Altered cortical glutamatergic and GABAergic signal transmission with glial involvement in depression. - Wikidata - awgldk - TriplyDB

Altered cortical glutamatergic and GABAergic signal transmission with glial involvement in depression.

Altered cortical glutamatergic and GABAergic signal transmission with glial involvement in depression.

http://www.wikidata.org/entity/Q34084102

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Antidepressant effects of ketamine: mechanisms underlying fast-acting novel antidepressants Towards a glutamate hypothesis of depression: an emerging frontier of neuropsychopharmacology for mood disorders A New Outlook on Mental Illnesses: Glial Involvement Beyond the Glue Depression as a Glial-Based Synaptic Dysfunction Innate and adaptive immunity in the development of depression: An update on current knowledge and technological advances Functional alterations of astrocytes in mental disorders: pharmacological significance as a drug target Glial abnormalities in mood disorders Stress-induced deficits in cognition and emotionality: a role of glutamate Astrocyte pathology in major depressive disorder: insights from human postmortem brain tissue Glial abnormalities in substance use disorders and depression: does shared glutamatergic dysfunction contribute to comorbidity?The stressed synapse: the impact of stress and glucocorticoids on glutamate transmission Better understanding of mechanisms of schizophrenia and bipolar disorder: from human gene expression profiles to mouse models Roles of glutamine synthetase inhibition in epilepsy Functional regulation of GABAA receptors in nervous system pathologies Toward a biosignature for suicide A brief history of the development of antidepressant drugs: from monoamines to glutamate Molecular and Genetic Characterization of Depression: Overlap with other Psychiatric Disorders and Aging Ketamine decreases resting state functional network connectivity in healthy subjects: implications for antidepressant drug action Sex-Dependent Depression-Like Behavior Induced by Respiratory Administration of Aluminum Oxide Nanoparticles Expression profiling of a genetic animal model of depression reveals novel molecular pathways underlying depressive-like behaviours The GABAergic deficit hypothesis of major depressive disorder.Identification of proteomic signatures associated with depression and psychotic depression in post-mortem brains from major depression patients.Fluoxetine affects GluK2 editing, glutamate-evoked Ca(2+) influx and extracellular signal-regulated kinase phosphorylation in mouse astrocytes.Quantitative traits for the tail suspension test: automation, optimization, and BXD RI mapping.Reduced conditioned fear response in mice that lack Dlx1 and show subtype-specific loss of interneurons Convergent functional genomic studies of ω-3 fatty acids in stress reactivity, bipolar disorder and alcoholism.Gene expression alterations in bipolar disorder postmortem brains.Glutamate and its receptors in the pathophysiology and treatment of major depressive disorder Brain Rewarding Stimulation Reduces Extracellular Glutamate Through Glial Modulation in Medial Prefrontal Cortex of Rats Suicide candidate genes associated with bipolar disorder and schizophrenia: an exploratory gene expression profiling analysis of post-mortem prefrontal cortex Disinhibition of somatostatin-positive GABAergic interneurons results in an anxiolytic and antidepressant-like brain state.Altered γ-aminobutyric acid neurotransmission in major depressive disorder: a critical review of the supporting evidence and the influence of serotonergic antidepressants Inflammation Effects on Brain Glutamate in Depression: Mechanistic Considerations and Treatment Implications.VTA dopamine neuron plasticity - the unusual suspects.Gene expression profiling of neurochemically defined regions of the human brain by in situ hybridization-guided laser capture microdissection Global brain gene expression analysis links glutamatergic and GABAergic alterations to suicide and major depression.Altered expression of glutamate signaling, growth factor, and glia genes in the locus coeruleus of patients with major depression.IFN-alpha-induced cortical and subcortical glutamate changes assessed by magnetic resonance spectroscopy.Gene expression studies in major depression.Altered Organization of GABA(A) Receptor mRNA Expression in the Depressed Suicide Brain

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Altered cortical glutamatergic and GABAergic signal transmission with glial involvement in depression.

http://www.wikidata.org/entity/Q34084102

description

2005 nî lūn-bûn

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2005 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

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2005 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2005年の論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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name

Altered cortical glutamatergic ...... ial involvement in depression.

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Altered cortical glutamatergic ...... ial involvement in depression.

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Altered cortical glutamatergic ...... ial involvement in depression.

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Altered cortical glutamatergic ...... ial involvement in depression.

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Altered cortical glutamatergic ...... ial involvement in depression.

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Altered cortical glutamatergic ...... ial involvement in depression.

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Proceedings of the National Academy of Sciences of the United States of America

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Altered cortical glutamatergic ...... ial involvement in depression.

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E G Jones

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Bioconductor: open software development for computational biology and bioinformatics

Detection of glutamine synthetase in the cerebrospinal fluid of Alzheimer diseased patients: a potential diagnostic biochemical marker

High-resolution mapping of the gamma-aminobutyric acid receptor subunit beta 3 and alpha 5 gene cluster on chromosome 15q11-q13, and localization of breakpoints in two Angelman syndrome patients

Expression monitoring by hybridization to high-density oligonucleotide arrays

Influence of life stress on depression: moderation by a polymorphism in the 5-HTT gene

Glutamate uptake

The glutamate/neutral amino acid transporter family SLC1: molecular, physiological and pharmacological aspects

GABA and glutamate in the human brain

GABAA receptor subunit gene expression in human prefrontal cortex: comparison of schizophrenics and controls

Selective alterations in gene expression for NMDA receptor subunits in prefrontal cortex of schizophrenics

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