AID stabilizes stem-cell phenotype by removing epigenetic memory of pluripotency genes.
about
Epigenetic Modulators of Monocytic Function: Implication for Steady State and Disease in the CNSEpigenetic Codes Programing Class Switch RecombinationThe epigenetic basis of diffuse large B-cell lymphomaEtoposide induces nuclear re-localisation of AIDProcessive DNA demethylation via DNA deaminase-induced lesion resolutionToward pluripotency by reprogramming: mechanisms and applicationActive demethylation in mouse zygotes involves cytosine deamination and base excision repairGeneration and repair of AID-initiated DNA lesions in B lymphocytes.TET enzymes, TDG and the dynamics of DNA demethylation.Mutations in human AID differentially affect its ability to deaminate cytidine and 5-methylcytidine in ssDNA substrates in vitroMolecular control of induced pluripotency.Regulation of immunoglobulin class-switch recombination: choreography of noncoding transcription, targeted DNA deamination, and long-range DNA repair.An interplay of the base excision repair and mismatch repair pathways in active DNA demethylation.Cancer-like epigenetic derangements of human pluripotent stem cells and their impact on applications in regeneration and repair.No evidence for AID/MBD4-coupled DNA demethylation in zebrafish embryos.Small molecules facilitate rapid and synchronous iPSC generation.Generation and characterization of induced pluripotent stem cells from Aid-deficient mice.Enhanced reduced representation bisulfite sequencing for assessment of DNA methylation at base pair resolution.Cell Cycle Regulates Nuclear Stability of AID and Determines the Cellular Response to AID.DNA Methylation Dynamics of Germinal Center B Cells Are Mediated by AIDMechanisms underlying the formation of induced pluripotent stem cells.TET family proteins: oxidation activity, interacting molecules, and functions in diseases.Epigenetic reprogramming of fallopian tube fimbriae in BRCA mutation carriers defines early ovarian cancer evolution.Defining chromosomal translocation risks in cancer.Activation-induced cytidine deaminase (AID) is necessary for the epithelial-mesenchymal transition in mammary epithelial cells.Enrichment of rare variants in population isolates: single AICDA mutation responsible for hyper-IgM syndrome type 2 in FinlandReversing DNA methylation: mechanisms, genomics, and biological functions.Gender Differences in Global but Not Targeted Demethylation in iPSC Reprogramming.AIDing Chromatin and Transcription-Coupled Orchestration of Immunoglobulin Class-Switch Recombination.Aid is a key regulator of myeloid/erythroid differentiation and DNA methylation in hematopoietic stem/progenitor cellsThe epigenome in pluripotency and differentiation.AID and APOBEC deaminases: balancing DNA damage in epigenetics and immunity.Epigenetic function of activation-induced cytidine deaminase and its link to lymphomagenesisTraumatic Spinal Cord Injury-Repair and Regeneration.Activation-induced cytidine deaminase selectively catalyzed active DNA demethylation in pluripotency gene and improved cell reprogramming in bovine SCNT embryo.Functions and Malfunctions of Mammalian DNA-Cytosine DeaminasesDNA demethylation pathways: Additional players and regulators.Skeletal Muscle Cell Induction from Pluripotent Stem Cells.Family-Wide Comparative Analysis of Cytidine and Methylcytidine Deamination by Eleven Human APOBEC Proteins.Grand challenges in the field of stem cell research.
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P2860
AID stabilizes stem-cell phenotype by removing epigenetic memory of pluripotency genes.
description
2013 nî lūn-bûn
@nan
2013 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2013 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
name
AID stabilizes stem-cell phenotype by removing epigenetic memory of pluripotency genes.
@ast
AID stabilizes stem-cell phenotype by removing epigenetic memory of pluripotency genes.
@en
AID stabilizes stem-cell phenotype by removing epigenetic memory of pluripotency genes.
@nl
type
label
AID stabilizes stem-cell phenotype by removing epigenetic memory of pluripotency genes.
@ast
AID stabilizes stem-cell phenotype by removing epigenetic memory of pluripotency genes.
@en
AID stabilizes stem-cell phenotype by removing epigenetic memory of pluripotency genes.
@nl
prefLabel
AID stabilizes stem-cell phenotype by removing epigenetic memory of pluripotency genes.
@ast
AID stabilizes stem-cell phenotype by removing epigenetic memory of pluripotency genes.
@en
AID stabilizes stem-cell phenotype by removing epigenetic memory of pluripotency genes.
@nl
P2093
P2860
P50
P356
P1433
P1476
AID stabilizes stem-cell phenotype by removing epigenetic memory of pluripotency genes
@en
P2093
Ali A Zarrin
Jayanta Chaudhuri
Lauren DiMenna
Olivier Elemento
Philipp Franck
Ritu Kumar
Ting-Chun Liu
Todd Evans
P2860
P2888
P356
10.1038/NATURE12299
P407
P577
2013-06-26T00:00:00Z
P5875
P6179
1037741286