Multiple cyclin-dependent kinases signals are critical mediators of ischemia/hypoxic neuronal death in vitro and in vivo. - Wikidata - awgldk - TriplyDB

Multiple cyclin-dependent kinases signals are critical mediators of ischemia/hypoxic neuronal death in vitro and in vivo.

Multiple cyclin-dependent kinases signals are critical mediators of ischemia/hypoxic neuronal death in vitro and in vivo.

http://www.wikidata.org/entity/Q34047944

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Ischemic stroke injury is mediated by aberrant Cdk5 The Importance of Thrombin in Cerebral Injury and Disease Combinations of Kinase Inhibitors Protecting Myoblasts against Hypoxia Regulation of ischemic neuronal death by E2F4-p130 protein complexes The expression of cyclins in neurons of rats after focal cerebral ischemia.Essential role of cytoplasmic cdk5 and Prx2 in multiple ischemic injury models, in vivo.Non-Coding RNAs as Potential Neuroprotectants against Ischemic Brain Injury.Conditional disruption of calpain in the CNS alters dendrite morphology, impairs LTP, and promotes neuronal survival following injury.Cell cycle molecules define a pathway required for neuron death in development and disease.Cell cycle activation and spinal cord injury.Stereotyped fetal brain disorganization is induced by hypoxia and requires lysophosphatidic acid receptor 1 (LPA1) signaling.Neuronal cell cycle: the neuron itself and its circumstances Cyclin D1 gene ablation confers neuroprotection in traumatic brain injury Tissue-type plasminogen activator has a neuroprotective effect in the ischemic brain mediated by neuronal TNF-α.Bilateral gene interaction hierarchy analysis of the cell death gene response emphasizes the significance of cell cycle genes following unilateral traumatic brain injury Remote Ischemic Conditioning Alters Methylation and Expression of Cell Cycle Genes in Aneurysmal Subarachnoid Hemorrhage.The Parkinson's disease gene DJ-1 is also a key regulator of stroke-induced damage.Notch signaling in response to excitotoxicity induces neurodegeneration via erroneous cell cycle reentry.Mouse model of diffuse brain damage following anoxia, evaluated by a new assay of generalized arousal Emerging molecular targets for brain repair after stroke Attenuation of Postischemic Genomic Alteration by Mesenchymal Stem Cells: a Microarray Study.The emerging role of epigenetics in stroke: II. RNA regulatory circuitry.Synergy of homocysteine, microRNA, and epigenetics: a novel therapeutic approach for stroke.Cyclin-dependent kinase 2 signaling regulates myocardial ischemia/reperfusion injury.Calpain-mediated signaling mechanisms in neuronal injury and neurodegeneration Mesenchymal Stem Cells Loaded with p5, Derived from CDK5 Activator p35, Inhibit Calcium-Induced CDK5 Activation in Endothelial Cells MiR-26b, upregulated in Alzheimer's disease, activates cell cycle entry, tau-phosphorylation, and apoptosis in postmitotic neurons Roscovitine reduces neuronal loss, glial activation, and neurologic deficits after brain trauma.Multifunctional drug treatment in neurotrauma.Cell cycle reactivation in mature neurons: a link with brain plasticity, neuronal injury and neurodegenerative diseases?Role of cell cycle re-entry in neurons: a common apoptotic mechanism of neuronal cell death.Comparison of gene expression during in vivo and in vitro postnatal retina development.MiRNAs: potential diagnostic and therapeutic targets for cerebral ischaemia.Role of CDK5/cyclin complexes in ischemia-induced death and survival of renal tubular cells.Structural Bioinformatics Approach of Cyclin-Dependent Kinases 1 and 3 Complexed with Inhibitors.Sublethal doses of β-amyloid peptide abrogate DNA-dependent protein kinase activity.The mitochondrial inner membrane GTPase, optic atrophy 1 (Opa1), restores mitochondrial morphology and promotes neuronal survival following excitotoxicity.CDK5 phosphorylates DRP1 and drives mitochondrial defects in NMDA-induced neuronal death.LXCXE-independent chromatin remodeling by Rb/E2f mediates neuronal quiescence.Novel rat Alzheimer's disease models based on AAV-mediated gene transfer to selectively increase hippocampal Abeta levels.

P2860

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P2860

Multiple cyclin-dependent kinases signals are critical mediators of ischemia/hypoxic neuronal death in vitro and in vivo.

http://www.wikidata.org/entity/Q34047944

description

2005 nî lūn-bûn

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2005 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

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2005年論文

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name

Multiple cyclin-dependent kina ...... al death in vitro and in vivo.

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Multiple cyclin-dependent kina ...... al death in vitro and in vivo.

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Multiple cyclin-dependent kina ...... al death in vitro and in vivo.

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Multiple cyclin-dependent kina ...... al death in vitro and in vivo.

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Multiple cyclin-dependent kina ...... al death in vitro and in vivo.

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Proceedings of the National Academy of Sciences of the United States of America

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Multiple cyclin-dependent kina ...... nal death in vitro and in vivo

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Grace Iyirhiaro

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Inez Vincent

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Juliet Rashidian

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Mario Rios

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Matthew J During

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Ross J Bland

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Ruth S Slack

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Steven Callaghan

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P2860

A simplified system for generating recombinant adenoviruses

Pathobiology of ischaemic stroke: an integrated view

Cdk5-mediated inhibition of the protective effects of transcription factor MEF2 in neurotoxicity-induced apoptosis

Cdk5 activation induces hippocampal CA1 cell death by directly phosphorylating NMDA receptors

Inhibition of cyclin-dependent kinases improves CA1 neuronal survival and behavioral performance after global ischemia in the rat

Cyclin D1/Cdk4 regulates retinoblastoma protein-mediated cell cycle arrest by site-specific phosphorylation

Distinct roles for cyclin-dependent kinases in cell cycle control

E2F1 mediates death of B-amyloid-treated cortical neurons in a manner independent of p53 and dependent on Bax and caspase 3.

Regulation of G(1) cyclin-dependent kinases in the mammalian cell cycle.

A decade of CDK5.

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14080-14085

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10.1073/PNAS.0500099102

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39

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102

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Hossein Aleyasin

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7597417

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