Systemic autoimmunity and lymphoproliferation are associated with excess IL-7 and inhibited by IL-7Rα blockade.
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IL-18 synergizes with IL-7 to drive slow proliferation of naive CD8 T cells by costimulating self-peptide-mediated TCR signals.FcRγ controls the fas-dependent regulatory function of lymphoproliferative double negative T cellsModulation of peripheral T-cell function by interleukin-7 in rheumatoid arthritis.Innate immune signaling induces interleukin-7 production from salivary gland cells and accelerates the development of primary Sjögren's syndrome in a mouse modelThe relationship of cytomegalovirus (CMV) infection with circulatory IFN-α levels and IL-7 receptor α expression on CD8+ T cells in human aging.Screening for peptides targeted to IL-7Rα for molecular imaging of rheumatoid arthritis synoviumInterleukins 7 and 15 Maintain Human T Cell Proliferative Capacity through STAT5 Signaling.Interleukin-7 is required for CD4(+) T cell activation and autoimmune neuroinflammation.Broad induction of immunoregulatory mechanisms after a short course of anti-IL-7Rα antibodies in NOD mice.Anti-IFN-α/β receptor antibody treatment ameliorates disease in lupus-predisposed miceCritical role of transmethylation in TLR signaling and systemic lupus erythematosus.Soluble IL7Rα potentiates IL-7 bioactivity and promotes autoimmunity.Interleukin-7 enhances the Th1 response to promote the development of Sjögren's syndrome-like autoimmune exocrinopathy in miceBioinformatics analysis of the factors controlling type I IFN gene expression in autoimmune disease and virus-induced immunity.cAMP-responsive element modulator α (CREMα) trans-represses the transmembrane glycoprotein CD8 and contributes to the generation of CD3+CD4-CD8- T cells in health and disease.IL-7 receptor blockade following T cell depletion promotes long-term allograft survival.Distinct pathophysiological cytokine profiles for discrimination between autoimmune pancreatitis, chronic pancreatitis, and pancreatic ductal adenocarcinoma.Interleukin-7 receptor single nucleotide polymorphism rs6897932 (C/T) and the susceptibility to systemic lupus erythematosus.Anti-IL-7 receptor-α treatment ameliorates newly established Sjögren's-like exocrinopathy in non-obese diabetic mice.Lupus acceleration by a MAVS-activating RNA virus requires endosomal TLR signaling and host genetic predispositionIL-7 receptor blockade blunts antigen-specific memory T cell responses and chronic inflammation in primatesPTPN2 attenuates T-cell lymphopenia-induced proliferation
P2860
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P2860
Systemic autoimmunity and lymphoproliferation are associated with excess IL-7 and inhibited by IL-7Rα blockade.
description
2011 nî lūn-bûn
@nan
2011 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Systemic autoimmunity and lymp ...... inhibited by IL-7Rα blockade.
@ast
Systemic autoimmunity and lymp ...... inhibited by IL-7Rα blockade.
@en
Systemic autoimmunity and lymp ...... inhibited by IL-7Rα blockade.
@nl
type
label
Systemic autoimmunity and lymp ...... inhibited by IL-7Rα blockade.
@ast
Systemic autoimmunity and lymp ...... inhibited by IL-7Rα blockade.
@en
Systemic autoimmunity and lymp ...... inhibited by IL-7Rα blockade.
@nl
prefLabel
Systemic autoimmunity and lymp ...... inhibited by IL-7Rα blockade.
@ast
Systemic autoimmunity and lymp ...... inhibited by IL-7Rα blockade.
@en
Systemic autoimmunity and lymp ...... inhibited by IL-7Rα blockade.
@nl
P2093
P2860
P1433
P1476
Systemic autoimmunity and lymp ...... inhibited by IL-7Rα blockade.
@en
P2093
Argyrios N Theofilopoulos
Brian R Lawson
Dwight H Kono
John C Scatizzi
Joseph Craft
Roberto Baccala
Rosana Gonzalez-Quintial
P2860
P304
P356
10.1371/JOURNAL.PONE.0027528
P407
P577
2011-11-10T00:00:00Z