Increased work in cardiac trabeculae causes decreased mitochondrial NADH fluorescence followed by slow recovery.
about
Functional response of the isolated, perfused normoxic heart to pyruvate dehydrogenase activation by dichloroacetate and pyruvateMetabolic imaging of the organ of corti--a window on cochlea bioenergetics.Lrrc10 is required for early heart development and function in zebrafishChanges in [Na(+)](i), compartmental [Ca(2+)], and NADH with dysfunction after global ischemia in intact hearts.Physical coupling supports the local Ca2+ transfer between sarcoplasmic reticulum subdomains and the mitochondria in heart muscle.Analysis of the mechanisms of mitochondrial NADH regulation in cardiac trabeculae.Simultaneous measurements of mitochondrial NADH and Ca(2+) during increased work in intact rat heart trabeculaeATP synthesis and storage.Age affects the contraction-induced mitochondrial redox response in skeletal muscle.Adding ROS quenchers to cold K+ cardioplegia reduces superoxide emission during 2-hour global cold cardiac ischemia.Ranolazine reduces Ca2+ overload and oxidative stress and improves mitochondrial integrity to protect against ischemia reperfusion injury in isolated heartsMeasuring mitochondrial function in intact cardiac myocytes.Surface fluorescence studies of tissue mitochondrial redox state in isolated perfused rat lungsExcitation-contraction coupling and mitochondrial energetics.Extra-matrix Mg2+ limits Ca2+ uptake and modulates Ca2+ uptake-independent respiration and redox state in cardiac isolated mitochondria.Enhanced Na+/H+ exchange during ischemia and reperfusion impairs mitochondrial bioenergetics and myocardial functionModulation of mitochondrial bioenergetics in the isolated Guinea pig beating heart by potassium and lidocaine cardioplegia: implications for cardioprotection.Enzyme-dependent fluorescence recovery of NADH after photobleaching to assess dehydrogenase activity of isolated perfused hearts.Calcium release microdomains and mitochondria.Mitochondrial Ca2+ uptake: tortoise or hare?Optical Cryoimaging Reveals a Heterogeneous Distribution of Mitochondrial Redox State in ex vivo Guinea Pig Hearts and Its Alteration During Ischemia and Reperfusion.Rapid changes in NADH and flavin autofluorescence in rat cardiac trabeculae reveal large mitochondrial complex II reserve capacity.Ischemic and anesthetic preconditioning reduces cytosolic [Ca2+] and improves Ca(2+) responses in intact hearts.Altered NADH and improved function by anesthetic and ischemic preconditioning in guinea pig intact hearts.Reduced contraction strength with increased intracellular [Ca2+] in left ventricular trabeculae from failing rat hearts.Cross-bridge kinetics modeled from myoplasmic [Ca2+] and LV pressure at 17 degrees C and after 37 degrees C and 17 degrees C ischemia.Myocardial contractile function during postischemic low-flow reperfusion: critical thresholds of NADH and O2 delivery.Warm ischemic preconditioning improves mitochondrial redox balance during and after mild hypothermic ischemia in guinea pig isolated hearts.Ischemia-reperfusion injury changes the dynamics of Ca2+-contraction coupling due to inotropic drugs in isolated hearts.Regulation of cardiac energetics: role of redox state and cellular compartmentation during ischemia.Criteria for the multiple use of isolated perfused hearts in electrophysiological and metabolic experiments.NAD(P)H fluorescence imaging of mitochondrial metabolism in contracting Xenopus skeletal muscle fibers: effect of oxygen availability.Oscillations and hypoxic changes of mitochondrial variables in neurons of the brainstem respiratory centre of mice.Successive contractile periods activate mitochondria at the onset of contractions in intact rat cardiac trabeculae.O2 delivery and redox state are determinants of compartment-specific reactive O2 species in myocardial reperfusion.Mechanisms of force inhibition by halothane and isoflurane in intact rat cardiac muscle.
P2860
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P2860
Increased work in cardiac trabeculae causes decreased mitochondrial NADH fluorescence followed by slow recovery.
description
1996 nî lūn-bûn
@nan
1996 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
1996 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
1996年の論文
@ja
1996年論文
@yue
1996年論文
@zh-hant
1996年論文
@zh-hk
1996年論文
@zh-mo
1996年論文
@zh-tw
1996年论文
@wuu
name
Increased work in cardiac trab ...... nce followed by slow recovery.
@ast
Increased work in cardiac trab ...... nce followed by slow recovery.
@en
Increased work in cardiac trab ...... nce followed by slow recovery.
@nl
type
label
Increased work in cardiac trab ...... nce followed by slow recovery.
@ast
Increased work in cardiac trab ...... nce followed by slow recovery.
@en
Increased work in cardiac trab ...... nce followed by slow recovery.
@nl
prefLabel
Increased work in cardiac trab ...... nce followed by slow recovery.
@ast
Increased work in cardiac trab ...... nce followed by slow recovery.
@en
Increased work in cardiac trab ...... nce followed by slow recovery.
@nl
P2860
P1433
P1476
Increased work in cardiac trab ...... ence followed by slow recovery
@en
P2093
P2860
P304
P356
10.1016/S0006-3495(96)79303-7
P407
P577
1996-08-01T00:00:00Z