Epigenetic GABAergic targets in schizophrenia and bipolar disorder. - Wikidata - awgldk - TriplyDB

Epigenetic GABAergic targets in schizophrenia and bipolar disorder.

Epigenetic GABAergic targets in schizophrenia and bipolar disorder.

http://www.wikidata.org/entity/Q34087810

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The involvement of Reelin in neurodevelopmental disorders.In Sickness and in Health: Perineuronal Nets and Synaptic Plasticity in Psychiatric Disorders Small molecule inhibitors of zinc-dependent histone deacetylases Current status and future prospects for epigenetic psychopharmacology Active DNA demethylation in post-mitotic neurons: a reason for optimism Epigenetic mechanisms in mood disorders: targeting neuroplasticity Mice that lack the C-terminal region of Reelin exhibit behavioral abnormalities related to neuropsychiatric disorders.Histone Deacetylase (HDAC) Inhibitor Kinetic Rate Constants Correlate with Cellular Histone Acetylation but Not Transcription and Cell Viability Metabotropic Glutamate 2/3 Receptors and Epigenetic Modifications in Psychotic Disorders: A Review Dissecting bipolar disorder complexity through epigenomic approach Treatment-resistant schizophrenia: current insights on the pharmacogenomics of antipsychotics Growth arrest and DNA-damage-inducible, beta (GADD45b)-mediated DNA demethylation in major psychosis Antidepressants inhibit DNA methyltransferase 1 through reducing G9a levels Searching human brain for mechanisms of psychiatric disorders. Implications for studies on schizophrenia.Epigenetic modifications of GABAergic interneurons are associated with the schizophrenia-like phenotype induced by prenatal stress in mice.Advancing drug discovery for schizophrenia.Distinct phenotypes of new transmembrane-domain neuregulin 1 mutant mice and the rescue effects of valproate on the observed schizophrenia-related cognitive deficits Behavioral and molecular neuroepigenetic alterations in prenatally stressed mice: relevance for the study of chromatin remodeling properties of antipsychotic drugs Theranostic Biomarkers for Schizophrenia PI3K/AKT/mTOR Signaling Mediates Valproic Acid-Induced Neuronal Differentiation of Neural Stem Cells through Epigenetic Modifications.Genetics of schizophrenia from a clinicial perspective.Epigenetic mechanisms during ageing and neurogenesis as novel therapeutic avenues in human brain disorders.DNA-methylation gene network dysregulation in peripheral blood lymphocytes of schizophrenia patients.Valproate as a treatment for dopamine dysregulation syndrome (DDS) in Parkinson's disease.Modeling the molecular epigenetic profile of psychosis in prenatally stressed mice.Redox-based epigenetic status in drug addiction: a potential contributor to gene priming and a mechanistic rationale for metabolic intervention.Activation of group II metabotropic glutamate receptors promotes DNA demethylation in the mouse brain.Extracellular matrix abnormalities in schizophrenia DNA-methyltransferase1 (DNMT1) binding to CpG rich GABAergic and BDNF promoters is increased in the brain of schizophrenia and bipolar disorder patients.Pharmacological activation of group-II metabotropic glutamate receptors corrects a schizophrenia-like phenotype induced by prenatal stress in mice.Losing the sugar coating: potential impact of perineuronal net abnormalities on interneurons in schizophrenia.Gadd45b and N-methyl-D-aspartate induced DNA demethylation in postmitotic neurons.Epigenetic management of major psychosis.Epigenetic Regulation of Glutamic Acid Decarboxylase 67 in a Hippocampal Circuit.Maternal immune activation produces neonatal excitability defects in offspring hippocampal neurons from pregnant rats treated with poly I:C The dynamics of DNA methylation in schizophrenia and related psychiatric disorders.Stress-induced epigenetic transcriptional memory of acetylcholinesterase by HDAC4.DNA Methylation of BDNF Gene in Schizophrenia.DNA methylation in schizophrenia: progress and challenges of epigenetic studies Epigenetic control of neurotransmitter expression in olfactory bulb interneurons.

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Epigenetic GABAergic targets in schizophrenia and bipolar disorder.

http://www.wikidata.org/entity/Q34087810

description

2010 nî lūn-bûn

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2010 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

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2010 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Epigenetic GABAergic targets in schizophrenia and bipolar disorder.

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Epigenetic GABAergic targets in schizophrenia and bipolar disorder.

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Epigenetic GABAergic targets in schizophrenia and bipolar disorder.

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type

label

Epigenetic GABAergic targets in schizophrenia and bipolar disorder.

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Epigenetic GABAergic targets in schizophrenia and bipolar disorder.

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Epigenetic GABAergic targets in schizophrenia and bipolar disorder.

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Epigenetic GABAergic targets in schizophrenia and bipolar disorder.

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Epigenetic GABAergic targets in schizophrenia and bipolar disorder.

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Epigenetic GABAergic targets in schizophrenia and bipolar disorder.

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J.NEUROPHARM.2010.10.021

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Neuropharmacology

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Epigenetic GABAergic targets in schizophrenia and bipolar disorder.

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P2093

A Guidotti

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D P Gavin

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D R Grayson

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F Matrisciano

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L Tremolizzo

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P2860

Genome-wide association identifies a common variant in the reelin gene that increases the risk of schizophrenia only in women

Translating the Histone Code

Reelin and glutamic acid decarboxylase67 promoter remodeling in an epigenetic methionine-induced mouse model of schizophrenia

Reelin promoter hypermethylation in schizophrenia

Maternal care effects on the hippocampal transcriptome and anxiety-mediated behaviors in the offspring that are reversible in adulthood

An epigenetic mouse model for molecular and behavioral neuropathologies related to schizophrenia vulnerability.

In psychosis, cortical interneurons overexpress DNA-methyltransferase 1

DNA-methyltransferase 1 mRNA is selectively overexpressed in telencephalic GABAergic interneurons of schizophrenia brains

Histone deactylase 1 expression is increased in the prefrontal cortex of schizophrenia subjects: analysis of the National Brain Databank microarray collection

Circuit-based framework for understanding neurotransmitter and risk gene interactions in schizophrenia

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bipolar disorder

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