Interactive comorbidity between opioid drug abuse and HIV-1 Tat: chronic exposure augments spine loss and sublethal dendritic pathology in striatal neurons - Wikidata - awgldk - TriplyDB

Interactive comorbidity between opioid drug abuse and HIV-1 Tat: chronic exposure augments spine loss and sublethal dendritic pathology in striatal neurons

Interactive comorbidity between opioid drug abuse and HIV-1 Tat: chronic exposure augments spine loss and sublethal dendritic pathology in striatal neurons

http://www.wikidata.org/entity/Q34086976

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HIV-associated neurocognitive disorder--pathogenesis and prospects for treatment Interactions of Opioids and HIV Infection in the Pathogenesis of Chronic Pain Interaction of paroxetine with mitochondrial proteins mediates neuroprotection.Morphine enhances HIV-1SF162-mediated neuron death and delays recovery of injured neurites Effects of opiates and HIV proteins on neurons: the role of ferritin heavy chain and a potential for synergism HIV-1 transgenic female rat: synaptodendritic alterations of medium spiny neurons in the nucleus accumbens.Neonatal intrahippocampal HIV-1 protein Tat(1-86) injection: neurobehavioral alterations in the absence of increased inflammatory cytokine activation.Anxiety-like behavior of mice produced by conditional central expression of the HIV-1 regulatory protein, Tat.Neuronal ferritin heavy chain and drug abuse affect HIV-associated cognitive dysfunction.HIV-1 alters neural and glial progenitor cell dynamics in the central nervous system: coordinated response to opiates during maturation.Synaptodendritic recovery following HIV Tat exposure: neurorestoration by phytoestrogens.Interactive HIV-1 Tat and morphine-induced synaptodendritic injury is triggered through focal disruptions in Na⁺ influx, mitochondrial instability, and Ca²⁺ overload.Transient hypercapnia reveals an underlying cerebrovascular pathology in a murine model for HIV-1 associated neuroinflammation: role of NO-cGMP signaling and normalization by inhibition of cyclic nucleotide phosphodiesterase-5 HIV transactivator of transcription enhances methamphetamine-induced Parkinson's-like behavior in the rats.Interactions of HIV and drugs of abuse: the importance of glia, neural progenitors, and host genetic factors.HIV-1 Tat activates a RhoA signaling pathway to reduce NMDA-evoked calcium responses in hippocampal neurons via an actin-dependent mechanism Effects of chronic HIV-1 Tat exposure in the CNS: heightened vulnerability of males versus females to changes in cell numbers, synaptic integrity, and behavior Drugs of Abuse in HIV infection and neurotoxicity.Creatine protects against mitochondrial dysfunction associated with HIV-1 Tat-induced neuronal injury.Sensitization of enteric neurons to morphine by HIV-1 Tat protein.Eradication of human immunodeficiency virus from brain reservoirs.Morphine potentiates neurodegenerative effects of HIV-1 Tat through actions at μ-opioid receptor-expressing glia.HIV-1 proteins, Tat and gp120, target the developing dopamine system Opiate addiction therapies and HIV-1 Tat: interactive effects on glial [Ca²⁺]i, oxyradical and neuroinflammatory chemokine production and correlative neurotoxicity.MicroRNAs in opioid pharmacology.Morphine and gp120 toxic interactions in striatal neurons are dependent on HIV-1 strain.Modeling HIV-associated neurocognitive disorders in mice: new approaches in the changing face of HIV neuropathogenesis Morphine efficacy is altered in conditional HIV-1 Tat transgenic mice.Subtype selective NMDA receptor antagonists induce recovery of synapses lost following exposure to HIV-1 Tat.Opiate drug use and the pathophysiology of neuroAIDS.Chronic HIV-1 Tat and HIV reduce Rbfox3/NeuN: evidence for sex-related effects Morphine and microRNA Activity: Is There a Relation with Addiction?HIV-1-Tat Protein Inhibits SC35-mediated Tau Exon 10 Inclusion through Up-regulation of DYRK1A Kinase.Morphine Tolerance and Physical Dependence Are Altered in Conditional HIV-1 Tat Transgenic Mice.Regulation and Functional Implications of Opioid Receptor Splicing in Opioid Pharmacology and HIV Pathogenesis Mechanism(s) involved in opioid drug abuse modulation of HAND Synaptic dysfunction in the hippocampus accompanies learning and memory deficits in human immunodeficiency virus type-1 Tat transgenic mice.Expression of HIV-Tat protein is associated with learning and memory deficits in the mouse.Morphine exposure during HIV encephalitis in SCID mice.Conditional Tat protein expression in the GT-tg bigenic mouse brain induces gray matter density reductions.

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P2860

Interactive comorbidity between opioid drug abuse and HIV-1 Tat: chronic exposure augments spine loss and sublethal dendritic pathology in striatal neurons

http://www.wikidata.org/entity/Q34086976

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2010 nî lūn-bûn

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2010 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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2010 թվականի հուլիսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Interactive comorbidity betwee ...... pathology in striatal neurons

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Interactive comorbidity betwee ...... pathology in striatal neurons

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Interactive comorbidity betwee ...... pathology in striatal neurons

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type

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Interactive comorbidity betwee ...... pathology in striatal neurons

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Interactive comorbidity betwee ...... pathology in striatal neurons

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Interactive comorbidity betwee ...... pathology in striatal neurons

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Interactive comorbidity betwee ...... pathology in striatal neurons

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Interactive comorbidity betwee ...... pathology in striatal neurons

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Interactive comorbidity betwee ...... pathology in striatal neurons

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The American Journal of Pathology

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Interactive comorbidity betwee ...... pathology in striatal neurons

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Avindra Nath

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Cecilia Bull

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Kurt F Hauser

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Shreya K Buch

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P2860

kappa opioid receptors in human microglia downregulate human immunodeficiency virus 1 expression

Mu-opioid receptors modulate the stability of dendritic spines

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

Mitochondrial dysfunction and oxidative stress in neurodegenerative diseases

Opioid-receptor mRNA expression in the rat CNS: anatomical and functional implications

The neuropathogenesis of AIDS

Synergistic neurotoxicity of opioids and human immunodeficiency virus-1 Tat protein in striatal neurons in vitro

Opioid system diversity in developing neurons, astroglia, and oligodendroglia in the subventricular zone and striatum: impact on gliogenesis in vivo

HIV-1 Tat-mediated activation of glycogen synthase kinase-3beta contributes to Tat-mediated neurotoxicity

Morphine exacerbates HIV-1 Tat-induced cytokine production in astrocytes through convergent effects on [Ca(2+)](i), NF-kappaB trafficking and transcription

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corpus striatum

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