Error-prone DNA polymerase IV is controlled by the stress-response sigma factor, RpoS, in Escherichia coli. - Wikidata - awgldk - TriplyDB

Error-prone DNA polymerase IV is controlled by the stress-response sigma factor, RpoS, in Escherichia coli.

Error-prone DNA polymerase IV is controlled by the stress-response sigma factor, RpoS, in Escherichia coli.

http://www.wikidata.org/entity/Q34049897

about

An SOS-regulated type 2 toxin-antitoxin system Adaptive amplification and point mutation are independent mechanisms: evidence for various stress-inducible mutation mechanisms Dispersal and regulation of an adaptive mutagenesis cassette in the bacteria domain.Culture history and population heterogeneity as determinants of bacterial adaptation: the adaptomics of a single environmental transition A strategically located serine residue is critical for the mutator activity of DNA polymerase IV from Escherichia coli The yeast environmental stress response regulates mutagenesis induced by proteotoxic stress Mutability and importance of a hypermutable cell subpopulation that produces stress-induced mutants in Escherichia coli.Competition of Escherichia coli DNA polymerases I, II and III with DNA Pol IV in stressed cells Error-prone DNA polymerase IV is regulated by the heat shock chaperone GroE in Escherichia coli Genomewide Mutational Diversity in Escherichia coli Population Evolving in Prolonged Stationary Phase.Global chromosomal structural instability in a subpopulation of starving Escherichia coli cells The sigma(E) stress response is required for stress-induced mutation and amplification in Escherichia coli.Dealing with oxidative stress and iron starvation in microorganisms: an overview.Separate DNA Pol II- and Pol IV-dependent pathways of stress-induced mutation during double-strand-break repair in Escherichia coli are controlled by RpoS.The SOS Regulatory Network.Roles of E. coli double-strand-break-repair proteins in stress-induced mutation The SMC-like protein complex SbcCD enhances DNA polymerase IV-dependent spontaneous mutation in Escherichia coli Translesion DNA Synthesis.General stress response regulator RpoS in adaptive mutation and amplification in Escherichia coli.Involvement of Y-family DNA polymerases in mutagenesis caused by oxidized nucleotides in Escherichia coli Escherichia coli Rep DNA helicase and error-prone DNA polymerase IV interact physically and functionally Escherichia coli YafP protein modulates DNA damaging property of the nitroaromatic compounds Phenotypic diversity caused by differential RpoS activity among environmental Escherichia coli isolates.Double-Strand Break Repair and Holliday Junction Processing Are Required for Chromosome Processing in Stationary-Phase Escherichia coli Cells Development of a stress-induced mutagenesis module for autonomous adaptive evolution of Escherichia coli to improve its stress tolerance Mutation as a stress response and the regulation of evolvability Interactions and Localization of Escherichia coli Error-Prone DNA Polymerase IV after DNA Damage.Stress responses and genetic variation in bacteria.Perspective on mutagenesis and repair: the standard model and alternate modes of mutagenesis.Diversify or die: generation of diversity in response to stress.Determinants of spontaneous mutation in the bacterium Escherichia coli as revealed by whole-genome sequencing.Roles of Nucleoid-Associated Proteins in Stress-Induced Mutagenic Break Repair in Starving Escherichia coli.A shifting mutational landscape in 6 nutritional states: Stress-induced mutagenesis as a series of distinct stress input-mutation output relationships.Stress-induced mutation via DNA breaks in Escherichia coli: a molecular mechanism with implications for evolution and medicine.Bacterial stationary-state mutagenesis and Mammalian tumorigenesis as stress-induced cellular adaptations and the role of epigenetics.β-Lactam antibiotics promote bacterial mutagenesis via an RpoS-mediated reduction in replication fidelity.Competitive fitness during feast and famine: how SOS DNA polymerases influence physiology and evolution in Escherichia coli Causes and consequences of DNA repair activity modulation during stationary phase in Escherichia coli.Adaptive amplification.Stationary phase mutagenesis in B. subtilis: a paradigm to study genetic diversity programs in cells under stress.

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P2860

Error-prone DNA polymerase IV is controlled by the stress-response sigma factor, RpoS, in Escherichia coli.

http://www.wikidata.org/entity/Q34049897

description

2003 nî lūn-bûn

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2003 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2003 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2003年の論文

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2003年論文

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2003年論文

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2003年論文

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2003年論文

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2003年論文

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2003年论文

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name

Error-prone DNA polymerase IV ...... or, RpoS, in Escherichia coli.

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Error-prone DNA polymerase IV ...... or, RpoS, in Escherichia coli.

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Error-prone DNA polymerase IV ...... or, RpoS, in Escherichia coli.

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Error-prone DNA polymerase IV ...... or, RpoS, in Escherichia coli.

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Error-prone DNA polymerase IV ...... or, RpoS, in Escherichia coli.

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Error-prone DNA polymerase IV ...... or, RpoS, in Escherichia coli.

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Error-prone DNA polymerase IV ...... or, RpoS, in Escherichia coli.

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Error-prone DNA polymerase IV ...... or, RpoS, in Escherichia coli.

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Error-prone DNA polymerase IV ...... or, RpoS, in Escherichia coli.

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J.1365-2958.2003.03704.X

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Molecular Microbiology

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Error-prone DNA polymerase IV ...... or, RpoS, in Escherichia coli.

@en

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Jill C Layton

http://www.w3.org/2001/XMLSchema#string

Patricia L Foster

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P2860

SOS-induced DNA polymerases enhance long-term survival and evolutionary fitness

Spontaneous point mutations that occur more often when advantageous than when neutral

Adaptive mutation: the uses of adversity

All three SOS-inducible DNA polymerases (Pol II, Pol IV and Pol V) are involved in induced mutagenesis

Multiple pathways for SOS-induced mutagenesis in Escherichia coli: an overexpression of dinB/dinP results in strongly enhancing mutagenesis in the absence of any exogenous treatment to damage DNA

Signal transduction and regulatory mechanisms involved in control of the sigma(S) (RpoS) subunit of RNA polymerase

A constant rate of spontaneous mutation in DNA-based microbes

The origin of mutants

Comparative gene expression profiles following UV exposure in wild-type and SOS-deficient Escherichia coli

The beta clamp targets DNA polymerase IV to DNA and strongly increases its processivity

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549-561

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scholarly article

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10.1046/J.1365-2958.2003.03704.X

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2

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2003-10-01T00:00:00Z

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9009699

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14617178

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Escherichia coli

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1237112

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