Acute mTOR inhibition induces insulin resistance and alters substrate utilization in vivo - Wikidata - awgldk - TriplyDB

Acute mTOR inhibition induces insulin resistance and alters substrate utilization in vivo

Acute mTOR inhibition induces insulin resistance and alters substrate utilization in vivo

http://www.wikidata.org/entity/Q34080562

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Influence of Acute and Chronic Exercise on Glucose Uptake Intermittent Administration of Rapamycin Extends the Life Span of Female C57BL/6J Mice The Mechanistic Target of Rapamycin: The Grand ConducTOR of Metabolism and Aging Age-Related Neurodegeneration Prevention Through mTOR Inhibition: Potential Mechanisms and Remaining Questions.Pulsatile delivery of a leucine supplement during long-term continuous enteral feeding enhances lean growth in term neonatal pigs.Optogenetic activation reveals distinct roles of PIP3 and Akt in adipocyte insulin action.Rapamycin: An InhibiTOR of Aging Emerges From the Soil of Easter Island.mTORC2 and AMPK differentially regulate muscle triglyceride content via Perilipin 3 mTORC2 signalling regulates M2 macrophage differentiation in response to helminth infection and adaptive thermogenesis Diabetes and dyslipidemia: characterizing lipoprotein metabolism.A high sucrose and high fat diet induced the development of insulin resistance in the skeletal muscle of Bama miniature pigs through the Akt/GLUT4 pathway.Mammalian target of rapamycin complex 2 regulates muscle glucose uptake during exercise in mice.A small molecule inhibitor of Rheb selectively targets mTORC1 signaling.Regulation of autophagy in human skeletal muscle: effects of exercise, exercise training and insulin stimulation.Reply from Lykke Sylow, Lisbeth L. V. Møller, Maximilian Kleinert, Erik A. Richter and Thomas E. Jensen

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P2860

Acute mTOR inhibition induces insulin resistance and alters substrate utilization in vivo

http://www.wikidata.org/entity/Q34080562

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2014 nî lūn-bûn

@nan

2014 թուականի Յունիսին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի հունիսին հրատարակված գիտական հոդված

@hy

2014年の論文

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2014年論文

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2014年論文

@zh-hant

2014年論文

@zh-hk

2014年論文

@zh-mo

2014年論文

@zh-tw

2014年论文

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name

Acute mTOR inhibition induces insulin resistance and alters substrate utilization in vivo

@ast

Acute mTOR inhibition induces insulin resistance and alters substrate utilization in vivo

@en

Acute mTOR inhibition induces insulin resistance and alters substrate utilization in vivo

@nl

type

label

Acute mTOR inhibition induces insulin resistance and alters substrate utilization in vivo

@ast

Acute mTOR inhibition induces insulin resistance and alters substrate utilization in vivo

@en

Acute mTOR inhibition induces insulin resistance and alters substrate utilization in vivo

@nl

prefLabel

Acute mTOR inhibition induces insulin resistance and alters substrate utilization in vivo

@ast

Acute mTOR inhibition induces insulin resistance and alters substrate utilization in vivo

@en

Acute mTOR inhibition induces insulin resistance and alters substrate utilization in vivo

@nl

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J.MOLMET.2014.06.004

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Molecular Metabolism

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Acute mTOR inhibition induces insulin resistance and alters substrate utilization in vivo

@en

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Andreas B Jordy

http://www.w3.org/2001/XMLSchema#string

Anne-Julie Oxbøll

http://www.w3.org/2001/XMLSchema#string

Bente Kiens

http://www.w3.org/2001/XMLSchema#string

Erik A Richter

http://www.w3.org/2001/XMLSchema#string

Guang Yang

http://www.w3.org/2001/XMLSchema#string

Kristen C Thomas

http://www.w3.org/2001/XMLSchema#string

Markus A Ruegg

http://www.w3.org/2001/XMLSchema#string

P2860

Phosphorylation and regulation of Akt/PKB by the rictor-mTOR complex

Nutrients suppress phosphatidylinositol 3-kinase/Akt signaling via raptor-dependent mTOR-mediated insulin receptor substrate 1 phosphorylation

AS160, the Akt substrate regulating GLUT4 translocation, has a functional Rab GTPase-activating protein domain

Rapamycin, but not resveratrol or simvastatin, extends life span of genetically heterogeneous mice

Rapamycin-induced insulin resistance is mediated by mTORC2 loss and uncoupled from longevity

Fat cell-specific ablation of rictor in mice impairs insulin-regulated fat cell and whole-body glucose and lipid metabolism

mTOR signaling in growth control and disease

Rapamycin fed late in life extends lifespan in genetically heterogeneous mice

SIN1/MIP1 maintains rictor-mTOR complex integrity and regulates Akt phosphorylation and substrate specificity

Ablation in mice of the mTORC components raptor, rictor, or mLST8 reveals that mTORC2 is required for signaling to Akt-FOXO and PKCalpha, but not S6K1

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630-641

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scholarly article

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10.1016/J.MOLMET.2014.06.004

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6

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3

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Peter Schjerling

Daniel Fazakerley

Thomas Elbenhardt Jensen

Maximilian Kleinert

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2014-06-27T00:00:00Z

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263472288

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25161886

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