Low TRBP levels support an innate human immunodeficiency virus type 1 resistance in astrocytes by enhancing the PKR antiviral response. - Wikidata - awgldk - TriplyDB

Low TRBP levels support an innate human immunodeficiency virus type 1 resistance in astrocytes by enhancing the PKR antiviral response.

Low TRBP levels support an innate human immunodeficiency virus type 1 resistance in astrocytes by enhancing the PKR antiviral response.

http://www.wikidata.org/entity/Q34045759

about

Interaction of human tRNA-dihydrouridine synthase-2 with interferon-induced protein kinase PKR Impact of protein kinase PKR in cell biology: from antiviral to antiproliferative action Small interfering RNAs against the TAR RNA binding protein, TRBP, a Dicer cofactor, inhibit human immunodeficiency virus type 1 long terminal repeat expression and viral production Dual role of TRBP in HIV replication and RNA interference: viral diversion of a cellular pathway or evasion from antiviral immunity?New pandemics: HIV and AIDS, HCV and chronic hepatitis, influenza virus and flu Targeting the Brain Reservoirs: Toward an HIV Cure Enhancement of replication of RNA viruses by ADAR1 via RNA editing and inhibition of RNA-activated protein kinase Characterization of the TRBP domain required for dicer interaction and function in RNA interference.STAT3 and its phosphorylation are involved in HIV-1 Tat-induced transactivation of glial fibrillary acidic protein Co-expression of miRNA targeting the expression of PERK, but not PKR, enhances cellular immunity from an HIV-1 Env DNA vaccine.Unperturbed posttranscriptional regulatory Rev protein function and HIV-1 replication in astrocytes HIV-1 transcriptional regulation in the central nervous system and implications for HIV cure research HIV-1 entry and trans-infection of astrocytes involves CD81 vesicles.HIV research in Australia: linking basic research findings with clinical and public health outcomes.Human immunodeficiency virus-restricted replication in astrocytes and the ability of gamma interferon to modulate this restriction are regulated by a downstream effector of the Wnt signaling pathway.Endocytosis of human immunodeficiency virus 1 (HIV-1) in astrocytes: a fiery path to its destination.The multiple functions of TRBP, at the hub of cell responses to viruses, stress, and cancer.Human immunodeficiency virus type 1 (HIV-1) transactivator of transcription through its intact core and cysteine-rich domains inhibits Wnt/β-catenin signaling in astrocytes: relevance to HIV neuropathogenesis dsRNA-dependent protein kinase PKR and its role in stress, signaling and HCV infection.Interplay between Wnt/β-catenin signaling and HIV: virologic and biologic consequences in the CNS.Reduced basal transcriptional activity of central nervous system-derived HIV type 1 long terminal repeats.Movements of HIV-1 genomic RNA-APOBEC3F complexes and PKR reveal cytoplasmic and nuclear PKR defenses and HIV-1 evasion strategies.Progress and prospects: RNA-based therapies for treatment of HIV infection.Sendai virus C protein plays a role in restricting PKR activation by limiting the generation of intracellular double-stranded RNA.TRBP control of PACT-induced phosphorylation of protein kinase R is reversed by stress.ADAR1 and PACT contribute to efficient translation of transcripts containing HIV-1 trans-activating response (TAR) element.HIV-1 endocytosis in astrocytes: a kiss of death or survival of the fittest?The cellular TAR RNA binding protein, TRBP, promotes HIV-1 replication primarily by inhibiting the activation of double-stranded RNA-dependent kinase PKR.The PKR activator, PACT, becomes a PKR inhibitor during HIV-1 replication.ADAR1 interacts with PKR during human immunodeficiency virus infection of lymphocytes and contributes to viral replication The presence of the TAR RNA structure alters the programmed -1 ribosomal frameshift efficiency of the human immunodeficiency virus type 1 (HIV-1) by modifying the rate of translation initiation.Tat RNA silencing suppressor activity contributes to perturbation of lymphocyte miRNA by HIV-1.HIV-1 NC-induced stress granule assembly and translation arrest are inhibited by the dsRNA binding protein Staufen1.Multiple Inhibitory Factors Act in the Late Phase of HIV-1 Replication: a Systematic Review of the Literature.

P2860

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P2860

Low TRBP levels support an innate human immunodeficiency virus type 1 resistance in astrocytes by enhancing the PKR antiviral response.

http://www.wikidata.org/entity/Q34045759

description

2005 nî lūn-bûn

@nan

2005 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2005 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2005年の論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年论文

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Low TRBP levels support an inn ...... ng the PKR antiviral response.

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Low TRBP levels support an inn ...... ng the PKR antiviral response.

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Low TRBP levels support an inn ...... ng the PKR antiviral response.

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Low TRBP levels support an inn ...... ng the PKR antiviral response.

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Low TRBP levels support an inn ...... ng the PKR antiviral response.

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Low TRBP levels support an inn ...... ng the PKR antiviral response.

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Low TRBP levels support an inn ...... ng the PKR antiviral response.

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Low TRBP levels support an inn ...... ng the PKR antiviral response.

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Low TRBP levels support an inn ...... ng the PKR antiviral response.

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P356

JVI.79.20.12763-12772.2005

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Journal of Virology

P1476

Low TRBP levels support an inn ...... ng the PKR antiviral response.

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P2093

Andrew J Mouland

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Anthony Jaworowski

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Chi L Ong

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Damian F J Purcell

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Guerline Clerzius

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Helen S Christensen

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Jane L Howard

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Janine C Thorpe

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Paul R Gorry

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Sean Chung

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P2860

THE HIV-1 REV PROTEIN

Organization of the human tarbp2 gene reveals two promoters that are repressed in an astrocytic cell line

The double-stranded RNA-binding protein Staufen is incorporated in human immunodeficiency virus type 1: evidence for a role in genomic RNA encapsidation

Relatedness of an RNA-binding motif in human immunodeficiency virus type 1 TAR RNA-binding protein TRBP to human P1/dsI kinase and Drosophila staufen

Mutant influenza viruses with a defective NS1 protein cannot block the activation of PKR in infected cells

Analysis of a binding difference between the two dsRNA-binding domains in TRBP reveals the modular function of a KR-helix motif

HIV-I TAT inhibits PKR activity by both RNA-dependent and RNA-independent mechanisms

Two dimerization domains in the trans-activation response RNA-binding protein (TRBP) individually reverse the protein kinase R inhibition of HIV-1 long terminal repeat expression

A molecular rheostat. Co-operative rev binding to stem I of the rev-response element modulates human immunodeficiency virus type-1 late gene expression

Characterization of a human TAR RNA-binding protein that activates the HIV-1 LTR

P304

12763-12772

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10.1128/JVI.79.20.12763-12772.2005

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20

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16188979

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