IL-17 drives psoriatic inflammation via distinct, target cell-specific mechanisms. - Wikidata - awgldk - TriplyDB

IL-17 drives psoriatic inflammation via distinct, target cell-specific mechanisms.

IL-17 drives psoriatic inflammation via distinct, target cell-specific mechanisms.

http://www.wikidata.org/entity/Q34082705

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The roles and functional mechanisms of interleukin-17 family cytokines in mucosal immunity Interleukin 17-Mediated Host Defense against Candida albicans IL-17-Mediated Immunity to the Opportunistic Fungal Pathogen Candida albicans Linking diet to acne metabolomics, inflammation, and comedogenesis: an update Chronic, not acute, skin-specific inflammation promotes thrombosis in psoriasis murine models Epigallocatechin-3-gallate (EGCG) inhibits imiquimod-induced psoriasis-like inflammation of BALB/c mice.IκBζ is a key driver in the development of psoriasis.TRAF2 regulates TNF and NF-κB signalling to suppress apoptosis and skin inflammation independently of Sphingosine kinase 1 Epidermal Notch1 recruits RORγ(+) group 3 innate lymphoid cells to orchestrate normal skin repair Therapeutic antibodies that target inflammatory cytokines in autoimmune diseases Keratinocytes contribute intrinsically to psoriasis upon loss of Tnip1 function.Increased circulatory levels of fractalkine (CX3CL1) are associated with inflammatory chemokines and cytokines in individuals with type-2 diabetes The AMPK enzyme-complex: from the regulation of cellular energy homeostasis to a possible new molecular target in the management of chronic inflammatory disorders.The role of IL-23 and the IL-23/TH 17 immune axis in the pathogenesis and treatment of psoriasis.IL-35 Decelerates the Inflammatory Process by Regulating Inflammatory Cytokine Secretion and M1/M2 Macrophage Ratio in Psoriasis.Targeting TRAF3IP2 by Genetic and Interventional Approaches Inhibits Ischemia/Reperfusion-induced Myocardial Injury and Adverse Remodeling.The Snowballing Literature on Imiquimod-Induced Skin Inflammation in Mice: A Critical Appraisal.Interleukin 17 Family Cytokines: Signaling Mechanisms, Biological Activities, and Therapeutic Implications."Inflammatory skin march" in atopic dermatitis and psoriasis.Cardiovascular and Metabolic Diseases Comorbid with Psoriasis: Beyond the Skin.IL-17 Receptor Signaling in Oral Epithelial Cells Is Critical for Protection against Oropharyngeal Candidiasis.Therapeutic Effects of Methanol Extract from Euphorbia kansui Radix on Imiquimod-Induced Psoriasis.Brief Report: Interleukin-17A-Dependent Asymmetric Stem Cell Divisions Are Increased in Human Psoriasis: A Mechanism Underlying Benign Hyperproliferation.Incorporation of tetanus-epitope into virus-like particles achieves vaccine responses even in older recipients in models of psoriasis, Alzheimer's and cat allergy.Contextual control of skin immunity and inflammation by Corynebacterium.MCPIP1/Regnase-1 Restricts IL-17A- and IL-17C-Dependent Skin Inflammation.Autoimmunity and autoimmune co-morbidities in psoriasis.Mucocutaneous IL-17 immunity in mice and humans: host defense vs. excessive inflammation.Role of podoplanin in the high interleukin-17A secretion resulting from interactions between activated lymphocytes and psoriatic skin-derived mesenchymal cells.IL-17 induces macrophages to M2-like phenotype via NF-κB Epithelial TRAF6 drives IL-17-mediated psoriatic inflammation

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IL-17 drives psoriatic inflammation via distinct, target cell-specific mechanisms.

http://www.wikidata.org/entity/Q34082705

description

2014 nî lūn-bûn

@nan

2014 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի օգոստոսին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

@zh-hant

2014年論文

@zh-hk

2014年論文

@zh-mo

2014年論文

@zh-tw

2014年论文

@wuu

name

IL-17 drives psoriatic inflammation via distinct, target cell-specific mechanisms.

@ast

IL-17 drives psoriatic inflammation via distinct, target cell-specific mechanisms.

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IL-17 drives psoriatic inflammation via distinct, target cell-specific mechanisms.

@nl

type

label

IL-17 drives psoriatic inflammation via distinct, target cell-specific mechanisms.

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IL-17 drives psoriatic inflammation via distinct, target cell-specific mechanisms.

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IL-17 drives psoriatic inflammation via distinct, target cell-specific mechanisms.

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prefLabel

IL-17 drives psoriatic inflammation via distinct, target cell-specific mechanisms.

@ast

IL-17 drives psoriatic inflammation via distinct, target cell-specific mechanisms.

@en

IL-17 drives psoriatic inflammation via distinct, target cell-specific mechanisms.

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PNAS.1400513111

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Proceedings of the National Academy of Sciences of the United States of America

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IL-17 drives psoriatic inflammation via distinct, target cell-specific mechanisms.

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P2093

Hongshan Wang

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Hye-Lin Ha

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Ilaria Tassi

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Jin-Chul Kim

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Maria I Morasso

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Mark C Udey

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Prapaporn Pisitkun

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Ulrich Siebenlist

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Wanhu Tang

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P2860

IL-17C regulates the innate immune function of epithelial cells in an autocrine manner

The emerging role of IL-17 in the pathogenesis of psoriasis: preclinical and clinical findings

Putting together the psoriasis puzzle: an update on developing targeted therapies

The antimicrobial protein REG3A regulates keratinocyte proliferation and differentiation after skin injury

Common variants at TRAF3IP2 are associated with susceptibility to psoriatic arthritis and psoriasis

Th17 cytokines stimulate CCL20 expression in keratinocytes in vitro and in vivo: implications for psoriasis pathogenesis.

Pivotal role of dermal IL-17-producing γδ T cells in skin inflammation.

SEF/IL-17R (SEFIR) is not enough: an extended SEFIR domain is required for il-17RA-mediated signal transduction.

Calcium regulation of growth and differentiation of mouse epidermal cells in culture.

The adaptor protein CIKS/ACT1 is necessary for collagen-induced arthritis, and it contributes to the production of collagen-specific antibody

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