about
Constrained release of lamina-associated enhancers and genes from the nuclear envelope during T-cell activation facilitates their association in chromosome compartments.Research Resource: A Reference Transcriptome for Constitutive Androstane Receptor and Pregnane X Receptor Xenobiotic SignalingIntestinal Microbiota Promotes Psoriasis-Like Skin Inflammation by Enhancing Th17 Response.The Atypical Inhibitor of NF-κB, IκBζ, Controls Macrophage Interleukin-10 Expression.Leptin deficiency in mice counteracts imiquimod (IMQ)-induced psoriasis-like skin inflammation while leptin stimulation induces inflammation in human keratinocytes.IL-17A plays a central role in the expression of psoriasis signature genes through the induction of IκB-ζ in keratinocytes.The resident pathobiont Staphylococcus xylosus in Nfkbiz-deficient skin accelerates spontaneous skin inflammationThe Nuclear Protein IκBζ Forms a Transcriptionally Active Complex with Nuclear Factor-κB (NF-κB) p50 and the Lcn2 Promoter via the N- and C-terminal Ankyrin Repeat Motifs.Effects of RNA interference combined with ultrasonic irradiation and SonoVue microbubbles on expression of STAT3 gene in keratinocytes of psoriatic lesions.Bcl-3 induced by IL-22 via STAT3 activation acts as a potentiator of psoriasis-related gene expression in epidermal keratinocytes.IL-17 Responses Are the Dominant Inflammatory Signal Linking Inverse, Erythrodermic, and Chronic Plaque Psoriasis.IL-17F regulates psoriasis-associated genes through IκBζ.Nuclear Factor-kappaB in Autoimmunity: Man and Mouse.Electrophilic properties of itaconate and derivatives regulate the IκBζ-ATF3 inflammatory axis.Sexy again: the renaissance of neutrophils in psoriasis.Effect of RNA Interference Targeting Gene Combined with Ultrasonic Irradiation and SonoVue Microbubbles on Proliferation and Apoptosis in Keratinocytes of Psoriatic Lesions
P2860
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P2860
description
2015 nî lūn-bûn
@nan
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
2015年论文
@zh
2015年论文
@zh-cn
name
IκBζ is a key driver in the development of psoriasis.
@ast
IκBζ is a key driver in the development of psoriasis.
@en
type
label
IκBζ is a key driver in the development of psoriasis.
@ast
IκBζ is a key driver in the development of psoriasis.
@en
prefLabel
IκBζ is a key driver in the development of psoriasis.
@ast
IκBζ is a key driver in the development of psoriasis.
@en
P2093
P2860
P50
P356
P1476
IκBζ is a key driver in the development of psoriasis.
@en
P2093
Hanne Vinter
Klaus Schulze-Osthoff
Maike Mose
Pernille Ommen
Sebastian Lorscheid
Stephan Hailfinger
Trine Bertelsen
P2860
P304
P356
10.1073/PNAS.1509971112
P407
P577
2015-10-12T00:00:00Z