Acquisition of chemoresistance in gliomas is associated with increased mitochondrial coupling and decreased ROS production.
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Generation of reactive oxygen species by polyenylpyrroles derivatives causes DNA damage leading to G2/M arrest and apoptosis in human oral squamous cell carcinoma cellsExogenous IGFBP-2 promotes proliferation, invasion, and chemoresistance to temozolomide in glioma cells via the integrin β1-ERK pathwayPreferential transfer of mitochondria from endothelial to cancer cells through tunneling nanotubes modulates chemoresistance.Repositioning chlorpromazine for treating chemoresistant glioma through the inhibition of cytochrome c oxidase bearing the COX4-1 regulatory subunit.Prognostic relevance of cytochrome C oxidase in primary glioblastoma multiforme.Nuclear-encoded cytochrome c oxidase subunit 4 regulates BMI1 expression and determines proliferative capacity of high-grade gliomasSuccessful Treatment of Intracranial Glioblastoma Xenografts With a Monoamine Oxidase B-Activated Pro-Drug.Glutathione depletion sensitizes cisplatin- and temozolomide-resistant glioma cells in vitro and in vivo.Aldehyde dehydrogenase 1A1--a new mediator of resistance to temozolomide in glioblastoma.Tumor Suppressor WWOX and p53 Alterations and Drug Resistance in Glioblastomas.Temozolomide promotes genomic and phenotypic changes in glioblastoma cells.Estrogen Enhances the Expression of the Multidrug Transporter Gene ABCG2-Increasing Drug Resistance of Breast Cancer Cells through Estrogen Receptors.Oncolytic viral therapy: targeting cancer stem cellsCurrent evidence of temozolomide and bevacizumab in treatment of gliomas.Detoxification of oxidative stress in glioma stem cells: mechanism, clinical relevance, and therapeutic development.Inhibitor of Nicotinamide Phosphoribosyltransferase Sensitizes Glioblastoma Cells to Temozolomide via Activating ROS/JNK Signaling Pathway.Glutathione-mediated antioxidant response and aerobic metabolism: two crucial factors involved in determining the multi-drug resistance of high-risk neuroblastoma.Identification of Small Molecule Inhibitors of Human Cytochrome c Oxidase That Target Chemoresistant Glioma Cells.Low-Dose DMC Significantly Enhances the Effect of TMZ on Glioma Cells by Targeting Multiple Signaling Pathways Both In Vivo and In Vitro.PDGF-BB protects mitochondria from rotenone in T98G cells.Apoptosis-inducing effects of Melissa officinalis L. essential oil in glioblastoma multiforme cells.Delivery of Functional Anti-miR-9 by Mesenchymal Stem Cell-derived Exosomes to Glioblastoma Multiforme Cells Conferred Chemosensitivity.Demethoxycurcumin was prior to temozolomide on inhibiting proliferation and induced apoptosis of glioblastoma stem cells.PGC1α induced by reactive oxygen species contributes to chemoresistance of ovarian cancer cells.Downregulation of cytochrome c oxidase 1 induced radioresistance in esophageal squamous cell carcinomaThe molecular mechanisms of chemoresistance in cancers.Metabolic shift toward oxidative phosphorylation in docetaxel resistant prostate cancer cellsThe different expression of TRPM7 and MagT1 impacts on the proliferation of colon carcinoma cells sensitive or resistant to doxorubicinABCG2 downregulation in glioma stem cells enhances the therapeutic efficacy of demethoxycurcumin.Inhibition of HIF1α and PDK Induces Cell Death of Glioblastoma Multiforme.Response to commentary by Champ and Klement: Is a ketogenic diet the solution for the hyperglycemia problem in glioblastoma therapy?Glutathione reductase mediates drug resistance in glioblastoma cells by regulating redox homeostasis.Blockade of Neuroglobin Reduces Protection of Conditioned Medium from Human Mesenchymal Stem Cells in Human Astrocyte Model (T98G) Under a Scratch Assay.Bevacizumab is superior to Temozolomide in causing mitochondrial dysfunction in human brain tumors.Decreasing GSH and increasing ROS in chemosensitivity gliomas with IDH1 mutation.Addition of carbonic anhydrase 9 inhibitor SLC-0111 to temozolomide treatment delays glioblastoma growth in vivo.Function of neuronal nitric oxide synthase enzyme in temozolomide-induced damage of astrocytic tumor cells.Coordinated autophagy modulation overcomes glioblastoma chemoresistance through disruption of mitochondrial bioenergetics.Comparison of Pre- and Post-translational Expressions of and Genes in Colorectal Adenoma-Carcinoma TissuesIGFBP6 controls the expansion of chemoresistant glioblastoma through paracrine IGF2/IGF-1R signaling
P2860
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P2860
Acquisition of chemoresistance in gliomas is associated with increased mitochondrial coupling and decreased ROS production.
description
2011 nî lūn-bûn
@nan
2011 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Acquisition of chemoresistance ...... and decreased ROS production.
@ast
Acquisition of chemoresistance ...... and decreased ROS production.
@en
Acquisition of chemoresistance ...... and decreased ROS production.
@nl
type
label
Acquisition of chemoresistance ...... and decreased ROS production.
@ast
Acquisition of chemoresistance ...... and decreased ROS production.
@en
Acquisition of chemoresistance ...... and decreased ROS production.
@nl
prefLabel
Acquisition of chemoresistance ...... and decreased ROS production.
@ast
Acquisition of chemoresistance ...... and decreased ROS production.
@en
Acquisition of chemoresistance ...... and decreased ROS production.
@nl
P2093
P2860
P1433
P1476
Acquisition of chemoresistance ...... and decreased ROS production.
@en
P2093
Claudia R Oliva
Corinne E Griguer
Douglas R Moellering
G Yancey Gillespie
P2860
P304
P356
10.1371/JOURNAL.PONE.0024665
P407
P577
2011-09-09T00:00:00Z