SERCA pump level is a critical determinant of Ca(2+)homeostasis and cardiac contractility.
about
Review of novel therapeutic targets for improving heart failure treatment based on experimental and clinical studiesEcophysiology of omega Fatty acids: a lid for every jarPerinatal loss of Nkx2-5 results in rapid conduction and contraction defectsPathophysiology of Heart Failure.Phenotypic screen quantifying differential regulation of cardiac myocyte hypertrophy identifies CITED4 regulation of myocyte elongation.Deregulated Ca2+ cycling underlies the development of arrhythmia and heart disease due to mutant obscurinSERCA1 expression enhances the metabolic efficiency of improved contractility in post-ischemic heart.Mimicking isovolumic contraction with combined electromechanical stimulation improves the development of engineered cardiac constructs.TEAD-1 overexpression in the mouse heart promotes an age-dependent heart dysfunction.Long-term obesity promotes alterations in diastolic function induced by reduction of phospholamban phosphorylation at serine-16 without affecting calcium handling.Discovery and structural characterization of a phospholamban-binding cyclic peptide and design of novel inhibitors of phospholamban.Neonatal gene transfer of Serca2a delays onset of hypertrophic remodeling and improves function in familial hypertrophic cardiomyopathy.Synthetic phosphopeptides enable quantitation of the content and function of the four phosphorylation states of phospholamban in cardiac muscleAerobic interval training enhances cardiomyocyte contractility and Ca2+ cycling by phosphorylation of CaMKII and Thr-17 of phospholamban.Myocardial function after fetal cardiac bypass in an ovine modelTherapeutic potential of CPT I inhibitors: cardiac gene transcription as a target.Acute inotropic and lusitropic effects of cardiomyopathic R9C mutation of phospholamban.Abnormalities of capillary microarchitecture in a rat model of coronary ischemic congestive heart failure.Sarcolipin overexpression improves muscle energetics and reduces fatigue.Resistance training regulates cardiac function through modulation of miRNA-214.Ablation of sarcolipin results in atrial remodelingIt's all in the timing: modeling isovolumic contraction through development and disease with a dynamic dual electromechanical bioreactor system.Sarcolipin is a novel regulator of muscle metabolism and obesity.Targeted deletion of microRNA-22 promotes stress-induced cardiac dilation and contractile dysfunction.Regulation of the sarcoplasmic reticulum Ca2+-ATPase expression in the hypertrophic and failing heart.Modulators of right ventricular apoptosis and contractility in a rat model of pulmonary hypertensionSarcolipin and phospholamban as regulators of cardiac sarcoplasmic reticulum Ca2+ ATPase.Prolonged administration of a dithiol antioxidant protects against ventricular remodeling due to ischemia-reperfusion in mice.Determinants of frequency-dependent contraction and relaxation of mammalian myocardium.Conditional increase in SERCA2a protein is able to reverse contractile dysfunction and abnormal calcium flux in established diabetic cardiomyopathy.Ca2+ clearance and contractility in vascular smooth muscle: evidence from gene-altered murine models.Acute heart failure with cardiomyocyte atrophy induced in adult mice by ablation of cardiac myosin light chain kinase.Limited functional and metabolic improvements in hypertrophic and healthy rat heart overexpressing the skeletal muscle isoform of SERCA1 by adenoviral gene transfer in vivo.Role of insulin-like growth factor 1 and phosphoinositide 3-kinase in a setting of heart disease.Molecular basis of diastolic dysfunction.Bendavia restores mitochondrial energy metabolism gene expression and suppresses cardiac fibrosis in the border zone of the infarcted heart.Phosphorylated phospholamban stabilizes a compact conformation of the cardiac calcium-ATPase.Pigment Epithelium-Derived Factor (PEDF) Improves Ischemic Cardiac Functional Reserve Through Decreasing Hypoxic Cardiomyocyte Contractility Through PEDF Receptor (PEDF-R).Designing heart performance by gene transfer.Extracellular matrix of mechanically stretched cardiac fibroblasts improves viability and metabolic activity of ventricular cells.
P2860
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P2860
SERCA pump level is a critical determinant of Ca(2+)homeostasis and cardiac contractility.
description
2001 nî lūn-bûn
@nan
2001 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
name
SERCA pump level is a critical determinant of Ca
@nl
SERCA pump level is a critical ...... sis and cardiac contractility.
@ast
SERCA pump level is a critical ...... sis and cardiac contractility.
@en
type
label
SERCA pump level is a critical determinant of Ca
@nl
SERCA pump level is a critical ...... sis and cardiac contractility.
@ast
SERCA pump level is a critical ...... sis and cardiac contractility.
@en
prefLabel
SERCA pump level is a critical determinant of Ca
@nl
SERCA pump level is a critical ...... sis and cardiac contractility.
@ast
SERCA pump level is a critical ...... sis and cardiac contractility.
@en
P356
P1476
SERCA pump level is a critical ...... sis and cardiac contractility.
@en
P2093
P304
P356
10.1006/JMCC.2001.1366
P577
2001-06-01T00:00:00Z