about
Customisation of the exome data analysis pipeline using a combinatorial approachHypermutation in human cancer genomes: footprints and mechanismsEvolution of the cancer genomeFine-scale signatures of molecular evolution reconcile models of indel-associated mutationMicrohomology-mediated end joining induces hypermutagenesis at breakpoint junctions.Molecular hyperdiversity and evolution in very large populations.Microhomology-mediated mechanisms underlie non-recurrent disease-causing microdeletions of the FOXL2 gene or its regulatory domain.Fragile DNA motifs trigger mutagenesis at distant chromosomal loci in saccharomyces cerevisiae.Damage-induced localized hypermutability.The pattern of DNA cleavage intensity around indelsOn the sequence-directed nature of human gene mutation: the role of genomic architecture and the local DNA sequence environment in mediating gene mutations underlying human inherited disease.Somatic rearrangements across cancer reveal classes of samples with distinct patterns of DNA breakage and rearrangement-induced hypermutability.A small indel mutation in an anthocyanin transporter causes variegated colouration of peach flowers.PGMRA: a web server for (phenotype x genotype) many-to-many relation analysis in GWAS.Loss of heterozygosity preferentially occurs in early replicating regions in cancer genomes.Copy neutral loss of heterozygosity is more frequent in older ovarian cancer patientsLate-replicating CNVs as a source of new genes.Insertions/Deletions-Associated Nucleotide Polymorphism in Arabidopsis thaliana.Origins and breakpoint analyses of copy number variations: up close and personal.Characterization of 26 deletion CNVs reveals the frequent occurrence of micro-mutations within the breakpoint-flanking regions and frequent repair of double-strand breaks by templated insertions derived from remote genomic regions.Protein insertions and deletions enabled by neutral roaming in sequence space.Genetic drift and mutational hazard in the evolution of salamander genomic gigantism.Small insertions are more deleterious than small deletions in human genomes.More single-nucleotide mutations surround small insertions than small deletions in primates.Evolution of complex adaptations in molecular systems.The non-random patterns of genetic variation induced by asymmetric somatic hybridization in wheat
P2860
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P2860
description
2010 nî lūn-bûn
@nan
2010 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
A time-invariant principle of genome evolution.
@ast
A time-invariant principle of genome evolution.
@en
A time-invariant principle of genome evolution.
@nl
type
label
A time-invariant principle of genome evolution.
@ast
A time-invariant principle of genome evolution.
@en
A time-invariant principle of genome evolution.
@nl
prefLabel
A time-invariant principle of genome evolution.
@ast
A time-invariant principle of genome evolution.
@en
A time-invariant principle of genome evolution.
@nl
P2860
P356
P1476
A time-invariant principle of genome evolution.
@en
P2860
P304
13004-13009
P356
10.1073/PNAS.0914454107
P407
P577
2010-07-06T00:00:00Z