Bcl-X(L) affects Ca(2+) homeostasis by altering expression of inositol 1,4,5-trisphosphate receptors
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IP3 receptor-mediated Ca2+ release in naive CD4 T cells dictates their cytokine programThe mitochondrial fission protein hFis1 requires the endoplasmic reticulum gateway to induce apoptosisBcl-x(L) blocks a mitochondrial inner membrane channel and prevents Ca2+ overload-mediated cell deathThe Bcl-2 protein family member Bok binds to the coupling domain of inositol 1,4,5-trisphosphate receptors and protects them from proteolytic cleavageProapoptotic BAX and BAK regulate the type 1 inositol trisphosphate receptor and calcium leak from the endoplasmic reticulumThe serine/threonine kinase Pim-2 is a transcriptionally regulated apoptotic inhibitorBax and Bak can localize to the endoplasmic reticulum to initiate apoptosisPhosphorylation of BCL-2 regulates ER Ca2+ homeostasis and apoptosis.Bcl-x(L)-mediated changes in metabolic pathways of breast cancer cells: from survival in the blood stream to organ-specific metastasis.Praf2 is a novel Bcl-xL/Bcl-2 interacting protein with the ability to modulate survival of cancer cellsCalcium and ER stress mediate hepatic apoptosis after burn injury.Inositol trisphosphate receptor Ca2+ release channels.Inhibitors of Bcl-2 protein family deplete ER Ca2+ stores in pancreatic acinar cells.The type III inositol 1,4,5-trisphosphate receptor preferentially transmits apoptotic Ca2+ signals into mitochondria.Selective regulation of IP3-receptor-mediated Ca2+ signaling and apoptosis by the BH4 domain of Bcl-2 versus Bcl-Xl.Bcl-2 and calcium: controversy beneath the surface.Bcl-2 family in inter-organelle modulation of calcium signaling; roles in bioenergetics and cell survival.Selective role for superoxide in InsP3 receptor-mediated mitochondrial dysfunction and endothelial apoptosisFlavopiridol causes early mitochondrial damage in chronic lymphocytic leukemia cells with impaired oxygen consumption and mobilization of intracellular calcium.Calcium signaling in cholangiocytesMicroRNAs and Glucocorticoid-Induced Apoptosis in Lymphoid MalignanciesThe type III inositol 1,4,5-trisphosphate receptor is associated with aggressiveness of colorectal carcinomaThe anti-apoptotic protein HAX-1 interacts with SERCA2 and regulates its protein levels to promote cell survival.Calcium overload is essential for the acceleration of staurosporine-induced cell death following neuronal differentiation in PC12 cellsThe proapoptotic factors Bax and Bak regulate T Cell proliferation through control of endoplasmic reticulum Ca(2+) homeostasisThe endoplasmic reticulum in apoptosis and autophagy: role of the BCL-2 protein family.The antiapoptotic protein Bcl-xL negatively regulates the bone-resorbing activity of osteoclasts in mice.Protect and serve: Bcl-2 proteins as guardians and rulers of cancer cell survival.Oxysterol-binding protein-related protein 4L promotes cell proliferation by sustaining intracellular Ca2+ homeostasis in cervical carcinoma cell linesBcl-2 interaction with the inositol 1,4,5-trisphosphate receptor: role in Ca(2+) signaling and disease.The selective BH4-domain biology of Bcl-2-family members: IP3Rs and beyond.Calcium signaling in the liverBcl-2 regulation of the inositol 1,4,5-trisphosphate receptor and calcium signaling in normal and malignant lymphocytes: potential new target for cancer treatment.Non-canonical roles of Bcl-2 and Bcl-xL proteins: relevance of BH4 domain.Bcl-2 proteins and calcium signaling: complexity beneath the surface.Deficits in the mitochondrial enzyme α-ketoglutarate dehydrogenase lead to Alzheimer's disease-like calcium dysregulationDual mechanisms of sHA 14-1 in inducing cell death through endoplasmic reticulum and mitochondria.BCL-xL: time-dependent dissociation between modulation of apoptosis and invasiveness in human malignant glioma cells.The anti-apoptotic protein Mcl-1 inhibits mitochondrial Ca2+ signals.The regulatory domain of the inositol 1,4,5-trisphosphate receptor is necessary to keep the channel domain closed: possible physiological significance of specific cleavage by caspase 3.
P2860
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P2860
Bcl-X(L) affects Ca(2+) homeostasis by altering expression of inositol 1,4,5-trisphosphate receptors
description
2002 nî lūn-bûn
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2002 թուականի Յուլիսին հրատարակուած գիտական յօդուած
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2002 թվականի հուլիսին հրատարակված գիտական հոդված
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2002年の論文
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2002年論文
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2002年論文
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2002年論文
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2002年論文
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2002年論文
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Bcl-X(L) affects Ca(2+) homeos ...... 1,4,5-trisphosphate receptors
@ast
Bcl-X(L) affects Ca(2+) homeos ...... 1,4,5-trisphosphate receptors
@en
Bcl-X(L) affects Ca(2+) homeos ...... 1,4,5-trisphosphate receptors
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label
Bcl-X(L) affects Ca(2+) homeos ...... 1,4,5-trisphosphate receptors
@ast
Bcl-X(L) affects Ca(2+) homeos ...... 1,4,5-trisphosphate receptors
@en
Bcl-X(L) affects Ca(2+) homeos ...... 1,4,5-trisphosphate receptors
@nl
prefLabel
Bcl-X(L) affects Ca(2+) homeos ...... 1,4,5-trisphosphate receptors
@ast
Bcl-X(L) affects Ca(2+) homeos ...... 1,4,5-trisphosphate receptors
@en
Bcl-X(L) affects Ca(2+) homeos ...... 1,4,5-trisphosphate receptors
@nl
P2093
P2860
P356
P1476
Bcl-X(L) affects Ca(2+) homeos ...... 1,4,5-trisphosphate receptors
@en
P2093
Casey J Fox
Craig B Thompson
Lewis A Chodosh
Stephen R Master
Vytautas P Bindokas
P2860
P304
P356
10.1073/PNAS.152571899
P407
P577
2002-07-12T00:00:00Z