Enhanced Na+/H+ exchange during ischemia and reperfusion impairs mitochondrial bioenergetics and myocardial function
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The number of X chromosomes influences protection from cardiac ischaemia/reperfusion injury in mice: one X is better than two.Mitochondrial VDAC1: A Key Gatekeeper as Potential Therapeutic TargetPotential therapeutic benefits of strategies directed to mitochondria.Mitochondrial approaches to protect against cardiac ischemia and reperfusion injury.Differential effects of buffer pH on Ca(2+)-induced ROS emission with inhibited mitochondrial complexes I and III.Adding ROS quenchers to cold K+ cardioplegia reduces superoxide emission during 2-hour global cold cardiac ischemia.Ranolazine reduces Ca2+ overload and oxidative stress and improves mitochondrial integrity to protect against ischemia reperfusion injury in isolated heartsMeasuring mitochondrial function in intact cardiac myocytes.Mitochondrial handling of excess Ca2+ is substrate-dependent with implications for reactive oxygen species generation.Modulation of mitochondrial bioenergetics in the isolated Guinea pig beating heart by potassium and lidocaine cardioplegia: implications for cardioprotection.Molecular mechanism and physiological role of active-deactive transition of mitochondrial complex I.Sarcoplasmic reticulum-mitochondria communication in cardiovascular pathophysiology.Mitochondrial NHE1: a newly identified target to prevent heart disease.Optical Cryoimaging Reveals a Heterogeneous Distribution of Mitochondrial Redox State in ex vivo Guinea Pig Hearts and Its Alteration During Ischemia and Reperfusion.Rapid changes in NADH and flavin autofluorescence in rat cardiac trabeculae reveal large mitochondrial complex II reserve capacity.Ischemic post-conditioning to counteract intestinal ischemia/reperfusion injury.Effect of monovalent cations on the kinetics of hypoxic conformational change of mitochondrial complex I.Reactive oxygen species production induced by pore opening in cardiac mitochondria: The role of complex III.Reactive oxygen species production induced by pore opening in cardiac mitochondria: The role of complex II.Optical metabolic imaging of irradiated rat heart exposed to ischemia-reperfusion injury.Myocardial Na+/H+ exchanger-1 (NHE1) content is decreased by exercise training.
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P2860
Enhanced Na+/H+ exchange during ischemia and reperfusion impairs mitochondrial bioenergetics and myocardial function
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on September 2008
@en
vedecký článok
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vetenskaplig artikel
@sv
videnskabelig artikel
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vědecký článek
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name
Enhanced Na+/H+ exchange durin ...... getics and myocardial function
@en
Enhanced Na+/H+ exchange durin ...... etics and myocardial function.
@nl
type
label
Enhanced Na+/H+ exchange durin ...... getics and myocardial function
@en
Enhanced Na+/H+ exchange durin ...... etics and myocardial function.
@nl
prefLabel
Enhanced Na+/H+ exchange durin ...... getics and myocardial function
@en
Enhanced Na+/H+ exchange durin ...... etics and myocardial function.
@nl
P2093
P2860
P1476
Enhanced Na+/H+ exchange durin ...... getics and myocardial function
@en
P2093
Amadou K S Camara
David F Stowe
James S Heisner
Marisha Spence
Mohammed Aldakkak
P2860
P304
P356
10.1097/FJC.0B013E3181831337
P407
P577
2008-09-01T00:00:00Z