章
@zh
1997年学术文章
@zh-cn
1997年学术文章
@zh-hans
1997年学术文章
@zh-my
1997年学术文章
@zh-sg
1997年學術文章
@yue
1997年學術文章
@zh-hant
name
Suppression of neuronal and ca ...... ant-negative Kv4.2 constructs.
@en
Suppression of neuronal and ca ...... ant-negative Kv4.2 constructs.
@nl
type
label
Suppression of neuronal and ca ...... ant-negative Kv4.2 constructs.
@en
Suppression of neuronal and ca ...... ant-negative Kv4.2 constructs.
@nl
prefLabel
Suppression of neuronal and ca ...... ant-negative Kv4.2 constructs.
@en
Suppression of neuronal and ca ...... ant-negative Kv4.2 constructs.
@nl
P2093
P2860
P356
P1476
Suppression of neuronal and ca ...... ant-negative Kv4.2 constructs.
@en
P2093
P2860
P304
31598-31603
P356
10.1074/JBC.272.50.31598
P407
P577
1997-12-01T00:00:00Z